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538 related items for PubMed ID: 31689123
1. Long noncoding RNA MALAT1 participates in the pathological angiogenesis of diabetic retinopathy in an oxygen-induced retinopathy mouse model by sponging miR-203a-3p. Yu L, Fu J, Yu N, Wu Y, Han N. Can J Physiol Pharmacol; 2020 Apr; 98(4):219-227. PubMed ID: 31689123 [Abstract] [Full Text] [Related]
2. MiR-203a-3p inhibits retinal angiogenesis and alleviates proliferative diabetic retinopathy in oxygen-induced retinopathy (OIR) rat model via targeting VEGFA and HIF-1α. Han N, Xu H, Yu N, Wu Y, Yu L. Clin Exp Pharmacol Physiol; 2020 Jan; 47(1):85-94. PubMed ID: 31408201 [Abstract] [Full Text] [Related]
3. LncRNA-MALAT1 promotes neovascularization in diabetic retinopathy through regulating miR-125b/VE-cadherin axis. Liu P, Jia SB, Shi JM, Li WJ, Tang LS, Zhu XH, Tong P. Biosci Rep; 2019 May 31; 39(5):. PubMed ID: 30988072 [Abstract] [Full Text] [Related]
4. YAP1 is required for the angiogenesis in retinal microvascular endothelial cells via the inhibition of MALAT1-mediated miR-200b-3p in high glucose-induced diabetic retinopathy. Han N, Tian W, Yu N, Yu L. J Cell Physiol; 2020 Feb 31; 235(2):1309-1320. PubMed ID: 31313295 [Abstract] [Full Text] [Related]
5. MicroRNA-199a-3p inhibits angiogenesis by targeting the VEGF/PI3K/AKT signalling pathway in an in vitro model of diabetic retinopathy. Wang L, Liu WX, Huang XG. Exp Mol Pathol; 2020 Oct 31; 116():104488. PubMed ID: 32622012 [Abstract] [Full Text] [Related]
11. Competing endogenous RNA network associated with oxygen-induced retinopathy: Expression of the network and identification of the MALAT1/miR-124-3p/EGR1 regulatory axis. Xia F, Xu Y, Zhang X, Lyu J, Zhao P. Exp Cell Res; 2021 Nov 01; 408(1):112783. PubMed ID: 34469714 [Abstract] [Full Text] [Related]
17. MSC-derived exosomal lncRNA SNHG7 suppresses endothelial-mesenchymal transition and tube formation in diabetic retinopathy via miR-34a-5p/XBP1 axis. Cao X, Xue LD, Di Y, Li T, Tian YJ, Song Y. Life Sci; 2021 May 01; 272():119232. PubMed ID: 33600866 [Abstract] [Full Text] [Related]