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Journal Abstract Search

414 related items for PubMed ID: 31522431

  • 1. The Toll-like Receptor 2 (TLR2)-related Immunopathological Responses in the Multiple Sclerosis and Experimental Autoimmune Encephalomyelitis.
    Jafarzadeh A, Nemati M, Khorramdelazad H, Mirshafiey A.
    Iran J Allergy Asthma Immunol; 2019 Jun 08; 18(3):230-250. PubMed ID: 31522431
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  • 2. Delineating the Role of Toll-Like Receptors in the Neuro-inflammation Model EAE.
    Fallarino F, Gargaro M, Mondanell G, Downer EJ, Hossain MJ, Gran B.
    Methods Mol Biol; 2016 Jun 08; 1390():383-411. PubMed ID: 26803641
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  • 3. C-Reactive Protein Suppresses the Th17 Response Indirectly by Attenuating the Antigen Presentation Ability of Monocyte Derived Dendritic Cells in Experimental Autoimmune Encephalomyelitis.
    Shen ZY, Zheng Y, Pecsok MK, Wang K, Li W, Gong MJ, Wu F, Zhang L.
    Front Immunol; 2021 Jun 08; 12():589200. PubMed ID: 33841391
    [Abstract] [Full Text] [Related]

  • 4. The influence of T cell Ig mucin-3 signaling on central nervous system autoimmune disease is determined by the effector function of the pathogenic T cells.
    Lee SY, Goverman JM.
    J Immunol; 2013 May 15; 190(10):4991-9. PubMed ID: 23562810
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  • 12. Systemic TLR2 tolerance enhances central nervous system remyelination.
    Wasko NJ, Kulak MH, Paul D, Nicaise AM, Yeung ST, Nichols FC, Khanna KM, Crocker S, Pachter JS, Clark RB.
    J Neuroinflammation; 2019 Jul 27; 16(1):158. PubMed ID: 31351476
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  • 13. Proteinase-activated receptor 2 modulates neuroinflammation in experimental autoimmune encephalomyelitis and multiple sclerosis.
    Noorbakhsh F, Tsutsui S, Vergnolle N, Boven LA, Shariat N, Vodjgani M, Warren KG, Andrade-Gordon P, Hollenberg MD, Power C.
    J Exp Med; 2006 Feb 20; 203(2):425-35. PubMed ID: 16476770
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  • 14. Interleukin-19 Abrogates Experimental Autoimmune Encephalomyelitis by Attenuating Antigen-Presenting Cell Activation.
    Horiuchi H, Parajuli B, Komiya H, Ogawa Y, Jin S, Takahashi K, Azuma YT, Tanaka F, Suzumura A, Takeuchi H.
    Front Immunol; 2021 Feb 20; 12():615898. PubMed ID: 33776998
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  • 15. Chronological changes of CD4(+) and CD8(+) T cell subsets in the experimental autoimmune encephalomyelitis, a mouse model of multiple sclerosis.
    Sonobe Y, Jin S, Wang J, Kawanokuchi J, Takeuchi H, Mizuno T, Suzumura A.
    Tohoku J Exp Med; 2007 Dec 20; 213(4):329-39. PubMed ID: 18075237
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  • 16. IL-7/IL-7 Receptor Signaling Differentially Affects Effector CD4+ T Cell Subsets Involved in Experimental Autoimmune Encephalomyelitis.
    Arbelaez CA, Glatigny S, Duhen R, Eberl G, Oukka M, Bettelli E.
    J Immunol; 2015 Sep 01; 195(5):1974-83. PubMed ID: 26223651
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  • 17. Microglia and Dendritic Cells as a Source of IL-6 in a Mouse Model of Multiple Sclerosis.
    Gogoleva VS, Nguyen QC, Drutskaya MS.
    Biochemistry (Mosc); 2024 May 01; 89(5):904-911. PubMed ID: 38880650
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  • 18. FGF/FGFR Pathways in Multiple Sclerosis and in Its Disease Models.
    Rajendran R, Böttiger G, Stadelmann C, Karnati S, Berghoff M.
    Cells; 2021 Apr 13; 10(4):. PubMed ID: 33924474
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  • 19. Opposing Functions of Microglial and Macrophagic TNFR2 in the Pathogenesis of Experimental Autoimmune Encephalomyelitis.
    Gao H, Danzi MC, Choi CS, Taherian M, Dalby-Hansen C, Ellman DG, Madsen PM, Bixby JL, Lemmon VP, Lambertsen KL, Brambilla R.
    Cell Rep; 2017 Jan 03; 18(1):198-212. PubMed ID: 28052249
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  • 20. Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4 + T lymphocytes in established experimental autoimmune encephalomyelitis, a model of multiple sclerosis.
    Hammer LA, Waldner H, Zagon IS, McLaughlin PJ.
    Exp Biol Med (Maywood); 2016 Jan 03; 241(1):71-8. PubMed ID: 26202376
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