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Journal Abstract Search

558 related items for PubMed ID: 33232676

  • 21. KANK deficiency leads to podocyte dysfunction and nephrotic syndrome.
    Gee HY, Zhang F, Ashraf S, Kohl S, Sadowski CE, Vega-Warner V, Zhou W, Lovric S, Fang H, Nettleton M, Zhu JY, Hoefele J, Weber LT, Podracka L, Boor A, Fehrenbach H, Innis JW, Washburn J, Levy S, Lifton RP, Otto EA, Han Z, Hildebrandt F.
    J Clin Invest; 2015 Jun; 125(6):2375-84. PubMed ID: 25961457
    [Abstract] [Full Text] [Related]

  • 22. Mutations in PRDM15 Are a Novel Cause of Galloway-Mowat Syndrome.
    Mann N, Mzoughi S, Schneider R, Kühl SJ, Schanze D, Klämbt V, Lovric S, Mao Y, Shi S, Tan W, Kühl M, Onuchic-Whitford AC, Treimer E, Kitzler TM, Kause F, Schumann S, Nakayama M, Buerger F, Shril S, van der Ven AT, Majmundar AJ, Holton KM, Kolb A, Braun DA, Rao J, Jobst-Schwan T, Mildenberger E, Lennert T, Kuechler A, Wieczorek D, Gross O, Ermisch-Omran B, Werberger A, Skalej M, Janecke AR, Soliman NA, Mane SM, Lifton RP, Kadlec J, Guccione E, Schmeisser MJ, Zenker M, Hildebrandt F.
    J Am Soc Nephrol; 2021 Mar; 32(3):580-596. PubMed ID: 33593823
    [Abstract] [Full Text] [Related]

  • 23. Regional heterogeneity of astrocyte morphogenesis dictated by the formin protein, Daam2, modifies circuit function.
    Jo J, Woo J, Cristobal CD, Choi JM, Wang CY, Ye Q, Smith JA, Ung K, Liu G, Cortes D, Jung SY, Arenkiel BR, Lee HK.
    EMBO Rep; 2021 Dec 06; 22(12):e53200. PubMed ID: 34633730
    [Abstract] [Full Text] [Related]

  • 24. Mutations to the formin homology 2 domain of INF2 protein have unexpected effects on actin polymerization and severing.
    Ramabhadran V, Gurel PS, Higgs HN.
    J Biol Chem; 2012 Oct 05; 287(41):34234-45. PubMed ID: 22879592
    [Abstract] [Full Text] [Related]

  • 25. Formins as effector proteins of Rho GTPases.
    Kühn S, Geyer M.
    Small GTPases; 2014 Oct 05; 5():e29513. PubMed ID: 24914801
    [Abstract] [Full Text] [Related]

  • 26. Altered expression of Crb2 in podocytes expands a variation of CRB2 mutations in steroid-resistant nephrotic syndrome.
    Udagawa T, Jo T, Yanagihara T, Shimizu A, Mitsui J, Tsuji S, Morishita S, Onai R, Miura K, Kanda S, Kajiho Y, Tsurumi H, Oka A, Hattori M, Harita Y.
    Pediatr Nephrol; 2017 May 05; 32(5):801-809. PubMed ID: 27942854
    [Abstract] [Full Text] [Related]

  • 27. Inverted formin 2 in focal adhesions promotes dorsal stress fiber and fibrillar adhesion formation to drive extracellular matrix assembly.
    Skau CT, Plotnikov SV, Doyle AD, Waterman CM.
    Proc Natl Acad Sci U S A; 2015 May 12; 112(19):E2447-56. PubMed ID: 25918420
    [Abstract] [Full Text] [Related]

  • 28. Mutations in the formin gene INF2 cause focal segmental glomerulosclerosis.
    Brown EJ, Schlöndorff JS, Becker DJ, Tsukaguchi H, Tonna SJ, Uscinski AL, Higgs HN, Henderson JM, Pollak MR.
    Nat Genet; 2010 Jan 12; 42(1):72-6. PubMed ID: 20023659
    [Abstract] [Full Text] [Related]

  • 29. Multiple formin proteins participate in glioblastoma migration.
    Heuser VD, Kiviniemi A, Lehtinen L, Munthe S, Kristensen BW, Posti JP, Sipilä JOT, Vuorinen V, Carpén O, Gardberg M.
    BMC Cancer; 2020 Jul 29; 20(1):710. PubMed ID: 32727404
    [Abstract] [Full Text] [Related]

  • 30. Disease causing mutations in inverted formin 2 regulate its binding to G-actin, F-actin capping protein (CapZ α-1) and profilin 2.
    Rollason R, Wherlock M, Heath JA, Heesom KJ, Saleem MA, Welsh GI.
    Biosci Rep; 2016 Jan 13; 36(1):e00302. PubMed ID: 26764407
    [Abstract] [Full Text] [Related]

