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Journal Abstract Search

167 related items for PubMed ID: 31092336

  • 21. Oxidative stress-induced JNK1/2 activation triggers proapoptotic signaling and apoptosis that leads to diabetic embryopathy.
    Li X, Weng H, Xu C, Reece EA, Yang P.
    Diabetes; 2012 Aug; 61(8):2084-92. PubMed ID: 22688338
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  • 22. Dominant negative FADD dissipates the proapoptotic signalosome of the unfolded protein response in diabetic embryopathy.
    Wang F, Weng H, Quon MJ, Yu J, Wang JY, Hueber AO, Yang P.
    Am J Physiol Endocrinol Metab; 2015 Nov 15; 309(10):E861-73. PubMed ID: 26419589
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  • 24. Polymorphic susceptibility to the molecular causes of neural tube defects during diabetic embryopathy.
    Pani L, Horal M, Loeken MR.
    Diabetes; 2002 Sep 15; 51(9):2871-4. PubMed ID: 12196484
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  • 25. Punicalagin exerts protective effect against high glucose-induced cellular stress and neural tube defects.
    Zhong J, Reece EA, Yang P.
    Biochem Biophys Res Commun; 2015 Nov 13; 467(2):179-84. PubMed ID: 26453010
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  • 26. Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress.
    Li R, Chase M, Jung SK, Smith PJ, Loeken MR.
    Am J Physiol Endocrinol Metab; 2005 Oct 13; 289(4):E591-9. PubMed ID: 15928021
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  • 30. Mechanisms of Congenital Malformations in Pregnancies with Pre-existing Diabetes.
    Loeken MR.
    Curr Diab Rep; 2020 Sep 12; 20(10):54. PubMed ID: 32918152
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  • 32. [Expression of Pax3 and Cx43 in NTD embryos induced by hyperglycemia].
    Mao DW, Zhang YJ, Li QM, Li SR.
    Zhonghua Yi Xue Za Zhi; 2003 Apr 10; 83(7):593-7. PubMed ID: 12887752
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  • 33. The essential role of protein kinase Cδ in diabetes-induced neural tube defects.
    Cao Y, Zhao Z, Eckert RL, Reece EA.
    J Matern Fetal Neonatal Med; 2012 Oct 10; 25(10):2020-4. PubMed ID: 22463764
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  • 34. Diabetic embryopathy in C57BL/6J mice. Altered fetal sex ratio and impact of the splotch allele.
    Machado AF, Zimmerman EF, Hovland DN, Weiss R, Collins MD.
    Diabetes; 2001 May 10; 50(5):1193-9. PubMed ID: 11334426
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  • 35. Experimental mechanisms of diabetic embryopathy and strategies for developing therapeutic interventions.
    Zhao Z, Reece EA.
    J Soc Gynecol Investig; 2005 Dec 10; 12(8):549-57. PubMed ID: 16325743
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  • 36. Recent studies on neural tube defects in embryos of diabetic pregnancy: an overview.
    Dheen ST, Tay SS, Boran J, Ting LW, Kumar SD, Fu J, Ling EA.
    Curr Med Chem; 2009 Dec 10; 16(18):2345-54. PubMed ID: 19519395
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  • 37. Wnt signaling in caudal dysgenesis and diabetic embryopathy.
    Pavlinkova G, Salbaum JM, Kappen C.
    Birth Defects Res A Clin Mol Teratol; 2008 Oct 10; 82(10):710-9. PubMed ID: 18937363
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  • 38. Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects.
    Morgan SC, Relaix F, Sandell LL, Loeken MR.
    Birth Defects Res A Clin Mol Teratol; 2008 Jun 10; 82(6):453-63. PubMed ID: 18435457
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  • 39. SOD1 suppresses maternal hyperglycemia-increased iNOS expression and consequent nitrosative stress in diabetic embryopathy.
    Weng H, Li X, Reece EA, Yang P.
    Am J Obstet Gynecol; 2012 May 10; 206(5):448.e1-7. PubMed ID: 22425406
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  • 40. Protein kinase Cβ2 inhibition reduces hyperglycemia-induced neural tube defects through suppression of a caspase 8-triggered apoptotic pathway.
    Cao Y, Zhao Z, Eckert RL, Reece EA.
    Am J Obstet Gynecol; 2011 Mar 10; 204(3):226.e1-5. PubMed ID: 21376163
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