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161 related items for PubMed ID: 31664084

  • 1. Focal segmental glomerulosclerosis ACTN4 mutants binding to actin: regulation by phosphomimetic mutations.
    Shao H, Wingert B, Weins A, Pollak MR, Camacho C, Wells A.
    Sci Rep; 2019 Oct 29; 9(1):15517. PubMed ID: 31664084
    [Abstract] [Full Text] [Related]

  • 2. Phosphorylation of alpha-actinin 4 upon epidermal growth factor exposure regulates its interaction with actin.
    Shao H, Wu C, Wells A.
    J Biol Chem; 2010 Jan 22; 285(4):2591-600. PubMed ID: 19920151
    [Abstract] [Full Text] [Related]

  • 3. Phosphorylation of ACTN4 Leads to Podocyte Vulnerability and Proteinuric Glomerulosclerosis.
    Feng D, Kumar M, Muntel J, Gurley SB, Birrane G, Stillman IE, Ding L, Wang M, Ahmed S, Schlondorff J, Alper SL, Ferrante T, Marquez SL, Ng CF, Novak R, Ingber DE, Steen H, Pollak MR.
    J Am Soc Nephrol; 2020 Jul 22; 31(7):1479-1495. PubMed ID: 32540856
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  • 6. α-Actinin 4 potentiates nuclear factor κ-light-chain-enhancer of activated B-cell (NF-κB) activity in podocytes independent of its cytoplasmic actin binding function.
    Zhao X, Hsu KS, Lim JH, Bruggeman LA, Kao HY.
    J Biol Chem; 2015 Jan 02; 290(1):338-49. PubMed ID: 25411248
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  • 7. Three-layered proteomic characterization of a novel ACTN4 mutation unravels its pathogenic potential in FSGS.
    Bartram MP, Habbig S, Pahmeyer C, Höhne M, Weber LT, Thiele H, Altmüller J, Kottoor N, Wenzel A, Krueger M, Schermer B, Benzing T, Rinschen MM, Beck BB.
    Hum Mol Genet; 2016 Mar 15; 25(6):1152-64. PubMed ID: 26740551
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  • 8. Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis.
    Kaplan JM, Kim SH, North KN, Rennke H, Correia LA, Tong HQ, Mathis BJ, Rodríguez-Pérez JC, Allen PG, Beggs AH, Pollak MR.
    Nat Genet; 2000 Mar 15; 24(3):251-6. PubMed ID: 10700177
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  • 10. FSGS-associated alpha-actinin-4 (K256E) impairs cytoskeletal dynamics in podocytes.
    Michaud JL, Chaisson KM, Parks RJ, Kennedy CR.
    Kidney Int; 2006 Sep 15; 70(6):1054-61. PubMed ID: 16837921
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  • 11. Tyro3-mediated phosphorylation of ACTN4 at tyrosines is FAK-dependent and decreases susceptibility to cleavage by m-Calpain.
    Shao H, Wang A, Lauffenburger D, Wells A.
    Int J Biochem Cell Biol; 2018 Feb 15; 95():73-84. PubMed ID: 29274473
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  • 12. Disease-causing mutation in α-actinin-4 promotes podocyte detachment through maladaptation to periodic stretch.
    Feng D, Notbohm J, Benjamin A, He S, Wang M, Ang LH, Bantawa M, Bouzid M, Del Gado E, Krishnan R, Pollak MR.
    Proc Natl Acad Sci U S A; 2018 Feb 13; 115(7):1517-1522. PubMed ID: 29378953
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  • 13. Analysis of mutations in alpha-actinin 4 and podocin genes of patients with chronic renal failure due to sporadic focal segmental glomerulosclerosis.
    Komatsuda A, Wakui H, Maki N, Kigawa A, Goto H, Ohtani H, Hamai K, Oyama Y, Makoto H, Sawada K, Imai H.
    Ren Fail; 2003 Jan 13; 25(1):87-93. PubMed ID: 12617336
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  • 14. Mice with podocyte-specific overexpression of wild type alpha-actinin-4 are healthy controls for K256E-alpha-actinin-4 mutant transgenic mice.
    Michaud JL, Stitt-Cavanaugh E, Endlich N, Endlich K, De Repentigny Y, Kothary R, Kennedy CR.
    Transgenic Res; 2010 Apr 13; 19(2):285-9. PubMed ID: 19585264
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  • 15. Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic Testing.
    Feng D, Steinke JM, Krishnan R, Birrane G, Pollak MR.
    PLoS One; 2016 Apr 13; 11(12):e0167467. PubMed ID: 27977723
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  • 16. Functional analysis of promoter mutations in the ACTN4 and SYNPO genes in focal segmental glomerulosclerosis.
    Dai S, Wang Z, Pan X, Wang W, Chen X, Ren H, Hao C, Han B, Chen N.
    Nephrol Dial Transplant; 2010 Mar 13; 25(3):824-35. PubMed ID: 19666657
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  • 17. Nonlinear viscoelasticity of actin transiently cross-linked with mutant α-actinin-4.
    Yao NY, Becker DJ, Broedersz CP, Depken M, Mackintosh FC, Pollak MR, Weitz DA.
    J Mol Biol; 2011 Sep 02; 411(5):1062-71. PubMed ID: 21762701
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  • 18. Patients with ACTN4 mutations demonstrate distinctive features of glomerular injury.
    Henderson JM, Alexander MP, Pollak MR.
    J Am Soc Nephrol; 2009 May 02; 20(5):961-8. PubMed ID: 19357256
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  • 19. Screening of ACTN4 and TRPC6 mutations in a Chinese cohort of patients with adult-onset familial focal segmental glomerulosclerosis.
    Zhang Q, Ma J, Xie J, Wang Z, Zhu B, Hao X, Yang L, Ren H, Chen N.
    Contrib Nephrol; 2013 May 02; 181():91-100. PubMed ID: 23689571
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  • 20. Alpha-actinin-4-mediated FSGS: an inherited kidney disease caused by an aggregated and rapidly degraded cytoskeletal protein.
    Yao J, Le TC, Kos CH, Henderson JM, Allen PG, Denker BM, Pollak MR.
    PLoS Biol; 2004 Jun 02; 2(6):e167. PubMed ID: 15208719
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