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576 related items for PubMed ID: 30021895

  • 1. Mutant Cellular AP-1 Proteins Promote Expression of a Subset of Epstein-Barr Virus Late Genes in the Absence of Lytic Viral DNA Replication.
    Lyons DE, Yu KP, Vander Heiden JA, Heston L, Dittmer DP, El-Guindy A, Miller G.
    J Virol; 2018 Oct 01; 92(19):. PubMed ID: 30021895
    [Abstract] [Full Text] [Related]

  • 2. The SWI/SNF Chromatin Regulator BRG1 Modulates the Transcriptional Regulatory Activity of the Epstein-Barr Virus DNA Polymerase Processivity Factor BMRF1.
    Su MT, Wang YT, Chen YJ, Lin SF, Tsai CH, Chen MR.
    J Virol; 2017 May 01; 91(9):. PubMed ID: 28228591
    [Abstract] [Full Text] [Related]

  • 3. Amino acid substitutions reveal distinct functions of serine 186 of the ZEBRA protein in activation of early lytic cycle genes and synergy with the Epstein-Barr virus R transactivator.
    Francis A, Ragoczy T, Gradoville L, Heston L, El-Guindy A, Endo Y, Miller G.
    J Virol; 1999 Jun 01; 73(6):4543-51. PubMed ID: 10233912
    [Abstract] [Full Text] [Related]

  • 4. A Noncanonical Basic Motif of Epstein-Barr Virus ZEBRA Protein Facilitates Recognition of Methylated DNA, High-Affinity DNA Binding, and Lytic Activation.
    Weber E, Buzovetsky O, Heston L, Yu KP, Knecht KM, El-Guindy A, Miller G, Xiong Y.
    J Virol; 2019 Jul 15; 93(14):. PubMed ID: 31068430
    [Abstract] [Full Text] [Related]

  • 5. Latency of Epstein-Barr virus is disrupted by gain-of-function mutant cellular AP-1 proteins that preferentially bind methylated DNA.
    Yu KP, Heston L, Park R, Ding Z, Wang'ondu R, Delecluse HJ, Miller G.
    Proc Natl Acad Sci U S A; 2013 May 14; 110(20):8176-81. PubMed ID: 23625009
    [Abstract] [Full Text] [Related]

  • 6. Amino acids in the basic domain of Epstein-Barr virus ZEBRA protein play distinct roles in DNA binding, activation of early lytic gene expression, and promotion of viral DNA replication.
    Heston L, El-Guindy A, Countryman J, Dela Cruz C, Delecluse HJ, Miller G.
    J Virol; 2006 Sep 14; 80(18):9115-33. PubMed ID: 16940523
    [Abstract] [Full Text] [Related]

  • 7. Functional and physical interactions between the Epstein-Barr virus (EBV) proteins BZLF1 and BMRF1: Effects on EBV transcription and lytic replication.
    Zhang Q, Hong Y, Dorsky D, Holley-Guthrie E, Zalani S, Elshiekh NA, Kiehl A, Le T, Kenney S.
    J Virol; 1996 Aug 14; 70(8):5131-42. PubMed ID: 8764021
    [Abstract] [Full Text] [Related]

  • 8. Mutations of amino acids in the DNA-recognition domain of Epstein-Barr virus ZEBRA protein alter its sub-nuclear localization and affect formation of replication compartments.
    Park R, Heston L, Shedd D, Delecluse HJ, Miller G.
    Virology; 2008 Dec 20; 382(2):145-62. PubMed ID: 18937960
    [Abstract] [Full Text] [Related]

  • 9. The Epstein-Barr Virus BMRF1 Protein Activates Transcription and Inhibits the DNA Damage Response by Binding NuRD.
    Salamun SG, Sitz J, De La Cruz-Herrera CF, Yockteng-Melgar J, Marcon E, Greenblatt J, Fradet-Turcotte A, Frappier L.
    J Virol; 2019 Nov 15; 93(22):. PubMed ID: 31462557
    [Abstract] [Full Text] [Related]

  • 10. DNA Damage Signaling Is Induced in the Absence of Epstein-Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA.
    Wang'ondu R, Teal S, Park R, Heston L, Delecluse H, Miller G.
    PLoS One; 2015 Nov 15; 10(5):e0126088. PubMed ID: 25950714
    [Abstract] [Full Text] [Related]

  • 11. Comparing transcriptional activation and autostimulation by ZEBRA and ZEBRA/c-Fos chimeras.
    Kolman JL, Taylor N, Gradoville L, Countryman J, Miller G.
    J Virol; 1996 Mar 15; 70(3):1493-504. PubMed ID: 8627667
    [Abstract] [Full Text] [Related]

  • 12. Alteration of a single serine in the basic domain of the Epstein-Barr virus ZEBRA protein separates its functions of transcriptional activation and disruption of latency.
    Francis AL, Gradoville L, Miller G.
    J Virol; 1997 Apr 15; 71(4):3054-61. PubMed ID: 9060666
    [Abstract] [Full Text] [Related]

  • 13. ZEBRA and a Fos-GCN4 chimeric protein differ in their DNA-binding specificities for sites in the Epstein-Barr virus BZLF1 promoter.
    Taylor N, Flemington E, Kolman JL, Baumann RP, Speck SH, Miller G.
    J Virol; 1991 Aug 15; 65(8):4033-41. PubMed ID: 1649314
    [Abstract] [Full Text] [Related]

  • 14. Role of the epstein-barr virus RTA protein in activation of distinct classes of viral lytic cycle genes.
    Ragoczy T, Miller G.
    J Virol; 1999 Dec 15; 73(12):9858-66. PubMed ID: 10559298
    [Abstract] [Full Text] [Related]

  • 15. Structural basis of DNA methylation-dependent site selectivity of the Epstein-Barr virus lytic switch protein ZEBRA/Zta/BZLF1.
    Bernaudat F, Gustems M, Günther J, Oliva MF, Buschle A, Göbel C, Pagniez P, Lupo J, Signor L, Müller CW, Morand P, Sattler M, Hammerschmidt W, Petosa C.
    Nucleic Acids Res; 2022 Jan 11; 50(1):490-511. PubMed ID: 34893887
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  • 19. Protein kinase C-independent activation of the Epstein-Barr virus lytic cycle.
    Gradoville L, Kwa D, El-Guindy A, Miller G.
    J Virol; 2002 Jun 11; 76(11):5612-26. PubMed ID: 11991990
    [Abstract] [Full Text] [Related]

  • 20. CAGE-seq analysis of Epstein-Barr virus lytic gene transcription: 3 kinetic classes from 2 mechanisms.
    Djavadian R, Hayes M, Johannsen E.
    PLoS Pathog; 2018 Jun 11; 14(6):e1007114. PubMed ID: 29864140
    [Abstract] [Full Text] [Related]

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