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24. Relative refractory period as a measure of peripheral nerve neurotoxicity. Anderson RJ. Toxicol Appl Pharmacol; 1983 Dec; 71(3):391-7. PubMed ID: 6318389 [Abstract] [Full Text] [Related]
25. Changes in Na-K ATPase and protein kinase C activities in peripheral nerve of acrylamide-treated rats. Lehning EJ, LoPachin RM, Mathew J, Eichberg J. J Toxicol Environ Health; 1994 Jul; 42(3):331-42. PubMed ID: 8021966 [Abstract] [Full Text] [Related]
27. Acrylamide neuropathy: changes in the composition of proteins of fast axonal transport resemble those observed in regenerating axons. Bisby MA, Redshaw JD. J Neurochem; 1987 Mar; 48(3):924-8. PubMed ID: 2433402 [Abstract] [Full Text] [Related]
37. A catechol derivative (4-methylcatechol) accelerates the recovery from experimental acrylamide-induced neuropathy. Saita K, Ohi T, Hanaoka Y, Furukawa S, Furukawa Y, Hayashi K, Matsukura S. J Pharmacol Exp Ther; 1996 Jan; 276(1):231-7. PubMed ID: 8558436 [Abstract] [Full Text] [Related]
38. Alterations in the neutral proteinase activities of central and peripheral nervous systems of acrylamide-, carbon disulfide-, or 2,5-hexanedione-treated rats. Gupta RP, Abou-Donia MB. Mol Chem Neuropathol; 1996 Sep; 29(1):53-66. PubMed ID: 8887940 [Abstract] [Full Text] [Related]
40. A simple, sensitive method for detecting early peripheral nerve dysfunction in the rat following acrylamide treatment. Miller MJ, Miller MS, Burks TF, Sipes IG. Neurotoxicology; 1984 Sep; 5(2):15-23. PubMed ID: 6095142 [Abstract] [Full Text] [Related] Page: [Previous] [Next] [New Search]