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PUBMED FOR HANDHELDS

Journal Abstract Search


318 related items for PubMed ID: 31678200

  • 1. Re-balancing cellular energy substrate metabolism to mend the failing heart.
    Glatz JFC, Nabben M, Young ME, Schulze PC, Taegtmeyer H, Luiken JJFP.
    Biochim Biophys Acta Mol Basis Dis; 2020 May 01; 1866(5):165579. PubMed ID: 31678200
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  • 3. Enhancing fatty acid utilization ameliorates mitochondrial fragmentation and cardiac dysfunction via rebalancing optic atrophy 1 processing in the failing heart.
    Guo Y, Wang Z, Qin X, Xu J, Hou Z, Yang H, Mao X, Xing W, Li X, Zhang X, Gao F.
    Cardiovasc Res; 2018 Jun 01; 114(7):979-991. PubMed ID: 29490017
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  • 4. Regulation of sarcolemmal transport of substrates in the healthy and diseased heart.
    Glatz JF, Bonen A, Ouwens DM, Luiken JJ.
    Cardiovasc Drugs Ther; 2006 Dec 01; 20(6):471-6. PubMed ID: 17119873
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  • 9. Myocardial fatty acid uptake through CD36 is indispensable for sufficient bioenergetic metabolism to prevent progression of pressure overload-induced heart failure.
    Umbarawan Y, Syamsunarno MRAA, Koitabashi N, Obinata H, Yamaguchi A, Hanaoka H, Hishiki T, Hayakawa N, Sano M, Sunaga H, Matsui H, Tsushima Y, Suematsu M, Kurabayashi M, Iso T.
    Sci Rep; 2018 Aug 13; 8(1):12035. PubMed ID: 30104639
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  • 11. Cardioprotection Resulting from Glucagon-Like Peptide-1 Administration Involves Shifting Metabolic Substrate Utilization to Increase Energy Efficiency in the Rat Heart.
    Aravindhan K, Bao W, Harpel MR, Willette RN, Lepore JJ, Jucker BM.
    PLoS One; 2015 Aug 13; 10(6):e0130894. PubMed ID: 26098939
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  • 12. Retained Metabolic Flexibility of the Failing Human Heart.
    Watson WD, Green PG, Lewis AJM, Arvidsson P, De Maria GL, Arheden H, Heiberg E, Clarke WT, Rodgers CT, Valkovič L, Neubauer S, Herring N, Rider OJ.
    Circulation; 2023 Jul 11; 148(2):109-123. PubMed ID: 37199155
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  • 16. [New cardiotonic/inodilator agents: energetic aspects].
    Hasenfuss G.
    Z Kardiol; 1992 Jul 11; 81 Suppl 4():57-63. PubMed ID: 1290307
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  • 17. Metabolic profiling of hearts exposed to sevoflurane and propofol reveals distinct regulation of fatty acid and glucose oxidation: CD36 and pyruvate dehydrogenase as key regulators in anesthetic-induced fuel shift.
    Wang L, Ko KW, Lucchinetti E, Zhang L, Troxler H, Hersberger M, Omar MA, Posse de Chaves EI, Lopaschuk GD, Clanachan AS, Zaugg M.
    Anesthesiology; 2010 Sep 11; 113(3):541-51. PubMed ID: 20683255
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  • 18. CD36 inhibition prevents lipid accumulation and contractile dysfunction in rat cardiomyocytes.
    Angin Y, Steinbusch LK, Simons PJ, Greulich S, Hoebers NT, Douma K, van Zandvoort MA, Coumans WA, Wijnen W, Diamant M, Ouwens DM, Glatz JF, Luiken JJ.
    Biochem J; 2012 Nov 15; 448(1):43-53. PubMed ID: 22780108
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  • 20. Cardiac lipid metabolism, mitochondrial function, and heart failure.
    Da Dalt L, Cabodevilla AG, Goldberg IJ, Norata GD.
    Cardiovasc Res; 2023 Aug 19; 119(10):1905-1914. PubMed ID: 37392421
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