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Journal Abstract Search

161 related items for PubMed ID: 32284013

  • 1. Sensitive Sequencing Analysis Suggests Thyrotropin Receptor and Guanine Nucleotide-Binding Protein G Subunit Alpha as Sole Driver Mutations in Hot Thyroid Nodules.
    Stephenson A, Eszlinger M, Stewardson P, McIntyre JB, Boesenberg E, Bircan R, Sancak S, Gozu HI, Ghaznavi S, Krohn K, Paschke R.
    Thyroid; 2020 Oct; 30(10):1482-1489. PubMed ID: 32284013
    [Abstract] [Full Text] [Related]

  • 2. Expression of G(alpha)(s) proteins and TSH receptor signalling in hyperfunctioning thyroid nodules with TSH receptor mutations.
    Holzapfel HP, Bergner B, Wonerow P, Paschke R.
    Eur J Endocrinol; 2002 Jul; 147(1):109-16. PubMed ID: 12088927
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  • 3. Prevalence of TSH receptor and Gsalpha mutations in 45 autonomously functioning thyroid nodules in Japan.
    Nishihara E, Amino N, Maekawa K, Yoshida H, Ito M, Kubota S, Fukata S, Miyauchi A.
    Endocr J; 2009 Jul; 56(6):791-8. PubMed ID: 19550078
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  • 4. Detection of thyroid-stimulating hormone receptor and Gsalpha mutations: in 75 toxic thyroid nodules by denaturing gradient gel electrophoresis.
    Trülzsch B, Krohn K, Wonerow P, Chey S, Holzapfel HP, Ackermann F, Führer D, Paschke R.
    J Mol Med (Berl); 2001 Jul; 78(12):684-91. PubMed ID: 11434721
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  • 5. Similar prevalence of somatic TSH receptor and Gsalpha mutations in toxic thyroid nodules in geographical regions with different iodine supply in Turkey.
    Gozu HI, Bircan R, Krohn K, Müller S, Vural S, Gezen C, Sargin H, Yavuzer D, Sargin M, Cirakoglu B, Paschke R.
    Eur J Endocrinol; 2006 Oct; 155(4):535-45. PubMed ID: 16990652
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  • 6. A family with congenital hypothyroidism caused by a combination of loss-of-function mutations in the thyrotropin receptor and adenylate cyclase-stimulating G alpha-protein subunit genes.
    Lado-Abeal J, Castro-Piedras I, Palos-Paz F, Labarta-Aizpún JI, Albero-Gamboa R.
    Thyroid; 2011 Feb; 21(2):103-9. PubMed ID: 21186955
    [Abstract] [Full Text] [Related]

  • 7. Prevalence of mutations in TSHR, GNAS, PRKAR1A and RAS genes in a large series of toxic thyroid adenomas from Galicia, an iodine-deficient area in NW Spain.
    Palos-Paz F, Perez-Guerra O, Cameselle-Teijeiro J, Rueda-Chimeno C, Barreiro-Morandeira F, Lado-Abeal J, Galician Group for the Study of Toxic Multinodular Goitre, Araujo Vilar D, Argueso R, Barca O, Botana M, Cabezas-Agrícola JM, Catalina P, Dominguez Gerpe L, Fernandez T, Mato A, Nuño A, Penin M, Victoria B.
    Eur J Endocrinol; 2008 Nov; 159(5):623-31. PubMed ID: 18694911
    [Abstract] [Full Text] [Related]

  • 8. High prevalence of TSHR/Gsα mutation-negative clonal hot thyroid nodules (HNs) in a Turkish cohort.
    Sancak S, Jaeschke H, Eren F, Tarcin O, Guellueoglu B, Sen LS, Sever Z, Gozu HI, Bircan R, Akalin S, Paschke R, Eszlinger M.
    Horm Metab Res; 2011 Jul; 43(8):562-8. PubMed ID: 21773967
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  • 10. Increased expression of G-protein-coupled receptor kinases 3 and 4 in hyperfunctioning thyroid nodules.
    Voigt C, Holzapfel HP, Meyer S, Paschke R.
    J Endocrinol; 2004 Jul; 182(1):173-82. PubMed ID: 15225142
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  • 13. United detection GNAS and TSHR mutations in subclinical toxic multinodular goiter.
    Liu C, Yang J, Wang F, Wu C, Zhou M.
    Eur Arch Otorhinolaryngol; 2010 Feb; 267(2):281-7. PubMed ID: 19826830
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  • 18. Diversity and prevalence of somatic mutations in the thyrotropin receptor and Gs alpha genes as a cause of toxic thyroid adenomas.
    Parma J, Duprez L, Van Sande J, Hermans J, Rocmans P, Van Vliet G, Costagliola S, Rodien P, Dumont JE, Vassart G.
    J Clin Endocrinol Metab; 1997 Aug; 82(8):2695-701. PubMed ID: 9253356
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  • 19. Rarity of oncogenic mutations in the thyrotropin receptor of autonomously functioning thyroid nodules in Japan.
    Takeshita A, Nagayama Y, Yokoyama N, Ishikawa N, Ito K, Yamashita T, Obara T, Murakami Y, Kuma K, Takamatsu J.
    J Clin Endocrinol Metab; 1995 Sep; 80(9):2607-11. PubMed ID: 7673402
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