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190 related items for PubMed ID: 3038697

  • 1. Induction of v-mos and activated Ha-ras oncogene expression in quiescent NIH 3T3 cells causes intracellular alkalinisation and cell-cycle progression.
    Doppler W, Jaggi R, Groner B.
    Gene; 1987; 54(1):147-53. PubMed ID: 3038697
    [Abstract] [Full Text] [Related]

  • 2. Oncogenes modulate cellular gene expression and repress glucocorticoid regulated gene transcription.
    Jaggi R, Friis R, Groner B.
    J Steroid Biochem; 1988 May; 29(5):457-63. PubMed ID: 3379954
    [Abstract] [Full Text] [Related]

  • 3. Desensitization of the Ca2+-mobilizing system to serum growth factors by Ha-ras and v-mos.
    Maly K, Doppler W, Oberhuber H, Meusburger H, Hofmann J, Jaggi R, Grunicke HH.
    Mol Cell Biol; 1988 Oct; 8(10):4212-6. PubMed ID: 3141785
    [Abstract] [Full Text] [Related]

  • 4. Effect of a dominant inhibitory Ha-ras mutation on mitogenic signal transduction in NIH 3T3 cells.
    Cai H, Szeberényi J, Cooper GM.
    Mol Cell Biol; 1990 Oct; 10(10):5314-23. PubMed ID: 2118993
    [Abstract] [Full Text] [Related]

  • 5. Effect of Ha-ras on phosphatidylinositol metabolism, Na+/H+-antiporter and mobilization of intracellular calcium.
    Maly K, Oberhuber H, Doppler W, Hoflacher J, Jaggi R, Groner B, Grunicke H.
    Adv Enzyme Regul; 1988 Oct; 27():121-31. PubMed ID: 2854946
    [Abstract] [Full Text] [Related]

  • 6. The v-mos and c-Ha-ras oncoproteins exert similar effects on the pattern of protein synthesis.
    Klemenz R, Hoffmann S, Jaggi R, Werenskiold AK.
    Oncogene; 1989 Jun; 4(6):799-803. PubMed ID: 2525241
    [Abstract] [Full Text] [Related]

  • 7. The v-mos and H-ras oncogene expression represses glucocorticoid hormone-dependent transcription from the mouse mammary tumor virus LTR.
    Jaggi R, Salmons B, Muellener D, Groner B.
    EMBO J; 1986 Oct; 5(10):2609-16. PubMed ID: 3023051
    [Abstract] [Full Text] [Related]

  • 8. Down-regulation of RI alpha subunit of cAMP-dependent protein kinase induces growth inhibition of human mammary epithelial cells transformed by c-Ha-ras and c-erbB-2 proto-oncogenes.
    Ciardiello F, Pepe S, Bianco C, Baldassarre G, Ruggiero A, Bianco C, Selvam MP, Bianco AR, Tortora G.
    Int J Cancer; 1993 Feb 01; 53(3):438-43. PubMed ID: 8094073
    [Abstract] [Full Text] [Related]

  • 9. Microinjection of ras p21 induces a rapid rise in intracellular pH.
    Hagag N, Lacal JC, Graber M, Aaronson S, Viola MV.
    Mol Cell Biol; 1987 May 01; 7(5):1984-8. PubMed ID: 3037340
    [Abstract] [Full Text] [Related]

  • 10. Transforming growth factor-alpha expression is enhanced in human mammary epithelial cells transformed by an activated c-Ha-ras protooncogene but not by the c-neu protooncogene, and overexpression of the transforming growth factor-alpha complementary DNA leads to transformation.
    Ciardiello F, McGeady ML, Kim N, Basolo F, Hynes N, Langton BC, Yokozaki H, Saeki T, Elliott JW, Masui H.
    Cell Growth Differ; 1990 Sep 01; 1(9):407-20. PubMed ID: 1981145
    [Abstract] [Full Text] [Related]

  • 11. Mechanism and biological significance of the Ha-ras-induced activation of the Na+/H(+)-antiporter.
    Maly K, Hochleitner B, Uberall F, Loferer H, Oberhuber H, Doppler W, Grunicke H.
    Adv Enzyme Regul; 1990 Sep 01; 30():63-74. PubMed ID: 2169700
    [Abstract] [Full Text] [Related]

  • 12. Evidence that the time of entry into S is determined by events occurring in early G1.
    Cosenza SC, Owen TA, Soprano DR, Soprano KJ.
    J Biol Chem; 1988 Sep 05; 263(25):12751-8. PubMed ID: 3137230
    [Abstract] [Full Text] [Related]

  • 13. Requirement for fos gene expression in the transcriptional activation of collagenase by other oncogenes and phorbol esters.
    Schönthal A, Herrlich P, Rahmsdorf HJ, Ponta H.
    Cell; 1988 Jul 29; 54(3):325-34. PubMed ID: 2840203
    [Abstract] [Full Text] [Related]

  • 14. Cytoskeletal reorganization in NIH 3T3 fibroblasts expressing the ras oncogene.
    Dartsch PC, Ritter M, Häussinger D, Lang F.
    Eur J Cell Biol; 1994 Apr 29; 63(2):316-25. PubMed ID: 8082656
    [Abstract] [Full Text] [Related]

  • 15. Lipid signalling pathways in normal and ras-transfected NIH/3T3 cells.
    Matyas GR, Fishman PH.
    Cell Signal; 1989 Apr 29; 1(4):395-404. PubMed ID: 2561989
    [Abstract] [Full Text] [Related]

  • 16. mos-induced inhibition of glucocorticoid receptor function is mediated by Fos.
    Touray M, Ryan F, Saurer S, Martin F, Jaggi R.
    Oncogene; 1991 Feb 29; 6(2):211-7. PubMed ID: 1847998
    [Abstract] [Full Text] [Related]

  • 17. Mechanism of carcinogenesis: the role of oncogenes, transcriptional enhancers and growth factors.
    Spandidos DA.
    Anticancer Res; 1985 Feb 29; 5(5):485-98. PubMed ID: 3904595
    [Abstract] [Full Text] [Related]

  • 18. Modification of Ha-ras oncogene p21 expression and cell cycle progression in the human colonic cancer cell line HT-29.
    Czerniak B, Herz F, Wersto RP, Koss LG.
    Cancer Res; 1987 Jun 01; 47(11):2826-30. PubMed ID: 3105868
    [Abstract] [Full Text] [Related]

  • 19. Different promoter elements are required for the induced expression of c-fos and c-jun proto-oncogenes by the v-mos oncogene product.
    Schönthal A, Feramisco JR.
    New Biol; 1990 Feb 01; 2(2):143-50. PubMed ID: 2150599
    [Abstract] [Full Text] [Related]

  • 20. Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain.
    Binétruy B, Smeal T, Karin M.
    Nature; 1991 May 09; 351(6322):122-7. PubMed ID: 1903181
    [Abstract] [Full Text] [Related]


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