These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

248 related items for PubMed ID: 29855476

  • 1. Programming for increased expression of hippocampal GAD67 mediated the hypersensitivity of the hypothalamic-pituitary-adrenal axis in male offspring rats with prenatal ethanol exposure.
    Lu J, Jiao Z, Yu Y, Zhang C, He X, Li Q, Xu D, Wang H.
    Cell Death Dis; 2018 May 31; 9(6):659. PubMed ID: 29855476
    [Abstract] [Full Text] [Related]

  • 2. Prenatal nicotine exposure induces HPA axis-hypersensitivity in offspring rats via the intrauterine programming of up-regulation of hippocampal GAD67.
    He X, Lu J, Dong W, Jiao Z, Zhang C, Yu Y, Zhang Z, Wang H, Xu D.
    Arch Toxicol; 2017 Dec 31; 91(12):3927-3943. PubMed ID: 28555334
    [Abstract] [Full Text] [Related]

  • 3. Programming of a developmental imbalance in hypothalamic glutamatergic/GABAergic afferents mediates low basal activity of the hypothalamic-pituitary-adrenal axis induced by prenatal dexamethasone exposure in male offspring rats.
    Lu J, Li Q, Xu D, Liao Y, Wang H.
    Toxicol Lett; 2020 Oct 01; 331():33-41. PubMed ID: 32445661
    [Abstract] [Full Text] [Related]

  • 4. Prenatal ethanol exposure-induced hypothalamic an imbalance of glutamatergic/GABAergic projections and low functional expression in male offspring rats.
    Lu J, Li Q, Ma G, Hong C, Zhang W, Wang Y, Wang H.
    Food Chem Toxicol; 2020 Jul 01; 141():111419. PubMed ID: 32437893
    [Abstract] [Full Text] [Related]

  • 5. High expression of hippocampal glutamic acid decarboxylase 67 mediates hypersensitivity of the hypothalamic-pituitary-adrenal axis in response to prenatal caffeine exposure in rats.
    Xu D, Zhang C, He X, Guo Z, Hu D, Lu J, Cheng S, Wang H.
    Toxicol Lett; 2018 Feb 01; 283():39-51. PubMed ID: 29111460
    [No Abstract] [Full Text] [Related]

  • 6. Excitotoxicity and compensatory upregulation of GAD67 in fetal rat hippocampus caused by prenatal nicotine exposure are associated with inhibition of the BDNF pathway.
    Pei Y, Jiao Z, Dong W, Pei L, He X, Wang H, Xu D.
    Food Chem Toxicol; 2019 Jan 01; 123():314-325. PubMed ID: 30389584
    [Abstract] [Full Text] [Related]

  • 7. The upregulation of glutamate decarboxylase 67 against hippocampal excitability damage in male fetal rats by prenatal caffeine exposure.
    Lu M, He X, Jiao Z, Hu Z, Guo Z, Dai S, Wang H, Xu D.
    Environ Toxicol; 2022 Nov 01; 37(11):2703-2717. PubMed ID: 35917217
    [Abstract] [Full Text] [Related]

  • 8. Prenatal ethanol exposure enhances the susceptibility to metabolic syndrome in offspring rats by HPA axis-associated neuroendocrine metabolic programming.
    Xia LP, Shen L, Kou H, Zhang BJ, Zhang L, Wu Y, Li XJ, Xiong J, Yu Y, Wang H.
    Toxicol Lett; 2014 Apr 07; 226(1):98-105. PubMed ID: 24472613
    [Abstract] [Full Text] [Related]

  • 9. Chronic prenatal ethanol exposure increases glucocorticoid-induced glutamate release in the hippocampus of the near-term foetal guinea pig.
    Iqbal U, Brien JF, Kapoor A, Matthews SG, Reynolds JN.
    J Neuroendocrinol; 2006 Nov 07; 18(11):826-34. PubMed ID: 17026532
    [Abstract] [Full Text] [Related]

