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Journal Abstract Search

99 related items for PubMed ID: 3001593

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  • 3. Unilateral substantia nigra lesions and schedule-induced polydipsia.
    Mittleman G, Valenstein ES.
    Physiol Behav; 1986; 36(3):437-40. PubMed ID: 3085114
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  • 5. Changes in substantia nigra pars reticulata activity following lesions of the substantia nigra pars compacta.
    Sanderson P, Mavoungou R, Albe-Fessard D.
    Neurosci Lett; 1986 Jun 06; 67(1):25-30. PubMed ID: 3014391
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  • 7. Unilateral 6-hydroxydopamine lesions of dopamine neurons produce bilateral self-stimulation deficits.
    Carey RJ.
    Behav Brain Res; 1982 Oct 06; 6(2):101-14. PubMed ID: 6814462
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  • 12. Exposure to amphetamine after substantia nigra lesion interferes with the process of behavioral recovery.
    Mintz M, Tomer R.
    Pharmacol Biochem Behav; 1986 Dec 06; 25(6):1307-11. PubMed ID: 3101073
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  • 13. Nocturnal rotation in normal rats: correlation with amphetamine-induced rotation and effects of nigrostriatal lesions.
    Glick SD, Cox RD.
    Brain Res; 1978 Jul 07; 150(1):149-61. PubMed ID: 566609
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  • 15. Behavioural recovery following transplantation of substantia nigra in rats subjected to 6-OHDA lesions of the nigrostriatal pathway. I. Unilateral lesions.
    Dunnett SB, Björklund A, Stenevi U, Iversen SD.
    Brain Res; 1981 Jun 29; 215(1-2):147-61. PubMed ID: 7260584
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  • 18. The influence of dopaminergic A10 neurons on the motor pattern evoked by substantia nigra (pars compacta) stimulation.
    Piazza PV, Ferdico M, Russo D, Crescimanno G, Benigno A, Amato G.
    Behav Brain Res; 1989 Jan 01; 31(3):273-8. PubMed ID: 2914078
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  • 19. Quantification of lesion-induced dopaminergic supersensitivity using the rotational model in the mouse.
    Mandel RJ, Randall PK.
    Brain Res; 1985 Mar 25; 330(2):358-63. PubMed ID: 3921193
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