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235 related items for PubMed ID: 32827692

  • 1. HGF protected against diabetic nephropathy via autophagy-lysosome pathway in podocyte by modulating PI3K/Akt-GSK3β-TFEB axis.
    Hou B, Li Y, Li X, Zhang C, Zhao Z, Chen Q, Zhang N, Li H.
    Cell Signal; 2020 Nov; 75():109744. PubMed ID: 32827692
    [Abstract] [Full Text] [Related]

  • 2. HGF alleviates high glucose-induced injury in podocytes by GSK3β inhibition and autophagy restoration.
    Zhang C, Hou B, Yu S, Chen Q, Zhang N, Li H.
    Biochim Biophys Acta; 2016 Nov; 1863(11):2690-2699. PubMed ID: 27526674
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  • 3. Tangshen formula protects against podocyte apoptosis via enhancing the TFEB-mediated autophagy-lysosome pathway in diabetic nephropathy.
    Wang Y, Peng L, Lu X, Zhang H, Zhao H, Zhao T, Yang L, Mao H, Ma F, Liu T, Li P, Zhan Y.
    J Ethnopharmacol; 2024 Apr 24; 324():117721. PubMed ID: 38199335
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  • 4. Sarsasapogenin restores podocyte autophagy in diabetic nephropathy by targeting GSK3β signaling pathway.
    Li XZ, Jiang H, Xu L, Liu YQ, Tang JW, Shi JS, Yu XJ, Wang X, Du L, Lu Q, Li CL, Liu YW, Yin XX.
    Biochem Pharmacol; 2021 Oct 24; 192():114675. PubMed ID: 34252407
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  • 5. Down-regulation of Risa improves podocyte injury by enhancing autophagy in diabetic nephropathy.
    Su PP, Liu DW, Zhou SJ, Chen H, Wu XM, Liu ZS.
    Mil Med Res; 2022 May 26; 9(1):23. PubMed ID: 35614465
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  • 11. Emerging role of podocyte autophagy in the progression of diabetic nephropathy.
    Yasuda-Yamahara M, Kume S, Tagawa A, Maegawa H, Uzu T.
    Autophagy; 2015 May 26; 11(12):2385-6. PubMed ID: 26565953
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  • 12. Hepatocyte growth factor attenuates high glucose-disturbed mitochondrial dynamics in podocytes by decreasing ARF6-dependent DRP1 translocation.
    Li Y, Li X, Yang Y, Li F, Chen Q, Zhao Z, Zhang N, Li H.
    Biochim Biophys Acta Mol Cell Res; 2024 Jan 26; 1871(1):119623. PubMed ID: 37913847
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  • 13. The redox-sensitive GSK3β is a key regulator of glomerular podocyte injury in type 2 diabetic kidney disease.
    Chen M, Fang Y, Ge Y, Qiu S, Dworkin L, Gong R.
    Redox Biol; 2024 Jun 26; 72():103127. PubMed ID: 38527400
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  • 14. Glycogen synthase kinase‑3β is required for epithelial‑mesenchymal transition and barrier dysfunction in mouse podocytes under high glucose conditions.
    Guo J, Yang L, Qiao Y, Liu Z.
    Mol Med Rep; 2016 Nov 26; 14(5):4091-4098. PubMed ID: 27748847
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  • 15. Hepatocyte growth factor signaling ameliorates podocyte injury and proteinuria.
    Dai C, Saleem MA, Holzman LB, Mathieson P, Liu Y.
    Kidney Int; 2010 Jun 26; 77(11):962-73. PubMed ID: 20375988
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  • 16. Reduced LRP6 expression and increase in the interaction of GSK3β with p53 contribute to podocyte apoptosis in diabetes mellitus and are prevented by green tea.
    Peixoto EB, Papadimitriou A, Teixeira DA, Montemurro C, Duarte DA, Silva KC, Joazeiro PP, Lopes de Faria JM, Lopes de Faria JB.
    J Nutr Biochem; 2015 Apr 26; 26(4):416-30. PubMed ID: 25655048
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  • 19. ATF4-dependent heme-oxygenase-1 attenuates diabetic nephropathy by inducing autophagy and inhibiting apoptosis in podocyte.
    Yuan S, Liang X, He W, Liang M, Jin J, He Q.
    Ren Fail; 2021 Dec 26; 43(1):968-979. PubMed ID: 34157937
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  • 20. p66Shc regulates podocyte autophagy in high glucose environment through the Notch-PTEN-PI3K/Akt/mTOR pathway.
    Zheng D, Tao M, Liang X, Li Y, Jin J, He Q.
    Histol Histopathol; 2020 Apr 26; 35(4):405-415. PubMed ID: 31650524
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