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402 related items for PubMed ID: 32896839

  • 1. Overexpression of Linc 4930556M19Rik Suppresses High Glucose-Triggered Podocyte Apoptosis, Fibrosis and Inflammation via the miR-27a-3p/Metalloproteinase 3 (TIMP3) Axis in Diabetic Nephropathy.
    Fan H, Zhang W.
    Med Sci Monit; 2020 Sep 08; 26():e925361. PubMed ID: 32896839
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  • 2. MiR-770-5p facilitates podocyte apoptosis and inflammation in diabetic nephropathy by targeting TIMP3.
    Wang L, Li H.
    Biosci Rep; 2020 Apr 30; 40(4):. PubMed ID: 32309847
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  • 3. Overexpression of HOXA-AS2 inhibits inflammation and apoptosis in podocytes via sponging miRNA-302b-3p to upregulate TIMP3.
    Li X, Yu HM.
    Eur Rev Med Pharmacol Sci; 2020 May 30; 24(9):4963-4970. PubMed ID: 32432759
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  • 4. Ablation of lncRNA MIAT mitigates high glucose-stimulated inflammation and apoptosis of podocyte via miR-130a-3p/TLR4 signaling axis.
    Zhang M, Zhao S, Xu C, Shen Y, Huang J, Shen S, Li Y, Chen X.
    Biochem Biophys Res Commun; 2020 Dec 10; 533(3):429-436. PubMed ID: 32972755
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  • 5. KCNQ1OT1 inhibition alleviates high glucose-induced podocyte injury by adsorbing miR-23b-3p and regulating Sema3A.
    Fei B, Zhou H, He Z, Wang S.
    Clin Exp Nephrol; 2022 May 10; 26(5):385-397. PubMed ID: 34997887
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  • 6. Long Noncoding RNA LINC01619 Regulates MicroRNA-27a/Forkhead Box Protein O1 and Endoplasmic Reticulum Stress-Mediated Podocyte Injury in Diabetic Nephropathy.
    Bai X, Geng J, Li X, Wan J, Liu J, Zhou Z, Liu X.
    Antioxid Redox Signal; 2018 Aug 01; 29(4):355-376. PubMed ID: 29334763
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  • 7. LncRNA 1500026H17Rik knockdown ameliorates high glucose-induced mouse podocyte injuries through the miR-205-5p/EGR1 pathway.
    Xia J, Sun W, Dun J.
    Int Urol Nephrol; 2023 Apr 01; 55(4):1045-1057. PubMed ID: 36306049
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  • 11. Retracted Article: Long non-coding RNA TUG1 alleviates high glucose induced podocyte inflammation, fibrosis and apoptosis in diabetic nephropathy via targeting the miR-27a-3p/E2F3 axis.
    Li Y, Huang D, Zheng L, Cao H, Gao Y, Yang Y, Fan Z.
    RSC Adv; 2019 Nov 13; 9(64):37620-37629. PubMed ID: 35542278
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  • 16. Overexpression of mm9_circ_013935 alleviates renal inflammation and fibrosis in diabetic nephropathy via the miR-153-3p/NFIC axis.
    Zhang L, Zhang L, Li S, Zhang Q, Luo Y, Zhang C, Huan Q, Zhang C.
    Can J Physiol Pharmacol; 2021 Nov 13; 99(11):1199-1206. PubMed ID: 34197715
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  • 17. miR-188-3p abolishes germacrone-mediated podocyte protection in a mouse model of diabetic nephropathy in type I diabetes through triggering mitochondrial injury.
    Wang Y, Feng F, He W, Sun L, He Q, Jin J.
    Bioengineered; 2022 Jan 13; 13(1):774-788. PubMed ID: 34847832
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  • 18. MiR-203-3p inhibits the oxidative stress, inflammatory responses and apoptosis of mice podocytes induced by high glucose through regulating Sema3A expression.
    Chen J, Xu Q, Zhang W, Zhen Y, Cheng F, Hua G, Lan J, Tu C.
    Open Life Sci; 2020 Jan 13; 15(1):939-950. PubMed ID: 33817280
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  • 20. Hsa_circ_0037128 aggravates high glucose-induced podocytes injury in diabetic nephropathy through mediating miR-31-5p/KLF9.
    Fang R, Cao X, Zhu Y, Chen Q.
    Autoimmunity; 2022 Jun 13; 55(4):254-263. PubMed ID: 35285770
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