  • 31. Planar cell polarity pathway regulates actin rearrangement, cell shape, motility, and nephrin distribution in podocytes.
    Babayeva S, Zilber Y, Torban E.
    Am J Physiol Renal Physiol; 2011 Feb 13; 300(2):F549-60. PubMed ID: 20534871
    [Abstract] [Full Text] [Related]

  • 32. FAT1 mutations cause a glomerulotubular nephropathy.
    Gee HY, Sadowski CE, Aggarwal PK, Porath JD, Yakulov TA, Schueler M, Lovric S, Ashraf S, Braun DA, Halbritter J, Fang H, Airik R, Vega-Warner V, Cho KJ, Chan TA, Morris LG, ffrench-Constant C, Allen N, McNeill H, Büscher R, Kyrieleis H, Wallot M, Gaspert A, Kistler T, Milford DV, Saleem MA, Keng WT, Alexander SI, Valentini RP, Licht C, Teh JC, Bogdanovic R, Koziell A, Bierzynska A, Soliman NA, Otto EA, Lifton RP, Holzman LB, Sibinga NE, Walz G, Tufro A, Hildebrandt F.
    Nat Commun; 2016 Feb 24; 7():10822. PubMed ID: 26905694
    [Abstract] [Full Text] [Related]

  • 33. Recessive variants in MYO1C as a potential novel cause of proteinuric kidney disease.
    Elmubarak I, Shril S, Mansour B, Bao A, Kolvenbach CM, Kari JA, Shalaby MA, El Desoky S, Hildebrandt F, Schneider R.
    Pediatr Nephrol; 2024 Oct 24; 39(10):2939-2945. PubMed ID: 38904753
    [Abstract] [Full Text] [Related]

  • 34. Schip1 is a novel podocyte foot process protein that mediates actin cytoskeleton rearrangements and forms a complex with Nherf2 and ezrin.
    Perisic L, Rodriguez PQ, Hultenby K, Sun Y, Lal M, Betsholtz C, Uhlén M, Wernerson A, Hedin U, Pikkarainen T, Tryggvason K, Patrakka J.
    PLoS One; 2015 Oct 24; 10(3):e0122067. PubMed ID: 25807495
    [Abstract] [Full Text] [Related]

  • 35. Actin monomers activate inverted formin 2 by competing with its autoinhibitory interaction.
    Ramabhadran V, Hatch AL, Higgs HN.
    J Biol Chem; 2013 Sep 13; 288(37):26847-55. PubMed ID: 23921379
    [Abstract] [Full Text] [Related]

  • 36. Novel mutations in the inverted formin 2 gene of Chinese families contribute to focal segmental glomerulosclerosis.
    Xie J, Hao X, Azeloglu EU, Ren H, Wang Z, Ma J, Liu J, Ma X, Wang W, Pan X, Zhang W, Zhong F, Li Y, Meng G, Kiryluk K, He JC, Gharavi AG, Chen N.
    Kidney Int; 2015 Sep 13; 88(3):593-604. PubMed ID: 26039629
    [Abstract] [Full Text] [Related]

  • 37. New Paradigm for Cytoskeletal Organization in Podocytes: Proteolytic Fragments of INF2 Formin Function Independently of INF2 Actin Regulatory Activity.
    Krendel M, Pruyne D.
    J Am Soc Nephrol; 2020 Feb 13; 31(2):235-236. PubMed ID: 31924669
    [No Abstract] [Full Text] [Related]

  • 38. Afadin regulates RhoA/Rho-associated protein kinase signaling to control formation of actin stress fibers in kidney podocytes.
    Saito K, Shiino T, Kurihara H, Harita Y, Hattori S, Ohta Y.
    Cytoskeleton (Hoboken); 2015 Mar 13; 72(3):146-56. PubMed ID: 25712270
    [Abstract] [Full Text] [Related]

  • 39. Genetic variants in the LAMA5 gene in pediatric nephrotic syndrome.
    Braun DA, Warejko JK, Ashraf S, Tan W, Daga A, Schneider R, Hermle T, Jobst-Schwan T, Widmeier E, Majmundar AJ, Nakayama M, Schapiro D, Rao J, Schmidt JM, Hoogstraten CA, Hugo H, Bakkaloglu SA, Kari JA, El Desoky S, Daouk G, Mane S, Lifton RP, Shril S, Hildebrandt F.
    Nephrol Dial Transplant; 2019 Mar 01; 34(3):485-493. PubMed ID: 29534211
    [Abstract] [Full Text] [Related]

  • 40. Circulating extracellular vesicles of patients with steroid-sensitive nephrotic syndrome have higher RAC1 and induce recapitulation of nephrotic syndrome phenotype in podocytes.
    Eroglu FK, Yazar V, Guler U, Yıldırım M, Yildirim T, Gungor T, Celikkaya E, Karakaya D, Turay N, Ciftci Dede E, Korkusuz P, Salih B, Bulbul M, Gursel I.
    Am J Physiol Renal Physiol; 2021 Nov 01; 321(5):F659-F673. PubMed ID: 34569252
    [Abstract] [Full Text] [Related]

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