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PUBMED FOR HANDHELDS

Journal Abstract Search


350 related items for PubMed ID: 30127460

  • 1. Contribution of BH3-domain and Transmembrane-domain to the Activity and Interaction of the Pore-forming Bcl-2 Proteins Bok, Bak, and Bax.
    Stehle D, Grimm M, Einsele-Scholz S, Ladwig F, Johänning J, Fischer G, Gillissen B, Schulze-Osthoff K, Essmann F.
    Sci Rep; 2018 Aug 20; 8(1):12434. PubMed ID: 30127460
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  • 5. Characterization of the anti-apoptotic mechanism of Bcl-B.
    Zhai D, Ke N, Zhang H, Ladror U, Joseph M, Eichinger A, Godzik A, Ng SC, Reed JC.
    Biochem J; 2003 Nov 15; 376(Pt 1):229-36. PubMed ID: 12921534
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  • 6. A splicing variant of the Bcl-2 member Bok with a truncated BH3 domain induces apoptosis but does not dimerize with antiapoptotic Bcl-2 proteins in vitro.
    Hsu SY, Hsueh AJ.
    J Biol Chem; 1998 Nov 13; 273(46):30139-46. PubMed ID: 9804769
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  • 7. Mcl-1 determines the Bax dependency of Nbk/Bik-induced apoptosis.
    Gillissen B, Essmann F, Hemmati PG, Richter A, Richter A, Oztop I, Chinnadurai G, Dörken B, Daniel PT.
    J Cell Biol; 2007 Nov 19; 179(4):701-15. PubMed ID: 18025305
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  • 9. Translocation of a Bak C-terminus mutant from cytosol to mitochondria to mediate cytochrome C release: implications for Bak and Bax apoptotic function.
    Ferrer PE, Frederick P, Gulbis JM, Dewson G, Kluck RM.
    PLoS One; 2012 Nov 19; 7(3):e31510. PubMed ID: 22442658
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  • 12. Visualization of BOK pores independent of BAX and BAK reveals a similar mechanism with differing regulation.
    Shalaby R, Diwan A, Flores-Romero H, Hertlein V, Garcia-Saez AJ.
    Cell Death Differ; 2023 Mar 19; 30(3):731-741. PubMed ID: 36289446
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  • 13. Bax/Bak activation in the absence of Bid, Bim, Puma, and p53.
    Zhang J, Huang K, O'Neill KL, Pang X, Luo X.
    Cell Death Dis; 2016 Jun 16; 7(6):e2266. PubMed ID: 27310874
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  • 14. Direct interaction of Bax and Bak proteins with Bcl-2 homology domain 3 (BH3)-only proteins in living cells revealed by fluorescence complementation.
    Vela L, Gonzalo O, Naval J, Marzo I.
    J Biol Chem; 2013 Feb 15; 288(7):4935-46. PubMed ID: 23283967
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  • 15. An interconnected hierarchical model of cell death regulation by the BCL-2 family.
    Chen HC, Kanai M, Inoue-Yamauchi A, Tu HC, Huang Y, Ren D, Kim H, Takeda S, Reyna DE, Chan PM, Ganesan YT, Liao CP, Gavathiotis E, Hsieh JJ, Cheng EH.
    Nat Cell Biol; 2015 Oct 15; 17(10):1270-81. PubMed ID: 26344567
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  • 16. The BCL-2 family member BID plays a role during embryonic development in addition to its BH3-only protein function by acting in parallel to BAX, BAK and BOK.
    Ke FS, Holloway S, Uren RT, Wong AW, Little MH, Kluck RM, Voss AK, Strasser A.
    EMBO J; 2022 Aug 01; 41(15):e110300. PubMed ID: 35758142
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  • 17. Differential regulation of Bax and Bak by anti-apoptotic Bcl-2 family proteins Bcl-B and Mcl-1.
    Zhai D, Jin C, Huang Z, Satterthwait AC, Reed JC.
    J Biol Chem; 2008 Apr 11; 283(15):9580-6. PubMed ID: 18178565
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  • 18. Inactivation of prosurvival Bcl-2 proteins activates Bax/Bak through the outer mitochondrial membrane.
    O'Neill KL, Huang K, Zhang J, Chen Y, Luo X.
    Genes Dev; 2016 Apr 15; 30(8):973-88. PubMed ID: 27056669
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  • 19. Bid chimeras indicate that most BH3-only proteins can directly activate Bak and Bax, and show no preference for Bak versus Bax.
    Hockings C, Anwari K, Ninnis RL, Brouwer J, O'Hely M, Evangelista M, Hinds MG, Czabotar PE, Lee EF, Fairlie WD, Dewson G, Kluck RM.
    Cell Death Dis; 2015 Apr 23; 6(4):e1735. PubMed ID: 25906158
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  • 20. Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or Bak.
    Willis SN, Fletcher JI, Kaufmann T, van Delft MF, Chen L, Czabotar PE, Ierino H, Lee EF, Fairlie WD, Bouillet P, Strasser A, Kluck RM, Adams JM, Huang DC.
    Science; 2007 Feb 09; 315(5813):856-9. PubMed ID: 17289999
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