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177 related items for PubMed ID: 31269481

  • 1. Association of Urinary Plasminogen-Plasmin with Edema and Epithelial Sodium Channel Activation in Patients with Nephrotic Syndrome.
    Chen JL, Wang L, Yao XM, Zang YJ, Wang Y, Li ZJ, Pearce D, Wang H.
    Am J Nephrol; 2019; 50(2):92-104. PubMed ID: 31269481
    [Abstract] [Full Text] [Related]

  • 2. Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC.
    Andersen RF, Buhl KB, Jensen BL, Svenningsen P, Friis UG, Jespersen B, Rittig S.
    Pediatr Nephrol; 2013 Aug; 28(8):1227-34. PubMed ID: 23503750
    [Abstract] [Full Text] [Related]

  • 3. Association of Plasminuria with Overhydration in Patients with CKD.
    Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Haerteis S, Korbmacher C, Heyne N, Peter A, Häring HU, Artunc F.
    Clin J Am Soc Nephrol; 2016 May 06; 11(5):761-769. PubMed ID: 26933188
    [Abstract] [Full Text] [Related]

  • 4. Aberrant glomerular filtration of urokinase-type plasminogen activator in nephrotic syndrome leads to amiloride-sensitive plasminogen activation in urine.
    Stæhr M, Buhl KB, Andersen RF, Svenningsen P, Nielsen F, Hinrichs GR, Bistrup C, Jensen BL.
    Am J Physiol Renal Physiol; 2015 Aug 01; 309(3):F235-41. PubMed ID: 25972510
    [Abstract] [Full Text] [Related]

  • 5. Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.
    Bohnert BN, Daiminger S, Wörn M, Sure F, Staudner T, Ilyaskin AV, Batbouta F, Janessa A, Schneider JC, Essigke D, Kanse S, Haerteis S, Korbmacher C, Artunc F.
    Acta Physiol (Oxf); 2019 Dec 01; 227(4):e13286. PubMed ID: 31006168
    [Abstract] [Full Text] [Related]

  • 6. Mechanisms of renal NaCl retention in proteinuric disease.
    Svenningsen P, Friis UG, Versland JB, Buhl KB, Møller Frederiksen B, Andersen H, Zachar RM, Bistrup C, Skøtt O, Jørgensen JS, Andersen RF, Jensen BL.
    Acta Physiol (Oxf); 2013 Mar 01; 207(3):536-45. PubMed ID: 23216619
    [Abstract] [Full Text] [Related]

  • 7. Plasmin in nephrotic urine activates the epithelial sodium channel.
    Svenningsen P, Bistrup C, Friis UG, Bertog M, Haerteis S, Krueger B, Stubbe J, Jensen ON, Thiesson HC, Uhrenholt TR, Jespersen B, Jensen BL, Korbmacher C, Skøtt O.
    J Am Soc Nephrol; 2009 Feb 01; 20(2):299-310. PubMed ID: 19073825
    [Abstract] [Full Text] [Related]

  • 8. Plasmin in urine from patients with type 2 diabetes and treatment-resistant hypertension activates ENaC in vitro.
    Buhl KB, Oxlund CS, Friis UG, Svenningsen P, Bistrup C, Jacobsen IA, Jensen BL.
    J Hypertens; 2014 Aug 01; 32(8):1672-7; discussion 1677. PubMed ID: 24805959
    [Abstract] [Full Text] [Related]

  • 9. Urinary plasmin activates collecting duct ENaC current in preeclampsia.
    Buhl KB, Friis UG, Svenningsen P, Gulaveerasingam A, Ovesen P, Frederiksen-Møller B, Jespersen B, Bistrup C, Jensen BL.
    Hypertension; 2012 Nov 01; 60(5):1346-51. PubMed ID: 22987920
    [Abstract] [Full Text] [Related]

