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Journal Abstract Search

602 related items for PubMed ID: 30233569

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  • 2. Keratin Retraction and Desmoglein3 Internalization Independently Contribute to Autoantibody-Induced Cell Dissociation in Pemphigus Vulgaris.
    Schlögl E, Radeva MY, Vielmuth F, Schinner C, Waschke J, Spindler V.
    Front Immunol; 2018; 9():858. PubMed ID: 29922278
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  • 5. Synergistic pathogenic effects of combined mouse monoclonal anti-desmoglein 3 IgG antibodies on pemphigus vulgaris blister formation.
    Kawasaki H, Tsunoda K, Hata T, Ishii K, Yamada T, Amagai M.
    J Invest Dermatol; 2006 Dec; 126(12):2621-30. PubMed ID: 16841036
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  • 6. A pathogenic autoantibody, pemphigus vulgaris-IgG, induces phosphorylation of desmoglein 3, and its dissociation from plakoglobin in cultured keratinocytes.
    Aoyama Y, Owada MK, Kitajima Y.
    Eur J Immunol; 1999 Jul; 29(7):2233-40. PubMed ID: 10427986
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  • 7. Signaling dependent and independent mechanisms in pemphigus vulgaris blister formation.
    Saito M, Stahley SN, Caughman CY, Mao X, Tucker DK, Payne AS, Amagai M, Kowalczyk AP.
    PLoS One; 2012 Jul; 7(12):e50696. PubMed ID: 23226536
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  • 11. Pemphigus vulgaris: recent advances in our understanding of its pathogenesis.
    Femiano F.
    Minerva Stomatol; 2007 Apr; 56(4):215-23. PubMed ID: 17452959
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  • 12. High-dose pemphigus antibodies against linear epitopes of desmoglein 3 (Dsg3) can induce acantholysis and depletion of Dsg3 from keratinocytes.
    Cirillo N, Femiano F, Gombos F, Lanza A.
    Immunol Lett; 2009 Feb 21; 122(2):208-13. PubMed ID: 19200441
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  • 16. Role of ADAM10 and ADAM17 in the Regulation of Keratinocyte Adhesion in Pemphigus Vulgaris.
    Kugelmann D, Anders M, Sigmund AM, Egu DT, Eichkorn RA, Yazdi AS, Sárdy M, Hertl M, Didona D, Hashimoto T, Waschke J.
    Front Immunol; 2022 Feb 21; 13():884248. PubMed ID: 35844545
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