These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Journal Abstract Search
584 related items for PubMed ID: 30855407
1. Ligustilide Ameliorates the Permeability of the Blood-Brain Barrier Model In Vitro During Oxygen-Glucose Deprivation Injury Through HIF/VEGF Pathway. Wu S, Wang N, Li J, Wang G, Seto SW, Chang D, Liang H. J Cardiovasc Pharmacol; 2019 May; 73(5):316-325. PubMed ID: 30855407 [Abstract] [Full Text] [Related]
4. Baicalin reduces the permeability of the blood-brain barrier during hypoxia in vitro by increasing the expression of tight junction proteins in brain microvascular endothelial cells. Zhu H, Wang Z, Xing Y, Gao Y, Ma T, Lou L, Lou J, Gao Y, Wang S, Wang Y. J Ethnopharmacol; 2012 Jun 01; 141(2):714-20. PubMed ID: 21920425 [Abstract] [Full Text] [Related]
8. Minocycline ameliorates hypoxia-induced blood-brain barrier damage by inhibition of HIF-1α through SIRT-3/PHD-2 degradation pathway. Yang F, Zhou L, Wang D, Wang Z, Huang QY. Neuroscience; 2015 Sep 24; 304():250-9. PubMed ID: 26211444 [Abstract] [Full Text] [Related]
9. Inhibition of MicroRNA-155 Supports Endothelial Tight Junction Integrity Following Oxygen-Glucose Deprivation. Pena-Philippides JC, Gardiner AS, Caballero-Garrido E, Pan R, Zhu Y, Roitbak T. J Am Heart Assoc; 2018 Jun 26; 7(13):. PubMed ID: 29945912 [Abstract] [Full Text] [Related]
11. HIF-1 is involved in high glucose-induced paracellular permeability of brain endothelial cells. Yan J, Zhang Z, Shi H. Cell Mol Life Sci; 2012 Jan 26; 69(1):115-28. PubMed ID: 21617913 [Abstract] [Full Text] [Related]
14. Salvianolic acid B improves the disruption of high glucose-mediated brain microvascular endothelial cells via the ROS/HIF-1α/VEGF and miR-200b/VEGF signaling pathways. Yang MC, You FL, Wang Z, Liu XN, Wang YF. Neurosci Lett; 2016 Sep 06; 630():233-240. PubMed ID: 27497919 [Abstract] [Full Text] [Related]
16. Potential contribution of hypoxia-inducible factor-1α, aquaporin-4, and matrix metalloproteinase-9 to blood-brain barrier disruption and brain edema after experimental subarachnoid hemorrhage. Wang Z, Meng CJ, Shen XM, Shu Z, Ma C, Zhu GQ, Liu HX, He WC, Sun XB, Huo L, Zhang J, Chen G. J Mol Neurosci; 2012 Sep 06; 48(1):273-80. PubMed ID: 22528459 [Abstract] [Full Text] [Related]
17. Neutralization of interleukin-9 ameliorates experimental stroke by repairing the blood-brain barrier via down-regulation of astrocyte-derived vascular endothelial growth factor-A. Tan S, Shan Y, Lin Y, Liao S, Zhang B, Zeng Q, Wang Y, Deng Z, Chen C, Hu X, Peng L, Qiu W, Lu Z. FASEB J; 2019 Mar 06; 33(3):4376-4387. PubMed ID: 30694693 [Abstract] [Full Text] [Related]
19. The extracellular matrix protein laminin-10 promotes blood-brain barrier repair after hypoxia and inflammation in vitro. Kangwantas K, Pinteaux E, Penny J. J Neuroinflammation; 2016 Feb 01; 13():25. PubMed ID: 26832174 [Abstract] [Full Text] [Related]