  • 10. Prenatal ethanol exposure induced disorder of hypothalamic-pituitary-adrenal axis-associated neuroendocrine metabolic programming alteration and dysfunction of glucose and lipid metabolism in 40-week-old female offspring rats.
    Xia L, Jiao Z, Pei L, Yuan C, Zhao Y, Guo Y, Wang H.
    Reprod Toxicol; 2020 Jun 07; 94():48-54. PubMed ID: 32333956
    [Abstract] [Full Text] [Related]

  • 11. Intergenerational genetic programming mechanism and sex differences of the adrenal corticosterone synthesis dysfunction in offspring induced by prenatal ethanol exposure.
    Cao J, Chen Y, Xia X, Qu H, Ao Y, Wang H.
    Toxicol Lett; 2021 Oct 15; 351():78-88. PubMed ID: 34454011
    [Abstract] [Full Text] [Related]

  • 12. Dysfunction of the hypothalamic‑pituitary‑adrenal axis in male rat offspring with prenatal food restriction: Fetal programming of hypothalamic hyperexcitability and poor hippocampal feedback.
    Wen Y, Cheng S, Lu J, He X, Jiao Z, Xu D, Wang H.
    Mol Med Rep; 2022 Jan 15; 25(1):. PubMed ID: 34796908
    [Abstract] [Full Text] [Related]

  • 13. Glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis programming mediated hepatic lipid-metabolic in offspring caused by prenatal ethanol exposure.
    Hu W, Yuan C, Luo H, Hu S, Shen L, Chen L, Xu D, Wang H.
    Toxicol Lett; 2020 Oct 01; 331():167-177. PubMed ID: 32535229
    [Abstract] [Full Text] [Related]

  • 14. Prenatal ethanol exposure-induced adrenal developmental abnormality of male offspring rats and its possible intrauterine programming mechanisms.
    Huang H, He Z, Zhu C, Liu L, Kou H, Shen L, Wang H.
    Toxicol Appl Pharmacol; 2015 Oct 01; 288(1):84-94. PubMed ID: 26188107
    [Abstract] [Full Text] [Related]

  • 15. An intergenerational effect of neuroendocrine metabolic programming alteration induced by prenatal ethanol exposure in rats.
    Kou H, Shen L, Luo HW, Chen LB, Wu DF, Wang H.
    Reprod Toxicol; 2017 Dec 01; 74():85-93. PubMed ID: 28911811
    [Abstract] [Full Text] [Related]

  • 16. Ethanol-induced inhibition of fetal hypothalamic-pituitary-adrenal axis due to prenatal overexposure to maternal glucocorticoid in mice.
    Liang G, Chen M, Pan XL, Zheng J, Wang H.
    Exp Toxicol Pathol; 2011 Nov 01; 63(7-8):607-11. PubMed ID: 20627497
    [Abstract] [Full Text] [Related]

  • 17. Intrauterine programming of the glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis mediates glomerulosclerosis in female adult offspring rats induced by prenatal ethanol exposure.
    He H, Xiong Y, Li B, Zhu Y, Chen H, Ao Y, Wang H.
    Toxicol Lett; 2019 Sep 01; 311():17-26. PubMed ID: 31039417
    [Abstract] [Full Text] [Related]

  • 18. Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats.
    Liu M, Zhang Q, Pei L, Zou Y, Chen G, Wang H.
    Epigenetics; 2019 Mar 01; 14(3):245-259. PubMed ID: 30821590
    [Abstract] [Full Text] [Related]

  • 19. 3-O-Acetyl-11-keto-β-boswellic acid ameliorates chronic unpredictable mild stress induced HPA axis dysregulation in relation with glutamate/GABA aberration in depressive rats.
    Gunasekaran V, Augustine A, Avarachan J, Khayum A, Ramasamy A.
    Clin Exp Pharmacol Physiol; 2021 Dec 01; 48(12):1633-1641. PubMed ID: 34343356
    [Abstract] [Full Text] [Related]

  • 20. Prenatal xenobiotic exposure and intrauterine hypothalamus-pituitary-adrenal axis programming alteration.
    Zhang C, Xu D, Luo H, Lu J, Liu L, Ping J, Wang H.
    Toxicology; 2014 Nov 05; 325():74-84. PubMed ID: 25194749
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 13.