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Journal Abstract Search

90 related items for PubMed ID: 3276214

  • 1. Adrenergic blockade does not abolish elevated glucose turnover during bacterial infection.
    Hargrove DM, Bagby GJ, Lang CH, Spitzer JJ.
    Am J Physiol; 1988 Jan; 254(1 Pt 1):E16-22. PubMed ID: 3276214
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  • 2. Adrenergic blockade prevents endotoxin-induced increases in glucose metabolism.
    Hargrove DM, Bagby GJ, Lang CH, Spitzer JJ.
    Am J Physiol; 1988 Nov; 255(5 Pt 1):E629-35. PubMed ID: 2903674
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  • 3. Platelet-activating factor-induced increases in glucose kinetics.
    Lang CH, Dobrescu C, Hargrove DM, Bagby GJ, Spitzer JJ.
    Am J Physiol; 1988 Feb; 254(2 Pt 1):E193-200. PubMed ID: 3348371
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  • 4. Epinephrine-induced increase in glucose turnover is diminished during sepsis.
    Hargrove DM, Lang CH, Bagby GJ, Spitzer JJ.
    Metabolism; 1989 Nov; 38(11):1070-6. PubMed ID: 2682135
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  • 8. Glucose, glucagon, and insulin during adrenergic blockade in exercising sheep.
    Brockman RP, Halvorson R.
    J Appl Physiol Respir Environ Exerc Physiol; 1982 Feb; 52(2):315-9. PubMed ID: 7037712
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  • 10. Importance of hyperglucagonemia in eliciting the sepsis-induced increase in glucose production.
    Lang CH, Bagby GJ, Blakesley HL, Spitzer JJ.
    Circ Shock; 1989 Nov; 29(3):181-91. PubMed ID: 2574079
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  • 15. Effect of beta-adrenergic blockade on lactate turnover in exercising dogs.
    Issekutz B.
    J Appl Physiol Respir Environ Exerc Physiol; 1984 Dec; 57(6):1754-9. PubMed ID: 6150924
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