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Journal Abstract Search

164 related items for PubMed ID: 33152396

  • 1. RUNX1/ETO and mutant KIT both contribute to programming the transcriptional and chromatin landscape in t(8;21) acute myeloid leukemia.
    Chin PS, Assi SA, Ptasinska A, Imperato MR, Cockerill PN, Bonifer C.
    Exp Hematol; 2020 Dec; 92():62-74. PubMed ID: 33152396
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  • 2. RUNX1-ETO Depletion in t(8;21) AML Leads to C/EBPα- and AP-1-Mediated Alterations in Enhancer-Promoter Interaction.
    Ptasinska A, Pickin A, Assi SA, Chin PS, Ames L, Avellino R, Gröschel S, Delwel R, Cockerill PN, Osborne CS, Bonifer C.
    Cell Rep; 2019 Sep 17; 28(12):3022-3031.e7. PubMed ID: 31533028
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  • 3. C-KIT mutation cooperates with full-length AML1-ETO to induce acute myeloid leukemia in mice.
    Wang YY, Zhao LJ, Wu CF, Liu P, Shi L, Liang Y, Xiong SM, Mi JQ, Chen Z, Ren R, Chen SJ.
    Proc Natl Acad Sci U S A; 2011 Feb 08; 108(6):2450-5. PubMed ID: 21262832
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  • 4. Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding.
    Ptasinska A, Assi SA, Mannari D, James SR, Williamson D, Dunne J, Hoogenkamp M, Wu M, Care M, McNeill H, Cauchy P, Cullen M, Tooze RM, Tenen DG, Young BD, Cockerill PN, Westhead DR, Heidenreich O, Bonifer C.
    Leukemia; 2012 Aug 08; 26(8):1829-41. PubMed ID: 22343733
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  • 5. t(8;21) Acute Myeloid Leukemia as a Paradigm for the Understanding of Leukemogenesis at the Level of Gene Regulation and Chromatin Programming.
    Kellaway S, Chin PS, Barneh F, Bonifer C, Heidenreich O.
    Cells; 2020 Dec 13; 9(12):. PubMed ID: 33322186
    [Abstract] [Full Text] [Related]

  • 6. The AML1-ETO fusion gene and the FLT3 length mutation collaborate in inducing acute leukemia in mice.
    Schessl C, Rawat VP, Cusan M, Deshpande A, Kohl TM, Rosten PM, Spiekermann K, Humphries RK, Schnittger S, Kern W, Hiddemann W, Quintanilla-Martinez L, Bohlander SK, Feuring-Buske M, Buske C.
    J Clin Invest; 2005 Aug 13; 115(8):2159-68. PubMed ID: 16025155
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  • 7. The Hematopoietic Transcription Factors RUNX1 and ERG Prevent AML1-ETO Oncogene Overexpression and Onset of the Apoptosis Program in t(8;21) AMLs.
    Mandoli A, Singh AA, Prange KHM, Tijchon E, Oerlemans M, Dirks R, Ter Huurne M, Wierenga ATJ, Janssen-Megens EM, Berentsen K, Sharifi N, Kim B, Matarese F, Nguyen LN, Hubner NC, Rao NA, van den Akker E, Altucci L, Vellenga E, Stunnenberg HG, Martens JHA.
    Cell Rep; 2016 Nov 15; 17(8):2087-2100. PubMed ID: 27851970
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  • 12. An AML1-ETO/miR-29b-1 regulatory circuit modulates phenotypic properties of acute myeloid leukemia cells.
    Zaidi SK, Perez AW, White ES, Lian JB, Stein JL, Stein GS.
    Oncotarget; 2017 Jun 20; 8(25):39994-40005. PubMed ID: 28611288
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  • 13. KIT-D816 mutations in AML1-ETO-positive AML are associated with impaired event-free and overall survival.
    Schnittger S, Kohl TM, Haferlach T, Kern W, Hiddemann W, Spiekermann K, Schoch C.
    Blood; 2006 Mar 01; 107(5):1791-9. PubMed ID: 16254134
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  • 19. Distinct classes of c-Kit-activating mutations differ in their ability to promote RUNX1-ETO-associated acute myeloid leukemia.
    Nick HJ, Kim HG, Chang CW, Harris KW, Reddy V, Klug CA.
    Blood; 2012 Feb 09; 119(6):1522-31. PubMed ID: 21937700
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