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156 related items for PubMed ID: 3110457
1. Use-dependent blocking action of newly developed lidocaine-analogs on maximum rate of rise of action potentials in guinea pig papillary muscle. Toyama J, Kodama I, Kusunoki T, Ishihara T, Hattori Y, Yamada K. Jpn Heart J; 1987 Mar; 28(2):273-85. PubMed ID: 3110457 [Abstract] [Full Text] [Related]
2. Effects of tocainide and lidocaine on the transmembrane action potentials as related to external potassium and calcium concentrations in guinea-pig papillary muscles. Oshita S, Sada H, Kojima M, Ban T. Naunyn Schmiedebergs Arch Pharmacol; 1980 Oct; 314(1):67-82. PubMed ID: 6777703 [Abstract] [Full Text] [Related]
4. Modification of the frequency- and voltage-dependent effects of quinidine when administered in combination with tocainide in canine Purkinje fibers. Valois M, Sasyniuk BI. Circulation; 1987 Aug; 76(2):427-41. PubMed ID: 2440617 [Abstract] [Full Text] [Related]
6. Rate-dependent depression of maximal rate of depolarization in guinea pig papillary muscle action potentials by changrolin. Kuang Y, Liu TP. Zhongguo Yao Li Xue Bao; 1990 May; 11(3):225-9. PubMed ID: 2087995 [Abstract] [Full Text] [Related]
7. Block of cardiac sodium channels by antiarrhythmic drugs. Kodama I, Toyama J. Jpn Circ J; 1988 Mar; 52(3):231-7. PubMed ID: 2453687 [Abstract] [Full Text] [Related]
8. Electrophysiological and antiarrhythmic actions of tocainide in isolated heart preparations of the guinea pig. Borchard U, Hafner D, Ewerbeck S. Arzneimittelforschung; 1985 Mar; 35(9):1367-74. PubMed ID: 3936506 [Abstract] [Full Text] [Related]
9. Block of activated and inactivated sodium channels by class-I antiarrhythmic drugs studied by using the maximum upstroke velocity (Vmax) of action potential in guinea-pig cardiac muscles. Kodama I, Toyama J, Takanaka C, Yamada K. J Mol Cell Cardiol; 1987 Apr; 19(4):367-77. PubMed ID: 2441073 [Abstract] [Full Text] [Related]
11. Electrophysiologic interactions between mexiletine-quinidine and mexiletine-ropitoin in guinea pig papillary muscle. Valenzuela C, Sanchez-Chapula J. J Cardiovasc Pharmacol; 1989 Nov; 14(5):783-9. PubMed ID: 2481194 [Abstract] [Full Text] [Related]
12. Interval-dependent effects of small antiarrhythmic drugs on excitability of guinea-pig myocardium. Courtney KR. J Mol Cell Cardiol; 1980 Nov; 12(11):1273-86. PubMed ID: 6777501 [No Abstract] [Full Text] [Related]
13. Frequency dependent effects of tocainide, quinidine, and flecainide on conduction as reflected in the rise time of the monophasic action potential in the isolated guinea pig heart. Duker BG. Cardiovasc Res; 1991 Mar; 25(3):217-22. PubMed ID: 1903081 [Abstract] [Full Text] [Related]
14. Nicorandil shortens action potential duration and antagonises the reduction of Vmax by lidocaine but not by disopyramide in guinea-pig papillary muscles. Kojima M, Ban T. Naunyn Schmiedebergs Arch Pharmacol; 1988 Feb; 337(2):203-12. PubMed ID: 2966899 [Abstract] [Full Text] [Related]
15. Electrophysiological effects of CI-980, a tubulin binding agent, on guinea-pig papillary muscles. Pérez O, Valenzuela C, Delpón E, Tamargo J. Br J Pharmacol; 1997 Jan; 120(2):187-92. PubMed ID: 9117108 [Abstract] [Full Text] [Related]
16. Effects of combined use of class I antiarrhythmic agents on Vmax of guinea-pig ventricular muscles. Toyama J, Kawamura T, Kodama I. Cardiovasc Drugs Ther; 1991 Aug; 5 Suppl 4():801-4. PubMed ID: 1931757 [Abstract] [Full Text] [Related]