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Journal Abstract Search

348 related items for PubMed ID: 30076216

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  • 44. A novel maladaptive unfolded protein response as a mechanism for small bowel resection-induced liver injury.
    Steinberger AE, Tecos ME, Phelps HM, Rubin DC, Davidson NO, Guo J, Warner BW.
    Am J Physiol Gastrointest Liver Physiol; 2022 Sep 01; 323(3):G165-G176. PubMed ID: 35727920
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  • 51. Free fatty acid induces endoplasmic reticulum stress and apoptosis of β-cells by Ca2+/calpain-2 pathways.
    Cui W, Ma J, Wang X, Yang W, Zhang J, Ji Q.
    PLoS One; 2013 Sep 01; 8(3):e59921. PubMed ID: 23527285
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  • 52. Polychlorinated biphenyl quinone induces endoplasmic reticulum stress, unfolded protein response, and calcium release.
    Xu D, Su C, Song X, Shi Q, Fu J, Hu L, Xia X, Song E, Song Y.
    Chem Res Toxicol; 2015 Jun 15; 28(6):1326-37. PubMed ID: 25950987
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  • 53. Augmentation of endoplasmic reticulum stress in cerebral ischemia/reperfusion injury associated with comorbid type 2 diabetes.
    Srinivasan K, Sharma SS.
    Neurol Res; 2011 Oct 15; 33(8):858-65. PubMed ID: 22004710
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  • 55. Suppression of cortical TRPM7 protein attenuates oxidative damage after traumatic brain injury via Akt/endothelial nitric oxide synthase pathway.
    Xu HL, Liu MD, Yuan XH, Liu CX.
    Neurochem Int; 2018 Jan 15; 112():197-205. PubMed ID: 28736242
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  • 56. Dexmedetomidine alleviates cerebral ischemia-reperfusion injury by inhibiting endoplasmic reticulum stress dependent apoptosis through the PERK-CHOP-Caspase-11 pathway.
    Liu C, Fu Q, Mu R, Wang F, Zhou C, Zhang L, Yu B, Zhang Y, Fang T, Tian F.
    Brain Res; 2018 Dec 15; 1701():246-254. PubMed ID: 30201260
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  • 57. Trinitrotoluene Induces Endoplasmic Reticulum Stress and Apoptosis in HePG2 Cells.
    Song L, Wang Y, Wang J, Yang F, Li X, Wu Y.
    Med Sci Monit; 2015 Nov 09; 21():3434-41. PubMed ID: 26551326
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  • 59. A crosstalk between p21 and UPR-induced transcription factor C/EBP homologous protein (CHOP) linked to type 2 diabetes.
    Mihailidou C, Papavassiliou AG, Kiaris H.
    Biochimie; 2014 Apr 09; 99():19-27. PubMed ID: 24239558
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  • 60. High Glucose-Induced Podocyte Injury Involves Activation of Mammalian Target of Rapamycin (mTOR)-Induced Endoplasmic Reticulum (ER) Stress.
    Lei J, Zhao L, Zhang Y, Wu Y, Liu Y.
    Cell Physiol Biochem; 2018 Apr 09; 45(6):2431-2443. PubMed ID: 29554648
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