These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

246 related items for PubMed ID: 31039417

  • 1. Intrauterine programming of the glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis mediates glomerulosclerosis in female adult offspring rats induced by prenatal ethanol exposure.
    He H, Xiong Y, Li B, Zhu Y, Chen H, Ao Y, Wang H.
    Toxicol Lett; 2019 Sep 01; 311():17-26. PubMed ID: 31039417
    [Abstract] [Full Text] [Related]

  • 2. Prenatal ethanol exposure increased the susceptibility of adult offspring rats to glomerulosclerosis.
    Chen H, Zhu Y, Zhao X, He H, Luo J, Ao Y, Wang H.
    Toxicol Lett; 2020 Mar 15; 321():44-53. PubMed ID: 31811911
    [Abstract] [Full Text] [Related]

  • 3. Glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis programming mediated hepatic lipid-metabolic in offspring caused by prenatal ethanol exposure.
    Hu W, Yuan C, Luo H, Hu S, Shen L, Chen L, Xu D, Wang H.
    Toxicol Lett; 2020 Oct 01; 331():167-177. PubMed ID: 32535229
    [Abstract] [Full Text] [Related]

  • 4. Prenatal ethanol exposure-induced adrenal developmental abnormality of male offspring rats and its possible intrauterine programming mechanisms.
    Huang H, He Z, Zhu C, Liu L, Kou H, Shen L, Wang H.
    Toxicol Appl Pharmacol; 2015 Oct 01; 288(1):84-94. PubMed ID: 26188107
    [Abstract] [Full Text] [Related]

  • 5. Prenatal ethanol exposure programs an increased susceptibility of non-alcoholic fatty liver disease in female adult offspring rats.
    Shen L, Liu Z, Gong J, Zhang L, Wang L, Magdalou J, Chen L, Wang H.
    Toxicol Appl Pharmacol; 2014 Jan 15; 274(2):263-73. PubMed ID: 24275070
    [Abstract] [Full Text] [Related]

  • 6. The expressional disorder of the renal RAS mediates nephrotic syndrome of male rat offspring induced by prenatal ethanol exposure.
    Zhu Y, Zuo N, Li B, Xiong Y, Chen H, He H, Sun Z, Hu S, Cheng H, Ao Y, Wang H.
    Toxicology; 2018 May 01; 400-401():9-19. PubMed ID: 29548890
    [Abstract] [Full Text] [Related]

  • 7. Glucocorticoid programming mechanism for hypercholesterolemia in prenatal ethanol-exposed adult offspring rats.
    Hu S, Qin J, Zhou J, Magdalou J, Chen L, Xu D, Wang H.
    Toxicol Appl Pharmacol; 2019 Jul 15; 375():46-56. PubMed ID: 31075344
    [Abstract] [Full Text] [Related]

  • 8. Low functional programming of renal AT2R mediates the developmental origin of glomerulosclerosis in adult offspring induced by prenatal caffeine exposure.
    Ao Y, Sun Z, Hu S, Zuo N, Li B, Yang S, Xia L, Wu Y, Wang L, He Z, Wang H.
    Toxicol Appl Pharmacol; 2015 Sep 01; 287(2):128-138. PubMed ID: 25986755
    [Abstract] [Full Text] [Related]

  • 9. Glucocorticoid-activation system mediated glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis programming alteration of adrenal dysfunction induced by prenatal caffeine exposure.
    He Z, Zhang J, Huang H, Yuan C, Zhu C, Magdalou J, Wang H.
    Toxicol Lett; 2019 Mar 01; 302():7-17. PubMed ID: 30528684
    [Abstract] [Full Text] [Related]

  • 10. Prenatal ethanol exposure induces the osteoarthritis-like phenotype in female adult offspring rats with a post-weaning high-fat diet and its intrauterine programming mechanisms of cholesterol metabolism.
    Ni Q, Wang L, Wu Y, Shen L, Qin J, Liu Y, Magdalou J, Chen L, Wang H.
    Toxicol Lett; 2015 Oct 14; 238(2):117-25. PubMed ID: 26220516
    [Abstract] [Full Text] [Related]

