These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


PUBMED FOR HANDHELDS

Journal Abstract Search


164 related items for PubMed ID: 31075344

  • 1. Glucocorticoid programming mechanism for hypercholesterolemia in prenatal ethanol-exposed adult offspring rats.
    Hu S, Qin J, Zhou J, Magdalou J, Chen L, Xu D, Wang H.
    Toxicol Appl Pharmacol; 2019 Jul 15; 375():46-56. PubMed ID: 31075344
    [Abstract] [Full Text] [Related]

  • 2. Glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis programming mediated hepatic lipid-metabolic in offspring caused by prenatal ethanol exposure.
    Hu W, Yuan C, Luo H, Hu S, Shen L, Chen L, Xu D, Wang H.
    Toxicol Lett; 2020 Oct 01; 331():167-177. PubMed ID: 32535229
    [Abstract] [Full Text] [Related]

  • 3. Two intrauterine programming mechanisms of adult hypercholesterolemia induced by prenatal nicotine exposure in male offspring rats.
    Zhou J, Zhu C, Luo H, Shen L, Gong J, Wu Y, Magdalou J, Chen L, Guo Y, Wang H.
    FASEB J; 2019 Jan 01; 33(1):1110-1123. PubMed ID: 30113880
    [Abstract] [Full Text] [Related]

  • 4. Intrauterine programming mechanism for hypercholesterolemia in prenatal caffeine-exposed female adult rat offspring.
    Xu D, Luo HW, Hu W, Hu SW, Yuan C, Wang GH, Zhang L, Yu H, Magdalou J, Chen LB, Wang H.
    FASEB J; 2018 Oct 01; 32(10):5563-5576. PubMed ID: 29718709
    [Abstract] [Full Text] [Related]

  • 5. Caffeine programs hepatic SIRT1-related cholesterol synthesis and hypercholesterolemia via A2AR/cAMP/PKA pathway in adult male offspring rats.
    Hu S, Liu K, Luo H, Xu D, Chen L, Zhang L, Wang H.
    Toxicology; 2019 Apr 15; 418():11-21. PubMed ID: 30825513
    [Abstract] [Full Text] [Related]

  • 6. Effects and Interactions of Prenatal Ethanol Exposure, a Post-Weaning High-Fat Diet and Gender on Adult Hypercholesterolemia Occurrence in Offspring Rats.
    Qi Y, Luo H, Hu S, Wu Y, Magdalou J, Chen L, Wang H.
    Cell Physiol Biochem; 2017 Apr 15; 44(2):657-670. PubMed ID: 29169169
    [Abstract] [Full Text] [Related]

  • 7. Intrauterine programming of the glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis mediates glomerulosclerosis in female adult offspring rats induced by prenatal ethanol exposure.
    He H, Xiong Y, Li B, Zhu Y, Chen H, Ao Y, Wang H.
    Toxicol Lett; 2019 Sep 01; 311():17-26. PubMed ID: 31039417
    [Abstract] [Full Text] [Related]

  • 8. Intergenerational genetic programming mechanism and sex differences of the adrenal corticosterone synthesis dysfunction in offspring induced by prenatal ethanol exposure.
    Cao J, Chen Y, Xia X, Qu H, Ao Y, Wang H.
    Toxicol Lett; 2021 Oct 15; 351():78-88. PubMed ID: 34454011
    [Abstract] [Full Text] [Related]

  • 9. Prenatal ethanol exposure-induced a low level of foetal blood cholesterol and its mechanism of IGF1-related placental cholesterol transport dysfunction.
    Zhang G, Zhou J, Huang W, Fang M, Yu L, Wang H, Zhang Y.
    Toxicology; 2019 Aug 01; 424():152237. PubMed ID: 31226463
    [Abstract] [Full Text] [Related]

  • 10. Prenatal ethanol exposure programs an increased susceptibility of non-alcoholic fatty liver disease in female adult offspring rats.
    Shen L, Liu Z, Gong J, Zhang L, Wang L, Magdalou J, Chen L, Wang H.
    Toxicol Appl Pharmacol; 2014 Jan 15; 274(2):263-73. PubMed ID: 24275070
    [Abstract] [Full Text] [Related]

