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Journal Abstract Search

122 related items for PubMed ID: 32452365

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  • 2. Cullin 3 mutant causing familial hyperkalemic hypertension lacks normal activity in the kidney.
    Maeoka Y, Cornelius RJ, Ferdaus MZ, Sharma A, Nguyen LT, McCormick JA.
    Am J Physiol Renal Physiol; 2022 Nov 01; 323(5):F564-F576. PubMed ID: 36007890
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  • 3. Familial Hyperkalemic Hypertension Genotype With a Negative Phenotype: A CUL3 Mosaicism.
    Ostrosky-Frid M, Chávez-Canales M, Romo M, Grunfeld B, Simsolo R, Segura-Kato Y, Tusié-Luna T, Gamba G.
    Am J Hypertens; 2020 Mar 13; 33(3):278-281. PubMed ID: 31802109
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  • 4. Rare cause of severe hypertension in an adolescent boy presenting with short stature: Answers.
    Yavas Abali Z, Yesil G, Kirkgoz T, Cicek N, Alpay H, Turan S, Bereket A, Guran T.
    Pediatr Nephrol; 2020 Mar 13; 35(3):405-407. PubMed ID: 31529157
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  • 10. Regulation of blood pressure and renal electrolyte balance by Cullin-RING ligases.
    Uchida S.
    Curr Opin Nephrol Hypertens; 2014 Sep 13; 23(5):487-93. PubMed ID: 24992566
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  • 12. A case of novel mutation of Cullin 3 gene in pseudohypoaldosteronism type II.
    Wang L, Nie M, Guo F, Tian Z, Guo X, Zhang S.
    J Hypertens; 2022 Jun 01; 40(6):1239-1242. PubMed ID: 35703886
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  • 14. Familial cases of pseudohypoaldosteronism type II harboring a novel mutation in the Cullin 3 gene.
    Nakano K, Kubota Y, Mori T, Chiga M, Mori T, Sonoda S, Ueda D, Asakura I, Ikegaya T, Kagawa J, Uchida S, Kubota A.
    Nephrology (Carlton); 2020 Nov 01; 25(11):818-821. PubMed ID: 32619053
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  • 16. [Causative genetic variants of pseudohypoaldosteronism type II and essential hypertension].
    Mori T, Uchida S.
    Nihon Jinzo Gakkai Shi; 2015 Nov 01; 57(4):751-7. PubMed ID: 26126332
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