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Journal Abstract Search

133 related items for PubMed ID: 3145581

  • 1.
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  • 2. Short-term effects of dopamine-depleting brain lesions on spontaneous activity of striatal neurons: relation to local dopamine concentration and behavior.
    Orr WB, Gardiner TW, Stricker EM, Zigmond MJ, Berger TW.
    Brain Res; 1986 Jun 18; 376(1):20-8. PubMed ID: 3087583
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  • 5. Fetal ventral mesencephalic grafts functionally reduce the dopamine D2 receptor supersensitivity in partially dopamine reinnervated host striatum.
    Strömberg I, Kehr J, Andbjer B, Fuxe K.
    Exp Neurol; 2000 Jul 18; 164(1):154-65. PubMed ID: 10877926
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  • 7. The effects of dopamine-depleting brain lesions on the electrophysiological activity of rat substantia nigra dopamine neurons.
    Hollerman JR, Grace AA.
    Brain Res; 1990 Nov 19; 533(2):203-12. PubMed ID: 2126975
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  • 8. Single administration of 1-benzyl-1,2,3,4-tetrahydroisoquinoline increases the extracellular concentration of dopamine in rat striatum.
    Katagiri N, Abe K, Kitabatake M, Utsunomiya I, Horiguchi Y, Hoshi K, Taguchi K.
    Neuroscience; 2009 Jun 02; 160(4):820-8. PubMed ID: 19285542
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  • 9. Stress-induced neurological impairments in an animal model of parkinsonism.
    Snyder AM, Stricker EM, Zigmond MJ.
    Ann Neurol; 1985 Nov 02; 18(5):544-51. PubMed ID: 3935041
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  • 10. Deprenyl (selegiline), a selective MAO-B inhibitor with active metabolites; effects on locomotor activity, dopaminergic neurotransmission and firing rate of nigral dopamine neurons.
    Engberg G, Elebring T, Nissbrandt H.
    J Pharmacol Exp Ther; 1991 Nov 02; 259(2):841-7. PubMed ID: 1658311
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  • 11. Long-term effects of dopamine-depleting brain lesions on spontaneous activity of type II striatal neurons: relation to behavioral recovery.
    Nisenbaum ES, Stricker EM, Zigmond MJ, Berger TW.
    Brain Res; 1986 Nov 29; 398(2):221-30. PubMed ID: 3099976
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  • 12. Limbic pallidal adaptations following long-term cessation of dopaminergic transmission: lack of upregulation of dopamine receptor function.
    Heidenreich BA, Mitrovic I, Battaglia G, Napier TC.
    Exp Neurol; 2004 Apr 29; 186(2):145-57. PubMed ID: 15026253
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  • 14. Dopamine depletion causes fragmented clustering of neurons in the sensorimotor striatum: evidence of lasting reorganization of corticostriatal input.
    Cho J, Duke D, Manzino L, Sonsalla PK, West MO.
    J Comp Neurol; 2002 Oct 07; 452(1):24-37. PubMed ID: 12205707
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  • 16. Lesions of the dopaminergic nigrostriatal system in neonatal rats: effects on the electrophysiological activity of striatal neurons recorded during adulthood.
    Onn SP, Balzer JR, Sidney JP, Stricker EM, Zigmond MJ, Berger TW.
    Brain Res; 1990 Jun 04; 518(1-2):274-8. PubMed ID: 2117988
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  • 17. Differential behavioural effects after subtotal depletions of dopamine or noradrenaline in the ventral striatum.
    Schwarting RK, Carey RJ.
    Funct Neurol; 1988 Jun 04; 3(1):29-36. PubMed ID: 3134274
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  • 18. Sensitivity to the motoric effects of a dopamine receptor antagonist differs as a function of age at the time of dopamine depletion.
    Sandstrom MI, Bruno JP.
    Dev Psychobiol; 1997 May 04; 30(4):293-300. PubMed ID: 9142505
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  • 19. Serotonergic afferent regulation of the basic physiology and pharmacological responsiveness of nigrostriatal dopamine neurons.
    Kelland MD, Freeman AS, Chiodo LA.
    J Pharmacol Exp Ther; 1990 May 04; 253(2):803-11. PubMed ID: 1971022
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  • 20. In vivo electrophysiology of dopamine-denervated striatum: focus on the nitric oxide/cGMP signaling pathway.
    Galati S, D'angelo V, Scarnati E, Stanzione P, Martorana A, Procopio T, Sancesario G, Stefani A.
    Synapse; 2008 Jun 04; 62(6):409-20. PubMed ID: 18361439
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