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417 related items for PubMed ID: 29863609
1. Inhibition of Fatty Acid Amide Hydrolase Improves Depressive-Like Behaviors Independent of Its Peripheral Antinociceptive Effects in a Rat Model of Neuropathic Pain. Jiang HX, Ke BW, Liu J, Ma G, Hai KR, Gong DY, Yang Z, Zhou C. Anesth Analg; 2019 Aug; 129(2):587-597. PubMed ID: 29863609 [Abstract] [Full Text] [Related]
2. Full inhibition of spinal FAAH leads to TRPV1-mediated analgesic effects in neuropathic rats and possible lipoxygenase-mediated remodeling of anandamide metabolism. Starowicz K, Makuch W, Korostynski M, Malek N, Slezak M, Zychowska M, Petrosino S, De Petrocellis L, Cristino L, Przewlocka B, Di Marzo V. PLoS One; 2013 Aug; 8(4):e60040. PubMed ID: 23573230 [Abstract] [Full Text] [Related]
3. Alterations in endocannabinoid tone following chemotherapy-induced peripheral neuropathy: effects of endocannabinoid deactivation inhibitors targeting fatty-acid amide hydrolase and monoacylglycerol lipase in comparison to reference analgesics following cisplatin treatment. Guindon J, Lai Y, Takacs SM, Bradshaw HB, Hohmann AG. Pharmacol Res; 2013 Jan; 67(1):94-109. PubMed ID: 23127915 [Abstract] [Full Text] [Related]
6. Brain-Permeant and -Impermeant Inhibitors of Fatty Acid Amide Hydrolase Synergize with the Opioid Analgesic Morphine to Suppress Chemotherapy-Induced Neuropathic Nociception Without Enhancing Effects of Morphine on Gastrointestinal Transit. Slivicki RA, Saberi SA, Iyer V, Vemuri VK, Makriyannis A, Hohmann AG. J Pharmacol Exp Ther; 2018 Dec; 367(3):551-563. PubMed ID: 30275151 [Abstract] [Full Text] [Related]
7. Brain permeant and impermeant inhibitors of fatty-acid amide hydrolase suppress the development and maintenance of paclitaxel-induced neuropathic pain without producing tolerance or physical dependence in vivo and synergize with paclitaxel to reduce tumor cell line viability in vitro. Slivicki RA, Xu Z, Mali SS, Hohmann AG. Pharmacol Res; 2019 Apr; 142():267-282. PubMed ID: 30739035 [Abstract] [Full Text] [Related]
8. Effects of the fatty acid amide hydrolase inhibitor URB597 on pain-stimulated and pain-depressed behavior in rats. Kwilasz AJ, Abdullah RA, Poklis JL, Lichtman AH, Negus SS. Behav Pharmacol; 2014 Apr; 25(2):119-29. PubMed ID: 24583930 [Abstract] [Full Text] [Related]
9. The multiplicity of spinal AA-5-HT anti-nociceptive action in a rat model of neuropathic pain. Malek N, Kostrzewa M, Makuch W, Pajak A, Kucharczyk M, Piscitelli F, Przewlocka B, Di Marzo V, Starowicz K. Pharmacol Res; 2016 Sep; 111():251-263. PubMed ID: 27326920 [Abstract] [Full Text] [Related]
10. Dysfunction in fatty acid amide hydrolase is associated with depressive-like behavior in Wistar Kyoto rats. Vinod KY, Xie S, Psychoyos D, Hungund BL, Cooper TB, Tejani-Butt SM. PLoS One; 2012 Sep; 7(5):e36743. PubMed ID: 22606285 [Abstract] [Full Text] [Related]
12. Lack of effect of chronic pre-treatment with the FAAH inhibitor URB597 on inflammatory pain behaviour: evidence for plastic changes in the endocannabinoid system. Okine BN, Norris LM, Woodhams S, Burston J, Patel A, Alexander SP, Barrett DA, Kendall DA, Bennett AJ, Chapman V. Br J Pharmacol; 2012 Oct; 167(3):627-40. PubMed ID: 22595021 [Abstract] [Full Text] [Related]
13. Endocannabinoids decrease neuropathic pain-related behavior in mice through the activation of one or both peripheral CB₁ and CB₂ receptors. Desroches J, Charron S, Bouchard JF, Beaulieu P. Neuropharmacology; 2014 Feb; 77():441-52. PubMed ID: 24148808 [Abstract] [Full Text] [Related]
15. Pharmacological blockade of fatty acid amide hydrolase (FAAH) by URB597 improves memory and changes the phenotype of hippocampal microglia despite ethanol exposure. Rivera P, Fernández-Arjona MDM, Silva-Peña D, Blanco E, Vargas A, López-Ávalos MD, Grondona JM, Serrano A, Pavón FJ, Rodríguez de Fonseca F, Suárez J. Biochem Pharmacol; 2018 Nov; 157():244-257. PubMed ID: 30098312 [Abstract] [Full Text] [Related]
16. Characterization of the fatty acid amide hydrolase inhibitor cyclohexyl carbamic acid 3'-carbamoyl-biphenyl-3-yl ester (URB597): effects on anandamide and oleoylethanolamide deactivation. Fegley D, Gaetani S, Duranti A, Tontini A, Mor M, Tarzia G, Piomelli D. J Pharmacol Exp Ther; 2005 Apr; 313(1):352-8. PubMed ID: 15579492 [Abstract] [Full Text] [Related]
17. Inhibition of fatty acid amide hydrolase activates Nrf2 signalling and induces heme oxygenase 1 transcription in breast cancer cells. Li H, Wood JT, Whitten KM, Vadivel SK, Seng S, Makriyannis A, Avraham HK. Br J Pharmacol; 2013 Oct; 170(3):489-505. PubMed ID: 23347118 [Abstract] [Full Text] [Related]
18. The fatty acid amide hydrolase inhibitor URB597 (cyclohexylcarbamic acid 3'-carbamoylbiphenyl-3-yl ester) reduces neuropathic pain after oral administration in mice. Russo R, Loverme J, La Rana G, Compton TR, Parrott J, Duranti A, Tontini A, Mor M, Tarzia G, Calignano A, Piomelli D. J Pharmacol Exp Ther; 2007 Jul; 322(1):236-42. PubMed ID: 17412883 [Abstract] [Full Text] [Related]
19. Reduced anxiety-like behaviour induced by genetic and pharmacological inhibition of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (FAAH) is mediated by CB1 receptors. Moreira FA, Kaiser N, Monory K, Lutz B. Neuropharmacology; 2008 Jan; 54(1):141-50. PubMed ID: 17709120 [Abstract] [Full Text] [Related]
20. Attenuation of cue-induced reinstatement of nicotine seeking by URB597 through cannabinoid CB1 receptor in rats. Forget B, Guranda M, Gamaleddin I, Goldberg SR, Le Foll B. Psychopharmacology (Berl); 2016 May; 233(10):1823-8. PubMed ID: 26864774 [Abstract] [Full Text] [Related] Page: [Next] [New Search]