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Journal Abstract Search

208 related items for PubMed ID: 32455507

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  • 3. Rodent models to study sodium retention in experimental nephrotic syndrome.
    Xiao M, Bohnert BN, Grahammer F, Artunc F.
    Acta Physiol (Oxf); 2022 Jul; 235(3):e13844. PubMed ID: 35569011
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  • 4. Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome.
    Larionov A, Dahlke E, Kunke M, Zanon Rodriguez L, Schiessl IM, Magnin JL, Kern U, Alli AA, Mollet G, Schilling O, Castrop H, Theilig F.
    J Cell Mol Med; 2019 Oct; 23(10):6543-6553. PubMed ID: 31368174
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  • 5. Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice.
    Hinrichs GR, Weyer K, Friis UG, Svenningsen P, Lund IK, Nielsen R, Mollet G, Antignac C, Bistrup C, Jensen BL, Birn H.
    Acta Physiol (Oxf); 2019 Dec; 227(4):e13362. PubMed ID: 31423748
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  • 7. Sodium retention in nephrotic syndrome is independent of the activation of the membrane-anchored serine protease prostasin (CAP1/PRSS8) and its enzymatic activity.
    Essigke D, Bohnert BN, Janessa A, Wörn M, Omage K, Kalbacher H, Birkenfeld AL, Bugge TH, Szabo R, Artunc F.
    Pflugers Arch; 2022 Jun; 474(6):613-624. PubMed ID: 35312839
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  • 9. Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice.
    Haerteis S, Schork A, Dörffel T, Bohnert BN, Nacken R, Wörn M, Xiao M, Essigke D, Janessa A, Schmaier AH, Feener EP, Häring HU, Bertog M, Korbmacher C, Artunc F.
    Acta Physiol (Oxf); 2018 Sep; 224(1):e13060. PubMed ID: 29489063
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  • 14. Plasminogen Deficiency and Amiloride Mitigate Angiotensin II-Induced Hypertension in Type 1 Diabetic Mice Suggesting Effects Through the Epithelial Sodium Channel.
    Andersen H, Hansen MH, Buhl KB, Stæhr M, Friis UG, Enggaard C, Supramaniyam S, Lund IK, Svenningsen P, Hansen PBL, Jensen BL.
    J Am Heart Assoc; 2020 Dec; 9(23):e016387. PubMed ID: 33215566
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  • 15. Aberrant glomerular filtration of urokinase-type plasminogen activator in nephrotic syndrome leads to amiloride-sensitive plasminogen activation in urine.
    Stæhr M, Buhl KB, Andersen RF, Svenningsen P, Nielsen F, Hinrichs GR, Bistrup C, Jensen BL.
    Am J Physiol Renal Physiol; 2015 Aug 01; 309(3):F235-41. PubMed ID: 25972510
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  • 16. Plasmin in nephrotic urine activates the epithelial sodium channel.
    Svenningsen P, Bistrup C, Friis UG, Bertog M, Haerteis S, Krueger B, Stubbe J, Jensen ON, Thiesson HC, Uhrenholt TR, Jespersen B, Jensen BL, Korbmacher C, Skøtt O.
    J Am Soc Nephrol; 2009 Feb 01; 20(2):299-310. PubMed ID: 19073825
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  • 18. Proteolytic Activity against the Distal Polybasic Tract of the Gamma Subunit of the Epithelial Sodium Channel ENaC in Nephrotic Urine.
    Wörn M, Kalbacher H, Artunc F.
    Curr Med Chem; 2022 Feb 01; 29(42):6433-6445. PubMed ID: 35676854
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  • 19. Proteinuric diseases with sodium retention: Is plasmin the link?
    Svenningsen P, Skøtt O, Jensen BL.
    Clin Exp Pharmacol Physiol; 2012 Jan 01; 39(1):117-24. PubMed ID: 21466573
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