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347 related items for PubMed ID: 32443253

  • 1. Calreticulin regulated intrinsic apoptosis through mitochondria-dependent and independent pathways mediated by ER stress in arsenite exposed HT-22 cells.
    Wang X, Liu X, Chen Y, Wang H, Zhang R, Zhang Q, Wei Y, Shi S, Li X.
    Chemosphere; 2020 Jul; 251():126466. PubMed ID: 32443253
    [Abstract] [Full Text] [Related]

  • 2. Silencing calreticulin gene might protect cardiomyocytes from angiotensin II-induced apoptosis.
    Zhang W, Liu Z, Zhang Y, Bao Q, Wu W, Huang H, Liu X.
    Life Sci; 2018 Apr 01; 198():119-127. PubMed ID: 29453988
    [Abstract] [Full Text] [Related]

  • 3. ROS-mediated PERK-eIF2α-ATF4 pathway plays an important role in arsenite-induced L-02 cells apoptosis via regulating CHOP-DR5 signaling.
    Liu C, Zhang A.
    Environ Toxicol; 2020 Oct 01; 35(10):1100-1113. PubMed ID: 32506763
    [Abstract] [Full Text] [Related]

  • 4. Rutin hydrate inhibits apoptosis in the brains of cadmium chloride-treated rats via preserving the mitochondrial integrity and inhibiting endoplasmic reticulum stress.
    Mostafa DG, Khaleel EF, Badi RM, Abdel-Aleem GA, Abdeen HM.
    Neurol Res; 2019 Jul 01; 41(7):594-608. PubMed ID: 30973085
    [Abstract] [Full Text] [Related]

  • 5. Endoplasmic reticulum stress is involved in arsenite-induced oxidative injury in rat brain.
    Lin AM, Chao PL, Fang SF, Chi CW, Yang CH.
    Toxicol Appl Pharmacol; 2007 Oct 15; 224(2):138-46. PubMed ID: 17681577
    [Abstract] [Full Text] [Related]

  • 6. Genes associated with pro-apoptotic and protective mechanisms are affected differently on exposure of neuronal cell cultures to arsenite. No indication for endoplasmic reticulum stress despite activation of grp78 and gadd153 expression.
    Mengesdorf T, Althausen S, Paschen W.
    Brain Res Mol Brain Res; 2002 Aug 15; 104(2):227-39. PubMed ID: 12225878
    [Abstract] [Full Text] [Related]

  • 7. Biphasic Increases of Cell Surface Calreticulin Following Treatment with Mitoxantrone.
    Azuma Y, Suzuki K, Higai K, Matsumoto K, Tada S.
    Biol Pharm Bull; 2020 Oct 01; 43(10):1595-1599. PubMed ID: 32727970
    [Abstract] [Full Text] [Related]

  • 8. Isoquercetin ameliorates tunicamycin-induced apoptosis in rat dorsal root ganglion neurons via suppressing ROS-dependent endoplasmic reticulum stress.
    Lu T, Zhang C, Chai M, An Y.
    Biomed Pharmacother; 2016 May 01; 80():343-351. PubMed ID: 27133075
    [Abstract] [Full Text] [Related]

  • 9. Calreticulin is a fine tuning molecule in epibrassinolide-induced apoptosis through activating endoplasmic reticulum stress in colon cancer cells.
    Obakan-Yerlikaya P, Arisan ED, Coker-Gurkan A, Adacan K, Ozbey U, Somuncu B, Baran D, Palavan-Unsal N.
    Mol Carcinog; 2017 Jun 01; 56(6):1603-1619. PubMed ID: 28112451
    [Abstract] [Full Text] [Related]

  • 10. ROS mediated ER stress induces Bax-Bak dependent and independent apoptosis in response to Thioridazine.
    Seervi M, Rani A, Sharma AK, Santhosh Kumar TR.
    Biomed Pharmacother; 2018 Oct 01; 106():200-209. PubMed ID: 29960166
    [Abstract] [Full Text] [Related]