  • 11. Prenatal ethanol exposure increased the susceptibility of adult offspring rats to glomerulosclerosis.
    Chen H, Zhu Y, Zhao X, He H, Luo J, Ao Y, Wang H.
    Toxicol Lett; 2020 Mar 15; 321():44-53. PubMed ID: 31811911
    [Abstract] [Full Text] [Related]

  • 12. Prenatal ethanol exposure-induced adrenal developmental abnormality of male offspring rats and its possible intrauterine programming mechanisms.
    Huang H, He Z, Zhu C, Liu L, Kou H, Shen L, Wang H.
    Toxicol Appl Pharmacol; 2015 Oct 01; 288(1):84-94. PubMed ID: 26188107
    [Abstract] [Full Text] [Related]

  • 13. Influencing factors, underlying mechanism and interactions affecting hypercholesterolemia in adult offspring with caffeine exposure during pregnancy.
    Guo Y, Luo H, Wu Y, Magdalou J, Chen L, Wang H.
    Reprod Toxicol; 2018 Aug 01; 79():47-56. PubMed ID: 29800656
    [Abstract] [Full Text] [Related]

  • 14. Prenatal ethanol exposure induces the osteoarthritis-like phenotype in female adult offspring rats with a post-weaning high-fat diet and its intrauterine programming mechanisms of cholesterol metabolism.
    Ni Q, Wang L, Wu Y, Shen L, Qin J, Liu Y, Magdalou J, Chen L, Wang H.
    Toxicol Lett; 2015 Oct 14; 238(2):117-25. PubMed ID: 26220516
    [Abstract] [Full Text] [Related]

  • 15. MiR-133a-3p/Sirt1 epigenetic programming mediates hypercholesterolemia susceptibility in female offspring induced by prenatal dexamethasone exposure.
    Li X, Hu W, Li L, Chen Z, Jiang T, Zhang D, Liu K, Wang H.
    Biochem Pharmacol; 2022 Dec 14; 206():115306. PubMed ID: 36326533
    [Abstract] [Full Text] [Related]

  • 16. Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats.
    Liu M, Zhang Q, Pei L, Zou Y, Chen G, Wang H.
    Epigenetics; 2019 Mar 14; 14(3):245-259. PubMed ID: 30821590
    [Abstract] [Full Text] [Related]

  • 17. The expressional disorder of the renal RAS mediates nephrotic syndrome of male rat offspring induced by prenatal ethanol exposure.
    Zhu Y, Zuo N, Li B, Xiong Y, Chen H, He H, Sun Z, Hu S, Cheng H, Ao Y, Wang H.
    Toxicology; 2018 May 01; 400-401():9-19. PubMed ID: 29548890
    [Abstract] [Full Text] [Related]

  • 18. Prenatal ethanol exposure induced disorder of hypothalamic-pituitary-adrenal axis-associated neuroendocrine metabolic programming alteration and dysfunction of glucose and lipid metabolism in 40-week-old female offspring rats.
    Xia L, Jiao Z, Pei L, Yuan C, Zhao Y, Guo Y, Wang H.
    Reprod Toxicol; 2020 Jun 01; 94():48-54. PubMed ID: 32333956
    [Abstract] [Full Text] [Related]

  • 19. Decreased H3K9ac level of KLF4 mediates podocyte developmental toxicity induced by prenatal caffeine exposure in male offspring rats.
    Zhu Y, Chen H, Zhao X, Li B, He H, Cheng H, Wang H, Ao Y.
    Toxicol Lett; 2019 Oct 10; 314():63-74. PubMed ID: 31306741
    [Abstract] [Full Text] [Related]

  • 20. miR-148a/LDLR mediates hypercholesterolemia induced by prenatal dexamethasone exposure in male offspring rats.
    Li L, Hu W, Liu K, Zhang D, Liu M, Li X, Wang H.
    Toxicol Appl Pharmacol; 2020 May 15; 395():114979. PubMed ID: 32234517
    [Abstract] [Full Text] [Related]


    Page: [Next] [New Search]
    of 9.