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PUBMED FOR HANDHELDS

Journal Abstract Search


227 related items for PubMed ID: 33569056

  • 1. Neutrophil-Derived Myeloperoxidase Facilitates Both the Induction and Elicitation Phases of Contact Hypersensitivity.
    Strzepa A, Gurski CJ, Dittel LJ, Szczepanik M, Pritchard KA, Dittel BN.
    Front Immunol; 2020; 11():608871. PubMed ID: 33569056
    [Abstract] [Full Text] [Related]

  • 2. IL-36α is involved in hapten-specific T-cell induction, but not local inflammation, during contact hypersensitivity.
    Numata T, Yoshizaki T, Yamaguchi S, Shimura E, Iwakura Y, Harada K, Sudo K, Tsuboi R, Nakae S.
    Biochem Biophys Res Commun; 2018 Nov 30; 506(3):429-436. PubMed ID: 30352688
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  • 3. Neutrophils are required for both the sensitization and elicitation phase of contact hypersensitivity.
    Weber FC, Németh T, Csepregi JZ, Dudeck A, Roers A, Ozsvári B, Oswald E, Puskás LG, Jakob T, Mócsai A, Martin SF.
    J Exp Med; 2015 Jan 12; 212(1):15-22. PubMed ID: 25512469
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  • 4. Oxazolone-induced contact hypersensitivity reduces lymphatic drainage but enhances the induction of adaptive immunity.
    Aebischer D, Willrodt AH, Halin C.
    PLoS One; 2014 Jan 12; 9(6):e99297. PubMed ID: 24911791
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  • 5. Complementary methods for contact hypersensitivity (CHS) evaluation in mice.
    Zemelka-Wiącek M, Majewska-Szczepanik M, Pyrczak W, Szczepanik M.
    J Immunol Methods; 2013 Jan 31; 387(1-2):270-5. PubMed ID: 23183274
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  • 6. Dissection of antigenic and irritative effects of epicutaneously applied haptens in mice. Evidence that not the antigenic component but nonspecific proinflammatory effects of haptens determine the concentration-dependent elicitation of allergic contact dermatitis.
    Grabbe S, Steinert M, Mahnke K, Schwartz A, Luger TA, Schwarz T.
    J Clin Invest; 1996 Sep 01; 98(5):1158-64. PubMed ID: 8787679
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  • 8. In Vivo Expansion of Endogenous Regulatory T Cell Populations Induces Long-Term Suppression of Contact Hypersensitivity.
    El Beidaq A, Link CW, Hofmann K, Frehse B, Hartmann K, Bieber K, Martin SF, Ludwig RJ, Manz RA.
    J Immunol; 2016 Sep 01; 197(5):1567-76. PubMed ID: 27439515
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  • 9. Ultraviolet radiation-induced regulatory T cells not only inhibit the induction but can suppress the effector phase of contact hypersensitivity.
    Schwarz A, Maeda A, Wild MK, Kernebeck K, Gross N, Aragane Y, Beissert S, Vestweber D, Schwarz T.
    J Immunol; 2004 Jan 15; 172(2):1036-43. PubMed ID: 14707077
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  • 12. Leukotriene B₄-leukotriene B₄ receptor axis promotes oxazolone-induced contact dermatitis by directing skin homing of neutrophils and CD8⁺ T cells.
    Lv J, Zou L, Zhao L, Yang W, Xiong Y, Li B, He R.
    Immunology; 2015 Sep 15; 146(1):50-8. PubMed ID: 25959240
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  • 13. Contact Hypersensitivity as a Murine Model of Allergic Contact Dermatitis.
    Zemelka-Wiacek M, Majewska-Szczepanik M, Gajdanowicz P, Szczepanik M.
    J Vis Exp; 2022 Sep 26; (187):. PubMed ID: 36282686
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  • 14. Contribution of Langerhans cell-derived IL-18 to contact hypersensitivity.
    Wang B, Feliciani C, Howell BG, Freed I, Cai Q, Watanabe H, Sauder DN.
    J Immunol; 2002 Apr 01; 168(7):3303-8. PubMed ID: 11907086
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  • 19. Vascular endothelial growth factor receptor tyrosine kinase inhibitor PTK787/ZK 222584 inhibits both the induction and elicitation phases of contact hypersensitivity.
    Yamamoto A, Watanabe H, Sueki H, Nakanishi T, Yasuhara H, Iijima M.
    J Dermatol; 2007 Jul 01; 34(7):419-29. PubMed ID: 17584317
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