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Journal Abstract Search

144 related items for PubMed ID: 33428878

  • 1. γ-Secretase inhibitors for breast cancer and hepatocellular carcinoma: From mechanism to treatment.
    Jia H, Wang Z, Zhang J, Feng F.
    Life Sci; 2021 Mar 01; 268():119007. PubMed ID: 33428878
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  • 2. Evaluation of selective gamma-secretase inhibitor PF-03084014 for its antitumor efficacy and gastrointestinal safety to guide optimal clinical trial design.
    Wei P, Walls M, Qiu M, Ding R, Denlinger RH, Wong A, Tsaparikos K, Jani JP, Hosea N, Sands M, Randolph S, Smeal T.
    Mol Cancer Ther; 2010 Jun 01; 9(6):1618-28. PubMed ID: 20530712
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  • 9. A novel triazole, NMK-T-057, induces autophagic cell death in breast cancer cells by inhibiting γ-secretase-mediated activation of Notch signaling.
    Das A, Narayanam MK, Paul S, Mukhnerjee P, Ghosh S, Dastidar DG, Chakrabarty S, Ganguli A, Basu B, Pal M, Chatterji U, Banerjee SK, Karmakar P, Kumar D, Chakrabarti G.
    J Biol Chem; 2019 Apr 26; 294(17):6733-6750. PubMed ID: 30824542
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  • 10. GSI-I has a better effect in inhibiting hepatocellular carcinoma cell growth than GSI-IX, GSI-X, or GSI-XXI.
    Shen Y, Lv D, Wang J, Yin Y, Miao F, Dou F, Zhang J.
    Anticancer Drugs; 2012 Aug 26; 23(7):683-90. PubMed ID: 22569108
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  • 11. Notch versus the proteasome: what is the target of gamma-secretase inhibitor-I?
    Clementz AG, Osipo C.
    Breast Cancer Res; 2009 Aug 26; 11(5):110. PubMed ID: 19849815
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  • 12. Pharmacological analysis of Drosophila melanogaster gamma-secretase with respect to differential proteolysis of Notch and APP.
    Groth C, Alvord WG, Quiñones OA, Fortini ME.
    Mol Pharmacol; 2010 Apr 26; 77(4):567-74. PubMed ID: 20064975
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  • 13. PEST domain mutations in Notch receptors comprise an oncogenic driver segment in triple-negative breast cancer sensitive to a γ-secretase inhibitor.
    Wang K, Zhang Q, Li D, Ching K, Zhang C, Zheng X, Ozeck M, Shi S, Li X, Wang H, Rejto P, Christensen J, Olson P.
    Clin Cancer Res; 2015 Mar 15; 21(6):1487-96. PubMed ID: 25564152
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  • 14. The cytotoxicity of gamma-secretase inhibitor I to breast cancer cells is mediated by proteasome inhibition, not by gamma-secretase inhibition.
    Han J, Ma I, Hendzel MJ, Allalunis-Turner J.
    Breast Cancer Res; 2009 Mar 15; 11(4):R57. PubMed ID: 19660128
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  • 18. Epidermal growth factor receptor and notch pathways participate in the tumor suppressor function of gamma-secretase.
    Li T, Wen H, Brayton C, Das P, Smithson LA, Fauq A, Fan X, Crain BJ, Price DL, Golde TE, Eberhart CG, Wong PC.
    J Biol Chem; 2007 Nov 02; 282(44):32264-73. PubMed ID: 17827153
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  • 19. Tumours with elevated levels of the Notch and Wnt pathways exhibit efficacy to PF-03084014, a γ-secretase inhibitor, in a preclinical colorectal explant model.
    Arcaroli JJ, Quackenbush KS, Purkey A, Powell RW, Pitts TM, Bagby S, Tan AC, Cross B, McPhillips K, Song EK, Tai WM, Winn RA, Bikkavilli K, Vanscoyk M, Eckhardt SG, Messersmith WA.
    Br J Cancer; 2013 Aug 06; 109(3):667-75. PubMed ID: 23868008
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  • 20. Gamma-secretase inhibitor prevents Notch3 activation and reduces proliferation in human lung cancers.
    Konishi J, Kawaguchi KS, Vo H, Haruki N, Gonzalez A, Carbone DP, Dang TP.
    Cancer Res; 2007 Sep 01; 67(17):8051-7. PubMed ID: 17804716
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