  • 10. Nephrotic syndrome is associated with increased plasma K+ concentration, intestinal K+ losses, and attenuated urinary K+ excretion: a study in rats and humans.
    Ydegaard R, Svenningsen P, Bistrup C, Andersen RF, Stubbe J, Buhl KB, Marcussen N, Hinrichs GR, Iraqi H, Zamani R, Dimke H, Jensen BL.
    Am J Physiol Renal Physiol; 2019 Dec 01; 317(6):F1549-F1562. PubMed ID: 31566427
    [Abstract] [Full Text] [Related]

  • 11. Perspectives on edema in childhood nephrotic syndrome.
    Teoh CW, Robinson LA, Noone D.
    Am J Physiol Renal Physiol; 2015 Oct 01; 309(7):F575-82. PubMed ID: 26290369
    [Abstract] [Full Text] [Related]

  • 12. Is urinary excretion of plasminogen associated with development of pre-eclampsia? An observational, explorative case-control study.
    Nielsen LH, Kronborg C, Vittinghus E, Kitlen G, Jensen BL, Knudsen UB, Ovesen PG.
    BMJ Open; 2019 Jun 21; 9(6):e026489. PubMed ID: 31230006
    [Abstract] [Full Text] [Related]

  • 13. Over- or underfill: not all nephrotic states are created equal.
    Bockenhauer D.
    Pediatr Nephrol; 2013 Aug 21; 28(8):1153-6. PubMed ID: 23529637
    [Abstract] [Full Text] [Related]

  • 14. Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.
    Bohnert BN, Menacher M, Janessa A, Wörn M, Schork A, Daiminger S, Kalbacher H, Häring HU, Daniel C, Amann K, Sure F, Bertog M, Haerteis S, Korbmacher C, Artunc F.
    Kidney Int; 2018 Jan 21; 93(1):159-172. PubMed ID: 29042083
    [Abstract] [Full Text] [Related]

  • 15. Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice.
    Hinrichs GR, Weyer K, Friis UG, Svenningsen P, Lund IK, Nielsen R, Mollet G, Antignac C, Bistrup C, Jensen BL, Birn H.
    Acta Physiol (Oxf); 2019 Dec 21; 227(4):e13362. PubMed ID: 31423748
    [Abstract] [Full Text] [Related]

  • 16. Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice.
    Haerteis S, Schork A, Dörffel T, Bohnert BN, Nacken R, Wörn M, Xiao M, Essigke D, Janessa A, Schmaier AH, Feener EP, Häring HU, Bertog M, Korbmacher C, Artunc F.
    Acta Physiol (Oxf); 2018 Sep 21; 224(1):e13060. PubMed ID: 29489063
    [Abstract] [Full Text] [Related]

  • 17. Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome.
    Xiao M, Bohnert BN, Aypek H, Kretz O, Grahammer F, Aukschun U, Wörn M, Janessa A, Essigke D, Daniel C, Amann K, Huber TB, Plow EF, Birkenfeld AL, Artunc F.
    Acta Physiol (Oxf); 2021 Jan 21; 231(1):e13512. PubMed ID: 32455507
    [Abstract] [Full Text] [Related]

  • 18. Proteinuric diseases with sodium retention: Is plasmin the link?
    Svenningsen P, Skøtt O, Jensen BL.
    Clin Exp Pharmacol Physiol; 2012 Jan 21; 39(1):117-24. PubMed ID: 21466573
    [Abstract] [Full Text] [Related]

  • 19. Diabetic nephropathy is associated with increased urine excretion of proteases plasmin, prostasin and urokinase and activation of amiloride-sensitive current in collecting duct cells.
    Andersen H, Friis UG, Hansen PB, Svenningsen P, Henriksen JE, Jensen BL.
    Nephrol Dial Transplant; 2015 May 21; 30(5):781-9. PubMed ID: 25609736
    [Abstract] [Full Text] [Related]

  • 20. Time-course of sodium transport along the nephron in nephrotic syndrome: The role of potassium.
    Dizin E, Olivier V, Maire C, Komarynets O, Sassi A, Roth I, Loffing J, de Seigneux S, Maillard M, Rutkowski JM, Edwards A, Feraille E.
    FASEB J; 2020 Feb 21; 34(2):2408-2424. PubMed ID: 31908015
    [Abstract] [Full Text] [Related]

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