  • 11. Alpha-lipoic acid protects against cadmium-induced neuronal injury by inhibiting the endoplasmic reticulum stress eIF2α-ATF4 pathway in rat cortical neurons in vitro and in vivo.
    Yuan Y, Yang J, Chen J, Zhao S, Wang T, Zou H, Wang Y, Gu J, Liu X, Bian J, Liu Z.
    Toxicology; 2019 Feb 15; 414():1-13. PubMed ID: 30605698
    [Abstract] [Full Text] [Related]

  • 12. The endoplasmic reticulum is a target organelle for trivalent dimethylarsinic acid (DMAIII)-induced cytotoxicity.
    Naranmandura H, Xu S, Koike S, Pan LQ, Chen B, Wang YW, Rehman K, Wu B, Chen Z, Suzuki N.
    Toxicol Appl Pharmacol; 2012 May 01; 260(3):241-9. PubMed ID: 22425709
    [Abstract] [Full Text] [Related]

  • 13. Calreticulin translocation aggravates endoplasmic reticulum stress-associated apoptosis during cardiomyocyte hypoxia/reoxygenation.
    Xu FF, Liu XH.
    Chin Med J (Engl); 2015 Feb 05; 128(3):353-60. PubMed ID: 25635431
    [Abstract] [Full Text] [Related]

  • 14. Involvement of endoplasmic reticulum stress in all-trans-retinal-induced retinal pigment epithelium degeneration.
    Li J, Cai X, Xia Q, Yao K, Chen J, Zhang Y, Naranmandura H, Liu X, Wu Y.
    Toxicol Sci; 2015 Jan 05; 143(1):196-208. PubMed ID: 25331497
    [Abstract] [Full Text] [Related]

  • 15. 20(S)-protopanaxadiol induces apoptosis in human umbilical vein endothelial cells by activating the PERK-eIF2alpha-ATF4 signaling pathway.
    Wang X, Xia HY, Qin HY, Kang XP, Hu HY, Zheng J, Jiang JY, Yao LA, Xu YW, Zhang T, Zhang XL.
    J Cell Biochem; 2019 Apr 05; 120(4):5085-5096. PubMed ID: 30259568
    [Abstract] [Full Text] [Related]

  • 16. Valproate Attenuates Endoplasmic Reticulum Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling Pathway.
    Li Z, Wu F, Zhang X, Chai Y, Chen D, Yang Y, Xu K, Yin J, Li R, Shi H, Wang Z, Li X, Xiao J, Zhang H.
    Int J Mol Sci; 2017 Feb 08; 18(2):. PubMed ID: 28208696
    [Abstract] [Full Text] [Related]

  • 17. Arsenite-induced endoplasmic reticulum-dependent apoptosis through disturbance of calcium homeostasis in HBE cell line.
    Chen C, Gu S, Jiang X, Zhang Z.
    Environ Toxicol; 2017 Jan 08; 32(1):197-216. PubMed ID: 26677073
    [Abstract] [Full Text] [Related]

  • 18. Picosecond pulsed electric fields induce apoptosis in HeLa cells via the endoplasmic reticulum stress and caspase-dependent signaling pathways.
    Chen WJ, Xiong ZA, Zhang M, Yao CG, Zhao ZY, Hua YY, Zhou W.
    Int J Oncol; 2013 Mar 08; 42(3):963-70. PubMed ID: 23338860
    [Abstract] [Full Text] [Related]

  • 19. Endoplasmic reticulum rather than mitochondria plays a major role in the neuronal apoptosis induced by polybrominated diphenyl ether-153.
    Li X, Yang X, Chang L, Wang Z, Wang T, Zhang Z, Bai J, Liang R, Niu Q, Zhang H.
    Toxicol Lett; 2019 Sep 01; 311():37-48. PubMed ID: 31029751
    [Abstract] [Full Text] [Related]

  • 20. Roles of ERK/Akt signals in mitochondria-dependent and endoplasmic reticulum stress-triggered neuronal cell apoptosis induced by 4-methyl-2,4-bis(4-hydroxyphenyl)pent-1-ene, a major active metabolite of bisphenol A.
    Huang CF, Liu SH, Su CC, Fang KM, Yen CC, Yang CY, Tang FC, Liu JM, Wu CC, Lee KI, Chen YW.
    Toxicology; 2021 May 15; 455():152764. PubMed ID: 33771661
    [Abstract] [Full Text] [Related]

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