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433 related items for PubMed ID: 30870435

  • 1. Defining the role of NG2-expressing cells in experimental models of multiple sclerosis. A biofunctional analysis of the neurovascular unit in wild type and NG2 null mice.
    Girolamo F, Errede M, Longo G, Annese T, Alias C, Ferrara G, Morando S, Trojano M, Kerlero de Rosbo N, Uccelli A, Virgintino D.
    PLoS One; 2019; 14(3):e0213508. PubMed ID: 30870435
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  • 2. NG2, a common denominator for neuroinflammation, blood-brain barrier alteration, and oligodendrocyte precursor response in EAE, plays a role in dendritic cell activation.
    Ferrara G, Errede M, Girolamo F, Morando S, Ivaldi F, Panini N, Bendotti C, Perris R, Furlan R, Virgintino D, Kerlero de Rosbo N, Uccelli A.
    Acta Neuropathol; 2016 Jul; 132(1):23-42. PubMed ID: 27026411
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  • 3. NG2-proteoglycan-dependent contributions of oligodendrocyte progenitors and myeloid cells to myelin damage and repair.
    Kucharova K, Stallcup WB.
    J Neuroinflammation; 2015 Sep 04; 12():161. PubMed ID: 26338007
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  • 4. Endothelial α6β4 integrin protects during experimental autoimmune encephalomyelitis-induced neuroinflammation by maintaining vascular integrity and tight junction protein expression.
    Welser JV, Halder SK, Kant R, Boroujerdi A, Milner R.
    J Neuroinflammation; 2017 Nov 09; 14(1):217. PubMed ID: 29121970
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  • 6. The response of NG2-expressing oligodendrocyte progenitors to demyelination in MOG-EAE and MS.
    Reynolds R, Dawson M, Papadopoulos D, Polito A, Di Bello IC, Pham-Dinh D, Levine J.
    J Neurocytol; 2002 Nov 09; 31(6-7):523-36. PubMed ID: 14501221
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  • 9. Lack of junctional adhesion molecule (JAM)-B ameliorates experimental autoimmune encephalomyelitis.
    Tietz S, Périnat T, Greene G, Enzmann G, Deutsch U, Adams R, Imhof B, Aurrand-Lions M, Engelhardt B.
    Brain Behav Immun; 2018 Oct 09; 73():3-20. PubMed ID: 29920328
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  • 10. Localization of claudin-3 in tight junctions of the blood-brain barrier is selectively lost during experimental autoimmune encephalomyelitis and human glioblastoma multiforme.
    Wolburg H, Wolburg-Buchholz K, Kraus J, Rascher-Eggstein G, Liebner S, Hamm S, Duffner F, Grote EH, Risau W, Engelhardt B.
    Acta Neuropathol; 2003 Jun 09; 105(6):586-92. PubMed ID: 12734665
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  • 11. The NG2 proteoglycan promotes oligodendrocyte progenitor proliferation and developmental myelination.
    Kucharova K, Stallcup WB.
    Neuroscience; 2010 Mar 10; 166(1):185-94. PubMed ID: 20006679
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  • 12. NG2 expressed by macrophages and oligodendrocyte precursor cells is dispensable in experimental autoimmune encephalomyelitis.
    Moransard M, Dann A, Staszewski O, Fontana A, Prinz M, Suter T.
    Brain; 2011 May 10; 134(Pt 5):1315-30. PubMed ID: 21596769
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  • 13. Visualizing Impairment of the Endothelial and Glial Barriers of the Neurovascular Unit during Experimental Autoimmune Encephalomyelitis In Vivo.
    Tietz SM, Engelhardt B.
    J Vis Exp; 2019 Mar 26; (145):. PubMed ID: 30985749
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  • 18. Reduced inflammation accompanies diminished myelin damage and repair in the NG2 null mouse spinal cord.
    Kucharova K, Chang Y, Boor A, Yong VW, Stallcup WB.
    J Neuroinflammation; 2011 Nov 13; 8():158. PubMed ID: 22078261
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  • 20. NG2-positive cells generate A2B5-positive oligodendrocyte precursor cells.
    Baracskay KL, Kidd GJ, Miller RH, Trapp BD.
    Glia; 2007 Aug 01; 55(10):1001-10. PubMed ID: 17503442
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