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Journal Abstract Search


1142 related items for PubMed ID: 30617209

  • 1. Increased Resurgent Sodium Currents in Nav1.8 Contribute to Nociceptive Sensory Neuron Hyperexcitability Associated with Peripheral Neuropathies.
    Xiao Y, Barbosa C, Pei Z, Xie W, Strong JA, Zhang JM, Cummins TR.
    J Neurosci; 2019 Feb 20; 39(8):1539-1550. PubMed ID: 30617209
    [Abstract] [Full Text] [Related]

  • 2. Tetrodotoxin-resistant sodium channels in sensory neurons generate slow resurgent currents that are enhanced by inflammatory mediators.
    Tan ZY, Piekarz AD, Priest BT, Knopp KL, Krajewski JL, McDermott JS, Nisenbaum ES, Cummins TR.
    J Neurosci; 2014 May 21; 34(21):7190-7. PubMed ID: 24849353
    [Abstract] [Full Text] [Related]

  • 3. Small-fiber neuropathy Nav1.8 mutation shifts activation to hyperpolarized potentials and increases excitability of dorsal root ganglion neurons.
    Huang J, Yang Y, Zhao P, Gerrits MM, Hoeijmakers JG, Bekelaar K, Merkies IS, Faber CG, Dib-Hajj SD, Waxman SG.
    J Neurosci; 2013 Aug 28; 33(35):14087-97. PubMed ID: 23986244
    [Abstract] [Full Text] [Related]

  • 4. Navβ4 regulates fast resurgent sodium currents and excitability in sensory neurons.
    Barbosa C, Tan ZY, Wang R, Xie W, Strong JA, Patel RR, Vasko MR, Zhang JM, Cummins TR.
    Mol Pain; 2015 Sep 25; 11():60. PubMed ID: 26408173
    [Abstract] [Full Text] [Related]

  • 5. The G1662S NaV1.8 mutation in small fibre neuropathy: impaired inactivation underlying DRG neuron hyperexcitability.
    Han C, Vasylyev D, Macala LJ, Gerrits MM, Hoeijmakers JG, Bekelaar KJ, Dib-Hajj SD, Faber CG, Merkies IS, Waxman SG.
    J Neurol Neurosurg Psychiatry; 2014 May 25; 85(5):499-505. PubMed ID: 24006052
    [Abstract] [Full Text] [Related]

  • 6. Activation of tetrodotoxin-resistant sodium channel NaV1.9 in rat primary sensory neurons contributes to melittin-induced pain behavior.
    Yu YQ, Zhao ZY, Chen XF, Xie F, Yang Y, Chen J.
    Neuromolecular Med; 2013 Mar 25; 15(1):209-17. PubMed ID: 23264124
    [Abstract] [Full Text] [Related]

  • 7. FHF2 isoforms differentially regulate Nav1.6-mediated resurgent sodium currents in dorsal root ganglion neurons.
    Barbosa C, Xiao Y, Johnson AJ, Xie W, Strong JA, Zhang JM, Cummins TR.
    Pflugers Arch; 2017 Feb 25; 469(2):195-212. PubMed ID: 27999940
    [Abstract] [Full Text] [Related]

  • 8. Sea-anemone toxin ATX-II elicits A-fiber-dependent pain and enhances resurgent and persistent sodium currents in large sensory neurons.
    Klinger AB, Eberhardt M, Link AS, Namer B, Kutsche LK, Schuy ET, Sittl R, Hoffmann T, Alzheimer C, Huth T, Carr RW, Lampert A.
    Mol Pain; 2012 Sep 15; 8():69. PubMed ID: 22978421
    [Abstract] [Full Text] [Related]

  • 9. Tetrodotoxin-Sensitive Sodium Channels Mediate Action Potential Firing and Excitability in Menthol-Sensitive Vglut3-Lineage Sensory Neurons.
    Griffith TN, Docter TA, Lumpkin EA.
    J Neurosci; 2019 Sep 04; 39(36):7086-7101. PubMed ID: 31300524
    [Abstract] [Full Text] [Related]

  • 10. Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli.
    Yang Y, Huang J, Mis MA, Estacion M, Macala L, Shah P, Schulman BR, Horton DB, Dib-Hajj SD, Waxman SG.
    J Neurosci; 2016 Jul 13; 36(28):7511-22. PubMed ID: 27413160
    [Abstract] [Full Text] [Related]