  • 11. Age-Characteristic Changes of Glucose Metabolism, Pancreatic Morphology and Function in Male Offspring Rats Induced by Prenatal Ethanol Exposure.
    Xiao D, Kou H, Gui S, Ji Z, Guo Y, Wu Y, Wang H.
    Front Endocrinol (Lausanne); 2019 Oct 14; 10():34. PubMed ID: 30778335
    [Abstract] [Full Text] [Related]

  • 12. Decreased H3K9ac level of AT2R mediates the developmental origin of glomerulosclerosis induced by prenatal dexamethasone exposure in male offspring rats.
    Li B, Zhu Y, Chen H, Gao H, He H, Zuo N, Pei L, Xie W, Chen L, Ao Y, Wang H.
    Toxicology; 2019 Jan 01; 411():32-42. PubMed ID: 30359671
    [Abstract] [Full Text] [Related]

  • 13. Prenatal caffeine exposure induces liver developmental dysfunction in offspring rats.
    He B, Wen Y, Hu S, Wang G, Hu W, Magdalou J, Chen L, Wang H.
    J Endocrinol; 2019 Jul 26; 242(3):211-226. PubMed ID: 31340202
    [Abstract] [Full Text] [Related]

  • 14. Prenatal ethanol exposure-induced a low level of foetal blood cholesterol and its mechanism of IGF1-related placental cholesterol transport dysfunction.
    Zhang G, Zhou J, Huang W, Fang M, Yu L, Wang H, Zhang Y.
    Toxicology; 2019 Aug 01; 424():152237. PubMed ID: 31226463
    [Abstract] [Full Text] [Related]

  • 15. Prenatal ethanol exposure increases osteoarthritis susceptibility in female rat offspring by programming a low-functioning IGF-1 signaling pathway.
    Ni Q, Tan Y, Zhang X, Luo H, Deng Y, Magdalou J, Chen L, Wang H.
    Sci Rep; 2015 Oct 05; 5():14711. PubMed ID: 26434683
    [Abstract] [Full Text] [Related]

  • 16. Intergenerational genetic programming mechanism and sex differences of the adrenal corticosterone synthesis dysfunction in offspring induced by prenatal ethanol exposure.
    Cao J, Chen Y, Xia X, Qu H, Ao Y, Wang H.
    Toxicol Lett; 2021 Oct 15; 351():78-88. PubMed ID: 34454011
    [Abstract] [Full Text] [Related]

  • 17. Prenatal ethanol exposure and changes in fetal neuroendocrine metabolic programming.
    Liu L, Wen Y, Ni Q, Chen L, Wang H.
    Biol Res; 2023 Nov 17; 56(1):61. PubMed ID: 37978540
    [Abstract] [Full Text] [Related]

  • 18. Effects of prenatal nicotine exposure on hepatic glucose and lipid metabolism in offspring rats and its hereditability.
    Hu W, Wang G, He B, Hu S, Luo H, Wen Y, Chen L, Wang H.
    Toxicology; 2020 Feb 28; 432():152378. PubMed ID: 31972234
    [Abstract] [Full Text] [Related]

  • 19. IGF1/MAPK/ERK signaling pathway-mediated programming alterations of adrenal cortex cell proliferation by prenatal caffeine exposure in male offspring rats.
    Chen G, Yuan C, Duan F, Liu Y, Zhang J, He Z, Huang H, He C, Wang H.
    Toxicol Appl Pharmacol; 2018 Feb 15; 341():64-76. PubMed ID: 29343424
    [Abstract] [Full Text] [Related]

  • 20. Autophagy as a compensation mechanism participates in ethanol-induced fetal adrenal dysfunction in female rats.
    Huang H, Liu L, Li J, Zhu C, Xie X, Ao Y, Wang H.
    Toxicol Appl Pharmacol; 2018 Apr 15; 345():36-47. PubMed ID: 29524503
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 13.