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Journal Abstract Search


217 related items for PubMed ID: 30824683

  • 1. Quantitative proteomic analyses of dynamic signalling events in cortical neurons undergoing excitotoxic cell death.
    Hoque A, Williamson NA, Ameen SS, Ciccotosto GD, Hossain MI, Oakhill JS, Ng DCH, Ang CS, Cheng HC.
    Cell Death Dis; 2019 Mar 01; 10(3):213. PubMed ID: 30824683
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  • 2. C-terminal fragment of tetanus toxin heavy chain activates Akt and MEK/ERK signalling pathways in a Trk receptor-dependent manner in cultured cortical neurons.
    Gil C, Chaib-Oukadour I, Aguilera J.
    Biochem J; 2003 Jul 15; 373(Pt 2):613-20. PubMed ID: 12710887
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  • 10. Hydrogen peroxide-mediated phosphorylation of ERK1/2, Akt/PKB and JNK in cortical neurones: dependence on Ca(2+) and PI3-kinase.
    Crossthwaite AJ, Hasan S, Williams RJ.
    J Neurochem; 2002 Jan 15; 80(1):24-35. PubMed ID: 11796740
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  • 14. Phosphorylation of microtubule-associated protein tau by isoforms of c-Jun N-terminal kinase (JNK).
    Yoshida H, Hastie CJ, McLauchlan H, Cohen P, Goedert M.
    J Neurochem; 2004 Jul 15; 90(2):352-8. PubMed ID: 15228592
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  • 16. Multiple pathways of apolipoprotein E signaling in primary neurons.
    Hoe HS, Harris DC, Rebeck GW.
    J Neurochem; 2005 Apr 15; 93(1):145-55. PubMed ID: 15773914
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  • 17. Phosphorylation sites on tau identified by nanoelectrospray mass spectrometry: differences in vitro between the mitogen-activated protein kinases ERK2, c-Jun N-terminal kinase and P38, and glycogen synthase kinase-3beta.
    Reynolds CH, Betts JC, Blackstock WP, Nebreda AR, Anderton BH.
    J Neurochem; 2000 Apr 15; 74(4):1587-95. PubMed ID: 10737616
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  • 18. Rapid tyrosine phosphorylation of neuronal proteins including tau and focal adhesion kinase in response to amyloid-beta peptide exposure: involvement of Src family protein kinases.
    Williamson R, Scales T, Clark BR, Gibb G, Reynolds CH, Kellie S, Bird IN, Varndell IM, Sheppard PW, Everall I, Anderton BH.
    J Neurosci; 2002 Jan 01; 22(1):10-20. PubMed ID: 11756483
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  • 19. Proteomic analysis of cortical neuronal cultures treated with poly-arginine peptide-18 (R18) and exposed to glutamic acid excitotoxicity.
    MacDougall G, Anderton RS, Mastaglia FL, Knuckey NW, Meloni BP.
    Mol Brain; 2019 Jul 17; 12(1):66. PubMed ID: 31315638
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