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Journal Abstract Search

591 related items for PubMed ID: 30876973

  • 1. Atrasentan alleviates high glucose-induced podocyte injury by the microRNA-21/forkhead box O1 axis.
    Wang J, Shen L, Hong H, Li J, Wang H, Li X.
    Eur J Pharmacol; 2019 Jun 05; 852():142-150. PubMed ID: 30876973
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  • 4. Protective effect of silencing Stat1 on high glucose-induced podocytes injury via Forkhead transcription factor O1-regulated the oxidative stress response.
    Wang H, Zhang Y, Xia F, Zhang W, Chen P, Yang G.
    BMC Mol Cell Biol; 2019 Jul 23; 20(1):27. PubMed ID: 31337338
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  • 8. MiR-770-5p facilitates podocyte apoptosis and inflammation in diabetic nephropathy by targeting TIMP3.
    Wang L, Li H.
    Biosci Rep; 2020 Apr 30; 40(4):. PubMed ID: 32309847
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  • 11. Astragalus polysaccharide attenuates diabetic nephropathy by reducing apoptosis and enhancing autophagy through activation of Sirt1/FoxO1 pathway.
    Xu Y, Xu C, Huang J, Xu C, Xiong Y.
    Int Urol Nephrol; 2024 Sep 30; 56(9):3067-3078. PubMed ID: 38653852
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  • 14. miR-15b-5p ameliorated high glucose-induced podocyte injury through repressing apoptosis, oxidative stress, and inflammatory responses by targeting Sema3A.
    Fu Y, Wang C, Zhang D, Chu X, Zhang Y, Li J.
    J Cell Physiol; 2019 Nov 30; 234(11):20869-20878. PubMed ID: 31025335
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  • 16. Overexpression of miR-34c inhibits high glucose-induced apoptosis in podocytes by targeting Notch signaling pathways.
    Liu XD, Zhang LY, Zhu TC, Zhang RF, Wang SL, Bao Y.
    Int J Clin Exp Pathol; 2015 Nov 30; 8(5):4525-34. PubMed ID: 26191142
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  • 17. MiR-874 alleviates renal injury and inflammatory response in diabetic nephropathy through targeting toll-like receptor-4.
    Yao T, Zha D, Gao P, Shui H, Wu X.
    J Cell Physiol; 2018 Jan 30; 234(1):871-879. PubMed ID: 30171701
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  • 18. Hsa_circ_0037128 aggravates high glucose-induced podocytes injury in diabetic nephropathy through mediating miR-31-5p/KLF9.
    Fang R, Cao X, Zhu Y, Chen Q.
    Autoimmunity; 2022 Jun 30; 55(4):254-263. PubMed ID: 35285770
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  • 19. Overexpression of FOXO1 ameliorates the podocyte epithelial-mesenchymal transition induced by high glucose in vitro and in vivo.
    Du M, Wang Q, Li W, Ma X, Wu L, Guo F, Zhao S, Huang F, Wang H, Qin G.
    Biochem Biophys Res Commun; 2016 Mar 18; 471(4):416-22. PubMed ID: 26902117
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  • 20. LncRNA CTBP1-AS2 alleviates high glucose-induced oxidative stress, ECM accumulation, and inflammation in diabetic nephropathy via miR-155-5p/FOXO1 axis.
    Wang G, Wu B, Zhang B, Wang K, Wang H.
    Biochem Biophys Res Commun; 2020 Nov 05; 532(2):308-314. PubMed ID: 32868076
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