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PUBMED FOR HANDHELDS

Journal Abstract Search


267 related items for PubMed ID: 33339872

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  • 3. Sost deficiency leads to reduced mechanical strains at the tibia midshaft in strain-matched in vivo loading experiments in mice.
    Albiol L, Cilla M, Pflanz D, Kramer I, Kneissel M, Duda GN, Willie BM, Checa S.
    J R Soc Interface; 2018 Apr; 15(141):. PubMed ID: 29669893
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  • 9. Evidence for the major contribution of remodeling-based bone formation in sclerostin-deficient mice.
    Koide M, Yamashita T, Nakamura K, Yasuda H, Udagawa N, Kobayashi Y.
    Bone; 2022 Jul; 160():116401. PubMed ID: 35381389
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  • 12. An Additional Lrp4 High Bone Mass Mutation Mitigates the Sost-Knockout Phenotype in Mice by Increasing Bone Remodeling.
    Hendrickx G, Boudin E, Mateiu L, Yorgan TA, Steenackers E, Kneissel M, Kramer I, Mortier G, Schinke T, Van Hul W.
    Calcif Tissue Int; 2024 Feb; 114(2):171-181. PubMed ID: 38051321
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  • 14. Targeted postnatal knockout of Sclerostin using a bone-targeted adeno-associated viral vector increases bone anabolism and decreases canalicular density.
    O'Donohue AK, Xiao Y, Lee LR, Schofield T, Cheng TL, Munns CF, Baldock PA, Schindeler A.
    Bone; 2023 Feb; 167():116636. PubMed ID: 36462771
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  • 15. Bone adaptation to mechanical loading in mice is affected by circadian rhythms.
    Bouchard AL, Dsouza C, Julien C, Rummler M, Gaumond MH, Cermakian N, Willie BM.
    Bone; 2022 Jan; 154():116218. PubMed ID: 34571201
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  • 19. Activation of Wnt Signaling by Mechanical Loading Is Impaired in the Bone of Old Mice.
    Holguin N, Brodt MD, Silva MJ.
    J Bone Miner Res; 2016 Dec; 31(12):2215-2226. PubMed ID: 27357062
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