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Journal Abstract Search

138 related items for PubMed ID: 31908015

  • 1. Time-course of sodium transport along the nephron in nephrotic syndrome: The role of potassium.
    Dizin E, Olivier V, Maire C, Komarynets O, Sassi A, Roth I, Loffing J, de Seigneux S, Maillard M, Rutkowski JM, Edwards A, Feraille E.
    FASEB J; 2020 Feb; 34(2):2408-2424. PubMed ID: 31908015
    [Abstract] [Full Text] [Related]

  • 2. Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na+ channel in the mouse kidney.
    Bohnert BN, Essigke D, Janessa A, Schneider JC, Wörn M, Kalo MZ, Xiao M, Kong L, Omage K, Hennenlotter J, Amend B, Birkenfeld AL, Artunc F.
    Am J Physiol Renal Physiol; 2021 Oct 01; 321(4):F480-F493. PubMed ID: 34423678
    [Abstract] [Full Text] [Related]

  • 3. Increased expression and apical targeting of renal ENaC subunits in puromycin aminonucleoside-induced nephrotic syndrome in rats.
    Kim SW, Wang W, Nielsen J, Praetorius J, Kwon TH, Knepper MA, Frøkiaer J, Nielsen S.
    Am J Physiol Renal Physiol; 2004 May 01; 286(5):F922-35. PubMed ID: 15075188
    [Abstract] [Full Text] [Related]

  • 4. Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome.
    Larionov A, Dahlke E, Kunke M, Zanon Rodriguez L, Schiessl IM, Magnin JL, Kern U, Alli AA, Mollet G, Schilling O, Castrop H, Theilig F.
    J Cell Mol Med; 2019 Oct 01; 23(10):6543-6553. PubMed ID: 31368174
    [Abstract] [Full Text] [Related]

  • 5. Increased apical targeting of renal ENaC subunits and decreased expression of 11betaHSD2 in HgCl2-induced nephrotic syndrome in rats.
    Kim SW, de Seigneux S, Sassen MC, Lee J, Kim J, Knepper MA, Frøkiaer J, Nielsen S.
    Am J Physiol Renal Physiol; 2006 Mar 01; 290(3):F674-87. PubMed ID: 16189294
    [Abstract] [Full Text] [Related]

  • 6. Inhibition of K+ secretion in the distal nephron in nephrotic syndrome: possible role of albuminuria.
    Fila M, Brideau G, Morla L, Cheval L, Deschênes G, Doucet A.
    J Physiol; 2011 Jul 15; 589(Pt 14):3611-21. PubMed ID: 21606114
    [Abstract] [Full Text] [Related]

  • 7. Proteolytic activation of the epithelial sodium channel (ENaC) by factor VII activating protease (FSAP) and its relevance for sodium retention in nephrotic mice.
    Artunc F, Bohnert BN, Schneider JC, Staudner T, Sure F, Ilyaskin AV, Wörn M, Essigke D, Janessa A, Nielsen NV, Birkenfeld AL, Etscheid M, Haerteis S, Korbmacher C, Kanse SM.
    Pflugers Arch; 2022 Feb 15; 474(2):217-229. PubMed ID: 34870751
    [Abstract] [Full Text] [Related]

  • 8. Severe Salt-Losing Syndrome and Hyperkalemia Induced by Adult Nephron-Specific Knockout of the Epithelial Sodium Channel α-Subunit.
    Perrier R, Boscardin E, Malsure S, Sergi C, Maillard MP, Loffing J, Loffing-Cueni D, Sørensen MV, Koesters R, Rossier BC, Frateschi S, Hummler E.
    J Am Soc Nephrol; 2016 Aug 15; 27(8):2309-18. PubMed ID: 26701978
    [Abstract] [Full Text] [Related]

  • 9. Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice.
    Haerteis S, Schork A, Dörffel T, Bohnert BN, Nacken R, Wörn M, Xiao M, Essigke D, Janessa A, Schmaier AH, Feener EP, Häring HU, Bertog M, Korbmacher C, Artunc F.
    Acta Physiol (Oxf); 2018 Sep 15; 224(1):e13060. PubMed ID: 29489063
    [Abstract] [Full Text] [Related]

