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Journal Abstract Search

265 related items for PubMed ID: 31981984

  • 1. Enhanced thrombospondin-1 causes dysfunction of vascular endothelial cells derived from Fabry disease-induced pluripotent stem cells.
    Do HS, Park SW, Im I, Seo D, Yoo HW, Go H, Kim YH, Koh GY, Lee BH, Han YM.
    EBioMedicine; 2020 Feb; 52():102633. PubMed ID: 31981984
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  • 4. Imbalanced Production of Reactive Oxygen Species and Mitochondrial Antioxidant SOD2 in Fabry Disease-Specific Human Induced Pluripotent Stem Cell-Differentiated Vascular Endothelial Cells.
    Tseng WL, Chou SJ, Chiang HC, Wang ML, Chien CS, Chen KH, Leu HB, Wang CY, Chang YL, Liu YY, Jong YJ, Lin SZ, Chiou SH, Lin SJ, Yu WC.
    Cell Transplant; 2017 Mar 13; 26(3):513-527. PubMed ID: 27938475
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  • 5. Generation of a CRISPR/Cas9-corrected-hiPSC line (DDLABi001-A) from Fabry disease (FD)-derived iPSCs having α-galactosidase (GLA) gene mutation (c.803_806del).
    Choi JB, Seo D, Do HS, Han YM.
    Stem Cell Res; 2023 Feb 13; 66():103001. PubMed ID: 36516658
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  • 6. Reversal of the Inflammatory Responses in Fabry Patient iPSC-Derived Cardiovascular Endothelial Cells by CRISPR/Cas9-Corrected Mutation.
    Song HY, Yang YP, Chien Y, Lai WY, Lin YY, Chou SJ, Wang ML, Wang CY, Leu HB, Yu WC, Chien CS.
    Int J Mol Sci; 2021 Feb 27; 22(5):. PubMed ID: 33673551
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  • 8. Generation of a GLA knock-out human-induced pluripotent stem cell line, KSBCi002-A-1, using CRISPR/Cas9.
    Kim YK, Yu JH, Min SH, Park SW.
    Stem Cell Res; 2020 Jan 27; 42():101676. PubMed ID: 31841972
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  • 12. CRISPR/Cas9-based GLA knockout to generate the female Fabry disease human induced pluripotent stem cell line MHHi001-A-15.
    Juchem M, Lehmann N, Behrens YL, Bär C, Thum T, Hoepfner J.
    Stem Cell Res; 2024 Sep 27; 79():103478. PubMed ID: 38905814
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  • 13. Globotriaosylceramide induces lysosomal degradation of endothelial KCa3.1 in fabry disease.
    Choi S, Kim JA, Na HY, Cho SE, Park S, Jung SC, Suh SH.
    Arterioscler Thromb Vasc Biol; 2014 Jan 27; 34(1):81-9. PubMed ID: 24158513
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  • 14. Expression of genes and their responses to enzyme replacement therapy in a Fabry disease mouse model.
    Park ES, Choi JO, Park JW, Lee MH, Park HY, Jung SC.
    Int J Mol Med; 2009 Sep 27; 24(3):401-7. PubMed ID: 19639234
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  • 15. Possible role of transforming growth factor-β1 and vascular endothelial growth factor in Fabry disease nephropathy.
    Lee MH, Choi EN, Jeon YJ, Jung SC.
    Int J Mol Med; 2012 Dec 27; 30(6):1275-80. PubMed ID: 23007467
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  • 16. Endothelial Cell Dysfunction and Hypoxia as Potential Mediators of Pain in Fabry Disease: A Human-Murine Translational Approach.
    Klug K, Spitzel M, Hans C, Klein A, Schottmann NM, Erbacher C, Üçeyler N.
    Int J Mol Sci; 2023 Oct 21; 24(20):. PubMed ID: 37895103
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  • 17. Endothelial Dysfunction in Fabry Disease Is Related to Glycocalyx Degradation.
    Pollmann S, Scharnetzki D, Manikowski D, Lenders M, Brand E.
    Front Immunol; 2021 Oct 21; 12():789142. PubMed ID: 34917096
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  • 18. Generation of GLA-Knockout Human Embryonic Stem Cell Lines to Model Autophagic Dysfunction and Exosome Secretion in Fabry Disease-Associated Hypertrophic Cardiomyopathy.
    Song HY, Chien CS, Yarmishyn AA, Chou SJ, Yang YP, Wang ML, Wang CY, Leu HB, Yu WC, Chang YL, Chiou SH.
    Cells; 2019 Apr 08; 8(4):. PubMed ID: 30965672
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  • 19. Generation of human induced pluripotent stem cell line MHHi029-A from a male Fabry disease patient carrying c.959A > T mutation.
    Jahn C, Juchem M, Sonnenschein K, Gietz A, Buchegger T, Lachmann N, Göhring G, Behrens YL, Bär C, Thum T, Hoepfner J.
    Stem Cell Res; 2024 Jun 08; 77():103404. PubMed ID: 38552356
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  • 20. Proteomic analysis unveils Gb3-independent alterations and mitochondrial dysfunction in a gla-/- zebrafish model of Fabry disease.
    Elsaid HOA, Rivedal M, Skandalou E, Svarstad E, Tøndel C, Birkeland E, Eikrem Ø, Babickova J, Marti HP, Furriol J.
    J Transl Med; 2023 Sep 05; 21(1):591. PubMed ID: 37670295
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