  • 11. Differential effect of D623N variant and wild-type Na(v)1.7 sodium channels on resting potential and interspike membrane potential of dorsal root ganglion neurons.
    Ahn HS, Vasylyev DV, Estacion M, Macala LJ, Shah P, Faber CG, Merkies IS, Dib-Hajj SD, Waxman SG.
    Brain Res; 2013 Sep 05; 1529():165-77. PubMed ID: 23850641
    [Abstract] [Full Text] [Related]

  • 12. A gain-of-function voltage-gated sodium channel 1.8 mutation drives intense hyperexcitability of A- and C-fiber neurons.
    Garrison SR, Weyer AD, Barabas ME, Beutler BA, Stucky CL.
    Pain; 2014 May 05; 155(5):896-905. PubMed ID: 24447515
    [Abstract] [Full Text] [Related]

  • 13. DRG Voltage-Gated Sodium Channel 1.7 Is Upregulated in Paclitaxel-Induced Neuropathy in Rats and in Humans with Neuropathic Pain.
    Li Y, North RY, Rhines LD, Tatsui CE, Rao G, Edwards DD, Cassidy RM, Harrison DS, Johansson CA, Zhang H, Dougherty PM.
    J Neurosci; 2018 Jan 31; 38(5):1124-1136. PubMed ID: 29255002
    [Abstract] [Full Text] [Related]

  • 14. Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents.
    Jarecki BW, Piekarz AD, Jackson JO, Cummins TR.
    J Clin Invest; 2010 Jan 31; 120(1):369-78. PubMed ID: 20038812
    [Abstract] [Full Text] [Related]

  • 15. Upregulation of the sodium channel NaVβ4 subunit and its contributions to mechanical hypersensitivity and neuronal hyperexcitability in a rat model of radicular pain induced by local dorsal root ganglion inflammation.
    Xie W, Tan ZY, Barbosa C, Strong JA, Cummins TR, Zhang JM.
    Pain; 2016 Apr 31; 157(4):879-891. PubMed ID: 26785322
    [Abstract] [Full Text] [Related]

  • 16. Inherited pain: sodium channel Nav1.7 A1632T mutation causes erythromelalgia due to a shift of fast inactivation.
    Eberhardt M, Nakajima J, Klinger AB, Neacsu C, Hühne K, O'Reilly AO, Kist AM, Lampe AK, Fischer K, Gibson J, Nau C, Winterpacht A, Lampert A.
    J Biol Chem; 2014 Jan 24; 289(4):1971-80. PubMed ID: 24311784
    [Abstract] [Full Text] [Related]

  • 17. Functional up-regulation of Nav1.8 sodium channel on dorsal root ganglia neurons contributes to the induction of scorpion sting pain.
    Ye P, Hua L, Jiao Y, Li Z, Qin S, Fu J, Jiang F, Liu T, Ji Y.
    Acta Biochim Biophys Sin (Shanghai); 2016 Feb 24; 48(2):132-44. PubMed ID: 26764239
    [Abstract] [Full Text] [Related]

  • 18. Ih current stabilizes excitability in rodent DRG neurons and reverses hyperexcitability in a nociceptive neuron model of inherited neuropathic pain.
    Vasylyev DV, Liu S, Waxman SG.
    J Physiol; 2023 Dec 24; 601(23):5341-5366. PubMed ID: 37846879
    [Abstract] [Full Text] [Related]

  • 19. Prefrontal cortex pyramidal neurons express functional Nav1.8 tetrodotoxin-resistant sodium currents.
    Szulczyk B, Pasierski M, Gawlak M.
    Clin Exp Pharmacol Physiol; 2022 Mar 24; 49(3):350-359. PubMed ID: 34750860
    [Abstract] [Full Text] [Related]

  • 20. Local knockdown of the NaV1.6 sodium channel reduces pain behaviors, sensory neuron excitability, and sympathetic sprouting in rat models of neuropathic pain.
    Xie W, Strong JA, Zhang JM.
    Neuroscience; 2015 Apr 16; 291():317-30. PubMed ID: 25686526
    [Abstract] [Full Text] [Related]


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