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Journal Abstract Search


228 related items for PubMed ID: 32416155

  • 1. Deficiency of the oxidative stress-responsive kinase p70S6K1 restores autophagy and ameliorates neural tube defects in diabetic embryopathy.
    Cao S, Shen WB, Reece EA, Yang P.
    Am J Obstet Gynecol; 2020 Nov; 223(5):753.e1-753.e14. PubMed ID: 32416155
    [Abstract] [Full Text] [Related]

  • 2. The increased activity of a transcription factor inhibits autophagy in diabetic embryopathy.
    Xu C, Chen X, Reece EA, Lu W, Yang P.
    Am J Obstet Gynecol; 2019 Jan; 220(1):108.e1-108.e12. PubMed ID: 30312583
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  • 3. Superoxide dismutase 2 overexpression alleviates maternal diabetes-induced neural tube defects, restores mitochondrial function and suppresses cellular stress in diabetic embryopathy.
    Zhong J, Xu C, Gabbay-Benziv R, Lin X, Yang P.
    Free Radic Biol Med; 2016 Jul; 96():234-44. PubMed ID: 27130031
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  • 4. Modulation of nuclear factor-κB signaling and reduction of neural tube defects by quercetin-3-glucoside in embryos of diabetic mice.
    Tan C, Meng F, Reece EA, Zhao Z.
    Am J Obstet Gynecol; 2018 Aug; 219(2):197.e1-197.e8. PubMed ID: 29733843
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  • 5. c-Jun NH2-terminal kinase 1/2 and endoplasmic reticulum stress as interdependent and reciprocal causation in diabetic embryopathy.
    Li X, Xu C, Yang P.
    Diabetes; 2013 Feb; 62(2):599-608. PubMed ID: 22961085
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  • 6. MicroRNA-322 overexpression reduces neural tube defects in diabetic pregnancies.
    Wang G, Song S, Shen WB, Reece EA, Yang P.
    Am J Obstet Gynecol; 2024 Feb; 230(2):254.e1-254.e13. PubMed ID: 37531989
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  • 7. Trehalose prevents neural tube defects by correcting maternal diabetes-suppressed autophagy and neurogenesis.
    Xu C, Li X, Wang F, Weng H, Yang P.
    Am J Physiol Endocrinol Metab; 2013 Sep 01; 305(5):E667-78. PubMed ID: 23880312
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  • 8. Decoding the oxidative stress hypothesis in diabetic embryopathy through proapoptotic kinase signaling.
    Yang P, Reece EA, Wang F, Gabbay-Benziv R.
    Am J Obstet Gynecol; 2015 May 01; 212(5):569-79. PubMed ID: 25434839
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  • 9. Oxidative stress-induced miR-27a targets the redox gene nuclear factor erythroid 2-related factor 2 in diabetic embryopathy.
    Zhao Y, Dong D, Reece EA, Wang AR, Yang P.
    Am J Obstet Gynecol; 2018 Jan 01; 218(1):136.e1-136.e10. PubMed ID: 29100869
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  • 11. Punicalagin exerts protective effect against high glucose-induced cellular stress and neural tube defects.
    Zhong J, Reece EA, Yang P.
    Biochem Biophys Res Commun; 2015 Nov 13; 467(2):179-84. PubMed ID: 26453010
    [Abstract] [Full Text] [Related]

  • 12. Maternal diabetes triggers DNA damage and DNA damage response in neurulation stage embryos through oxidative stress.
    Dong D, Yu J, Wu Y, Fu N, Villela NA, Yang P.
    Biochem Biophys Res Commun; 2015 Nov 13; 467(2):407-12. PubMed ID: 26427872
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  • 16. Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis.
    Wu Y, Reece EA, Zhong J, Dong D, Shen WB, Harman CR, Yang P.
    Am J Obstet Gynecol; 2016 Sep 13; 215(3):366.e1-366.e10. PubMed ID: 27038779
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  • 17. The Nrf2 Activator Vinylsulfone Reduces High Glucose-Induced Neural Tube Defects by Suppressing Cellular Stress and Apoptosis.
    Dong D, Reece EA, Yang P.
    Reprod Sci; 2016 Aug 13; 23(8):993-1000. PubMed ID: 26802109
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  • 18. Restoring BMP4 expression in vascular endothelial progenitors ameliorates maternal diabetes-induced apoptosis and neural tube defects.
    Cao S, Reece EA, Shen WB, Yang P.
    Cell Death Dis; 2020 Oct 15; 11(10):859. PubMed ID: 33060561
    [Abstract] [Full Text] [Related]

  • 19. Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.
    Chen X, Zhong J, Dong D, Liu G, Yang P.
    Toxicol Sci; 2017 Aug 01; 158(2):275-285. PubMed ID: 28482072
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