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Journal Abstract Search

327 related items for PubMed ID: 30446867

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  • 28. Early recognition of CLN3 disease facilitated by visual electrophysiology and multimodal imaging.
    Sakti DH, Cornish EE, Fraser CL, Nash BM, Sandercoe TM, Jones MM, Rowe NA, Jamieson RV, Johnson AM, Grigg JR.
    Doc Ophthalmol; 2023 Jun; 146(3):241-256. PubMed ID: 36964447
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  • 29. OPTICAL COHERENCE TOMOGRAPHY IN JUVENILE NEURONAL CEROID LIPOFUSCINOSIS.
    Hansen MS, Hove MN, Jensen H, Larsen M.
    Retin Cases Brief Rep; 2016 Jun; 10(2):137-9. PubMed ID: 26308342
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  • 31. Histopathologic and immunocytochemical analysis of the retina and ocular tissues in Batten disease.
    Bensaoula T, Shibuya H, Katz ML, Smith JE, Johnson GS, John SK, Milam AH.
    Ophthalmology; 2000 Sep; 107(9):1746-53. PubMed ID: 10964839
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  • 32. Transcript and in silico analysis of CLN3 in juvenile neuronal ceroid lipofuscinosis and associated mouse models.
    Chan CH, Mitchison HM, Pearce DA.
    Hum Mol Genet; 2008 Nov 01; 17(21):3332-9. PubMed ID: 18678598
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  • 33. Self-Complementary AAV9 Gene Delivery Partially Corrects Pathology Associated with Juvenile Neuronal Ceroid Lipofuscinosis (CLN3).
    Bosch ME, Aldrich A, Fallet R, Odvody J, Burkovetskaya M, Schuberth K, Fitzgerald JA, Foust KD, Kielian T.
    J Neurosci; 2016 Sep 14; 36(37):9669-82. PubMed ID: 27629717
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  • 35. A novel deletion variant in CLN3 with highly variable expressivity is responsible for juvenile neuronal ceroid lipofuscinoses.
    Gilani N, Razmara E, Ozaslan M, Abdulzahra IK, Arzhang S, Tavasoli AR, Garshasbi M.
    Acta Neurol Belg; 2021 Jun 14; 121(3):737-748. PubMed ID: 33783722
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  • 36. Retinal pathology and function in a Cln3 knockout mouse model of juvenile Neuronal Ceroid Lipofuscinosis (batten disease).
    Seigel GM, Lotery A, Kummer A, Bernard DJ, Greene ND, Turmaine M, Derksen T, Nussbaum RL, Davidson B, Wagner J, Mitchison HM.
    Mol Cell Neurosci; 2002 Apr 14; 19(4):515-27. PubMed ID: 11988019
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  • 37. Neuronal ceroid lipofuscinoses: research update.
    Wisniewski KE, Kida E, Connell F, Zhong N.
    Neurol Sci; 2000 Apr 14; 21(3 Suppl):S49-56. PubMed ID: 11073228
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  • 38. Large-scale phenotyping of an accurate genetic mouse model of JNCL identifies novel early pathology outside the central nervous system.
    Staropoli JF, Haliw L, Biswas S, Garrett L, Hölter SM, Becker L, Skosyrski S, Da Silva-Buttkus P, Calzada-Wack J, Neff F, Rathkolb B, Rozman J, Schrewe A, Adler T, Puk O, Sun M, Favor J, Racz I, Bekeredjian R, Busch DH, Graw J, Klingenspor M, Klopstock T, Wolf E, Wurst W, Zimmer A, Lopez E, Harati H, Hill E, Krause DS, Guide J, Dragileva E, Gale E, Wheeler VC, Boustany RM, Brown DE, Breton S, Ruether K, Gailus-Durner V, Fuchs H, de Angelis MH, Cotman SL.
    PLoS One; 2012 Apr 14; 7(6):e38310. PubMed ID: 22701626
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  • 39. Synapse alterations precede neuronal damage and storage pathology in a human cerebral organoid model of CLN3-juvenile neuronal ceroid lipofuscinosis.
    Gomez-Giro G, Arias-Fuenzalida J, Jarazo J, Zeuschner D, Ali M, Possemis N, Bolognin S, Halder R, Jäger C, Kuper WFE, van Hasselt PM, Zaehres H, Del Sol A, van der Putten H, Schöler HR, Schwamborn JC.
    Acta Neuropathol Commun; 2019 Dec 30; 7(1):222. PubMed ID: 31888773
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  • 40. Recognizing differentiating clinical signs of CLN3 disease (Batten disease) at presentation.
    Kuper WFE, Talsma HE, van Schooneveld MJ, Pott JWR, Huijgen BCH, de Wit GC, van Hasselt PM, van Genderen MM.
    Acta Ophthalmol; 2021 Jun 30; 99(4):397-404. PubMed ID: 33073538
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