  • 10. Sodium retention in nephrotic syndrome is independent of the activation of the membrane-anchored serine protease prostasin (CAP1/PRSS8) and its enzymatic activity.
    Essigke D, Bohnert BN, Janessa A, Wörn M, Omage K, Kalbacher H, Birkenfeld AL, Bugge TH, Szabo R, Artunc F.
    Pflugers Arch; 2022 Jun 15; 474(6):613-624. PubMed ID: 35312839
    [Abstract] [Full Text] [Related]

  • 11. Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.
    Bohnert BN, Daiminger S, Wörn M, Sure F, Staudner T, Ilyaskin AV, Batbouta F, Janessa A, Schneider JC, Essigke D, Kanse S, Haerteis S, Korbmacher C, Artunc F.
    Acta Physiol (Oxf); 2019 Dec 15; 227(4):e13286. PubMed ID: 31006168
    [Abstract] [Full Text] [Related]

  • 12. Responses of distal nephron Na+ transporters to acute volume depletion and hyperkalemia.
    Frindt G, Yang L, Uchida S, Weinstein AM, Palmer LG.
    Am J Physiol Renal Physiol; 2017 Jul 01; 313(1):F62-F73. PubMed ID: 28356292
    [Abstract] [Full Text] [Related]

  • 13. In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron.
    Nesterov V, Krueger B, Bertog M, Dahlmann A, Palmisano R, Korbmacher C.
    Hypertension; 2016 Jun 01; 67(6):1256-62. PubMed ID: 27170740
    [Abstract] [Full Text] [Related]

  • 14. Severe hyperkalemia is rescued by low-potassium diet in renal βENaC-deficient mice.
    Boscardin E, Perrier R, Sergi C, Maillard M, Loffing J, Loffing-Cueni D, Koesters R, Rossier BC, Hummler E.
    Pflugers Arch; 2017 Oct 01; 469(10):1387-1399. PubMed ID: 28567665
    [Abstract] [Full Text] [Related]

  • 15. Association of Urinary Plasminogen-Plasmin with Edema and Epithelial Sodium Channel Activation in Patients with Nephrotic Syndrome.
    Chen JL, Wang L, Yao XM, Zang YJ, Wang Y, Li ZJ, Pearce D, Wang H.
    Am J Nephrol; 2019 Oct 01; 50(2):92-104. PubMed ID: 31269481
    [Abstract] [Full Text] [Related]

  • 16. Rodent models to study sodium retention in experimental nephrotic syndrome.
    Xiao M, Bohnert BN, Grahammer F, Artunc F.
    Acta Physiol (Oxf); 2022 Jul 01; 235(3):e13844. PubMed ID: 35569011
    [Abstract] [Full Text] [Related]

  • 17. Nephrotic syndrome is associated with increased plasma K+ concentration, intestinal K+ losses, and attenuated urinary K+ excretion: a study in rats and humans.
    Ydegaard R, Svenningsen P, Bistrup C, Andersen RF, Stubbe J, Buhl KB, Marcussen N, Hinrichs GR, Iraqi H, Zamani R, Dimke H, Jensen BL.
    Am J Physiol Renal Physiol; 2019 Dec 01; 317(6):F1549-F1562. PubMed ID: 31566427
    [Abstract] [Full Text] [Related]

  • 18. [Role of distal nephron in the control of extracellular volume in physiology and in nephrotic syndrome].
    Feraille É, Olivier V.
    Med Sci (Paris); 2021 Apr 01; 37(4):359-365. PubMed ID: 33908853
    [Abstract] [Full Text] [Related]

  • 19. Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.
    Bohnert BN, Menacher M, Janessa A, Wörn M, Schork A, Daiminger S, Kalbacher H, Häring HU, Daniel C, Amann K, Sure F, Bertog M, Haerteis S, Korbmacher C, Artunc F.
    Kidney Int; 2018 Jan 01; 93(1):159-172. PubMed ID: 29042083
    [Abstract] [Full Text] [Related]

  • 20. Increased expression but not targeting of ENaC in adrenalectomized rats with PAN-induced nephrotic syndrome.
    de Seigneux S, Kim SW, Hemmingsen SC, Frøkiaer J, Nielsen S.
    Am J Physiol Renal Physiol; 2006 Jul 01; 291(1):F208-17. PubMed ID: 16403831
    [Abstract] [Full Text] [Related]

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