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778 related items for PubMed ID: 31026418

  • 1. Receptor-Interacting Serine/Threonine-Protein Kinase 3 (RIPK3)-Mixed Lineage Kinase Domain-Like Protein (MLKL)-Mediated Necroptosis Contributes to Ischemia-Reperfusion Injury of Steatotic Livers.
    Ni HM, Chao X, Kaseff J, Deng F, Wang S, Shi YH, Li T, Ding WX, Jaeschke H.
    Am J Pathol; 2019 Jul; 189(7):1363-1374. PubMed ID: 31026418
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  • 2. The role of MLKL in Hepatic Ischemia-Reperfusion Injury of Alcoholic Steatotic Livers.
    Chen H, McKeen T, Chao X, Chen A, Deng F, Jaeschke H, Ding WX, Ni HM.
    Int J Biol Sci; 2022 Jul; 18(3):1096-1106. PubMed ID: 35173541
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  • 3. Inhibition of receptor-interacting protein kinase 1 improves experimental non-alcoholic fatty liver disease.
    Majdi A, Aoudjehane L, Ratziu V, Islam T, Afonso MB, Conti F, Mestiri T, Lagouge M, Foufelle F, Ballenghien F, Ledent T, Moldes M, Cadoret A, Fouassier L, Delaunay JL, Aït-Slimane T, Courtois G, Fève B, Scatton O, Prip-Buus C, Rodrigues CMP, Housset C, Gautheron J.
    J Hepatol; 2020 Apr; 72(4):627-635. PubMed ID: 31760070
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  • 7. Knockdown of RIPK1 Markedly Exacerbates Murine Immune-Mediated Liver Injury through Massive Apoptosis of Hepatocytes, Independent of Necroptosis and Inhibition of NF-κB.
    Suda J, Dara L, Yang L, Aghajan M, Song Y, Kaplowitz N, Liu ZX.
    J Immunol; 2016 Oct 15; 197(8):3120-3129. PubMed ID: 27605011
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  • 8. A cytosolic heat shock protein 90 and co-chaperone p23 complex activates RIPK3/MLKL during necroptosis of endothelial cells in acute respiratory distress syndrome.
    Yu X, Mao M, Liu X, Shen T, Li T, Yu H, Zhang J, Chen X, Zhao X, Zhu D.
    J Mol Med (Berl); 2020 Apr 15; 98(4):569-583. PubMed ID: 32072232
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  • 9. Phenytoin inhibits necroptosis.
    von Mässenhausen A, Tonnus W, Himmerkus N, Parmentier S, Saleh D, Rodriguez D, Ousingsawat J, Ang RL, Weinberg JM, Sanz AB, Ortiz A, Zierleyn A, Becker JU, Baratte B, Desban N, Bach S, Schiessl IM, Nogusa S, Balachandran S, Anders HJ, Ting AT, Bleich M, Degterev A, Kunzelmann K, Bornstein SR, Green DR, Hugo C, Linkermann A.
    Cell Death Dis; 2018 Mar 02; 9(3):359. PubMed ID: 29500402
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  • 12. Macrophage Notch1 inhibits TAK1 function and RIPK3-mediated hepatocyte necroptosis through activation of β-catenin signaling in liver ischemia and reperfusion injury.
    Xu D, Qu X, Tian Y, Jie Z, Xi Z, Xue F, Ma X, Zhu J, Xia Q.
    Cell Commun Signal; 2022 Sep 16; 20(1):144. PubMed ID: 36114543
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  • 15. The neurotoxicant PCB-95 by increasing the neuronal transcriptional repressor REST down-regulates caspase-8 and increases Ripk1, Ripk3 and MLKL expression determining necroptotic neuronal death.
    Guida N, Laudati G, Serani A, Mascolo L, Molinaro P, Montuori P, Di Renzo G, Canzoniero LMT, Formisano L.
    Biochem Pharmacol; 2017 Oct 15; 142():229-241. PubMed ID: 28676433
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  • 16. Does necroptosis have a crucial role in hepatic ischemia-reperfusion injury?
    Saeed WK, Jun DW, Jang K, Chae YJ, Lee JS, Kang HT.
    PLoS One; 2017 Oct 15; 12(9):e0184752. PubMed ID: 28957350
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  • 17. Necroptosis signalling is tuned by phosphorylation of MLKL residues outside the pseudokinase domain activation loop.
    Tanzer MC, Tripaydonis A, Webb AI, Young SN, Varghese LN, Hall C, Alexander WS, Hildebrand JM, Silke J, Murphy JM.
    Biochem J; 2015 Oct 15; 471(2):255-65. PubMed ID: 26283547
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  • 18. Loss of MLKL (Mixed Lineage Kinase Domain-Like Protein) Decreases Necrotic Core but Increases Macrophage Lipid Accumulation in Atherosclerosis.
    Rasheed A, Robichaud S, Nguyen MA, Geoffrion M, Wyatt H, Cottee ML, Dennison T, Pietrangelo A, Lee R, Lagace TA, Ouimet M, Rayner KJ.
    Arterioscler Thromb Vasc Biol; 2020 May 15; 40(5):1155-1167. PubMed ID: 32212851
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  • 19. Decrease in fat de novo synthesis and chemokine ligand expression in non-alcoholic fatty liver disease caused by inhibition of mixed lineage kinase domain-like pseudokinase.
    Saeed WK, Jun DW, Jang K, Oh JH, Chae YJ, Lee JS, Koh DH, Kang HT.
    J Gastroenterol Hepatol; 2019 Dec 15; 34(12):2206-2218. PubMed ID: 31132314
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  • 20. Receptor-Interacting Protein Kinases 1 and 3, and Mixed Lineage Kinase Domain-Like Protein Are Activated by Sublytic Complement and Participate in Complement-Dependent Cytotoxicity.
    Lusthaus M, Mazkereth N, Donin N, Fishelson Z.
    Front Immunol; 2018 Dec 15; 9():306. PubMed ID: 29527209
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