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Journal Abstract Search

231 related items for PubMed ID: 31562546

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  • 4. The redox chemistry of the Alzheimer's disease amyloid beta peptide.
    Smith DG, Cappai R, Barnham KJ.
    Biochim Biophys Acta; 2007 Aug; 1768(8):1976-90. PubMed ID: 17433250
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  • 5. Interactions of Zn(II) and Cu(II) ions with Alzheimer's amyloid-beta peptide. Metal ion binding, contribution to fibrillization and toxicity.
    Tõugu V, Tiiman A, Palumaa P.
    Metallomics; 2011 Mar; 3(3):250-61. PubMed ID: 21359283
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  • 6. Copper and zinc binding to amyloid-beta: coordination, dynamics, aggregation, reactivity and metal-ion transfer.
    Faller P.
    Chembiochem; 2009 Dec 14; 10(18):2837-45. PubMed ID: 19877000
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  • 10. Transition Metal Ion Interactions with Disordered Amyloid-β Peptides in the Pathogenesis of Alzheimer's Disease: Insights from Computational Chemistry Studies.
    Strodel B, Coskuner-Weber O.
    J Chem Inf Model; 2019 May 28; 59(5):1782-1805. PubMed ID: 30933519
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  • 16. Natural Tripeptide-Based Inhibitor of Multifaceted Amyloid β Toxicity.
    Rajasekhar K, Madhu C, Govindaraju T.
    ACS Chem Neurosci; 2016 Sep 21; 7(9):1300-10. PubMed ID: 27355515
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  • 18. β-amyloid fibrils in Alzheimer disease are not inert when bound to copper ions but can degrade hydrogen peroxide and generate reactive oxygen species.
    Mayes J, Tinker-Mill C, Kolosov O, Zhang H, Tabner BJ, Allsop D.
    J Biol Chem; 2014 Apr 25; 289(17):12052-12062. PubMed ID: 24619420
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  • 19. Amyloid beta-Cu2+ complexes in both monomeric and fibrillar forms do not generate H2O2 catalytically but quench hydroxyl radicals.
    Nadal RC, Rigby SE, Viles JH.
    Biochemistry; 2008 Nov 04; 47(44):11653-64. PubMed ID: 18847222
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  • 20. Identifying, by first-principles simulations, Cu[amyloid-β] species making Fenton-type reactions in Alzheimer's disease.
    La Penna G, Hureau C, Andreussi O, Faller P.
    J Phys Chem B; 2013 Dec 27; 117(51):16455-67. PubMed ID: 24313818
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