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Journal Abstract Search

226 related items for PubMed ID: 33687977

  • 1. Crumbs2 Is an Essential Slit Diaphragm Protein of the Renal Filtration Barrier.
    Möller-Kerutt A, Rodriguez-Gatica JE, Wacker K, Bhatia R, Siebrasse JP, Boon N, Van Marck V, Boor P, Kubitscheck U, Wijnholds J, Pavenstädt H, Weide T.
    J Am Soc Nephrol; 2021 May 03; 32(5):1053-1070. PubMed ID: 33687977
    [Abstract] [Full Text] [Related]

  • 2. Altered expression of Crb2 in podocytes expands a variation of CRB2 mutations in steroid-resistant nephrotic syndrome.
    Udagawa T, Jo T, Yanagihara T, Shimizu A, Mitsui J, Tsuji S, Morishita S, Onai R, Miura K, Kanda S, Kajiho Y, Tsurumi H, Oka A, Hattori M, Harita Y.
    Pediatr Nephrol; 2017 May 03; 32(5):801-809. PubMed ID: 27942854
    [Abstract] [Full Text] [Related]

  • 3. Disruption of the exocyst induces podocyte loss and dysfunction.
    Nihalani D, Solanki AK, Arif E, Srivastava P, Rahman B, Zuo X, Dang Y, Fogelgren B, Fermin D, Gillies CE, Sampson MG, Lipschutz JH.
    J Biol Chem; 2019 Jun 28; 294(26):10104-10119. PubMed ID: 31073028
    [Abstract] [Full Text] [Related]

  • 4. Novel variants in CRB2 targeting the malfunction of slit diaphragm related to focal segmental glomerulosclerosis.
    Yang Q, Tang D, Gan C, Bai M, Song X, Jiang W, Li Q, Chen Y, Zhang A, Wang M.
    Pediatr Nephrol; 2024 Jan 28; 39(1):149-165. PubMed ID: 37452832
    [Abstract] [Full Text] [Related]

  • 5. Neurexin-1, a presynaptic adhesion molecule, localizes at the slit diaphragm of the glomerular podocytes in kidneys.
    Saito A, Miyauchi N, Hashimoto T, Karasawa T, Han GD, Kayaba M, Sumi T, Tomita M, Ikezumi Y, Suzuki K, Koitabashi Y, Shimizu F, Kawachi H.
    Am J Physiol Regul Integr Comp Physiol; 2011 Feb 28; 300(2):R340-8. PubMed ID: 21048075
    [Abstract] [Full Text] [Related]

  • 6. Loss of surface transport is a main cellular pathomechanism of CRB2 variants causing podocytopathies.
    Möller-Kerutt A, Schönhoff B, Rellmann Y, George B, Braun DA, Pavenstädt H, Weide T.
    Life Sci Alliance; 2023 Mar 28; 6(3):. PubMed ID: 36549870
    [Abstract] [Full Text] [Related]

  • 7. A Novel Mouse Model of Idiopathic Nephrotic Syndrome Induced by Immunization with the Podocyte Protein Crb2.
    Hada I, Shimizu A, Takematsu H, Nishibori Y, Kimura T, Fukutomi T, Kudo A, Ito-Nitta N, Kiuchi Z, Patrakka J, Mikami N, Leclerc S, Akimoto Y, Hirayama Y, Mori S, Takano T, Yan K.
    J Am Soc Nephrol; 2022 Nov 28; 33(11):2008-2025. PubMed ID: 35985815
    [Abstract] [Full Text] [Related]

  • 8. Slit diaphragm dysfunction in proteinuric states: identification of novel therapeutic targets for nephrotic syndrome.
    Kawachi H, Suzuki K, Miyauchi N, Hashimoto T, Otaki Y, Shimizu F.
    Clin Exp Nephrol; 2009 Aug 28; 13(4):275-280. PubMed ID: 19266252
    [Abstract] [Full Text] [Related]

  • 9. SIRPα interacts with nephrin at the podocyte slit diaphragm.
    Kajiho Y, Harita Y, Kurihara H, Horita S, Matsunaga A, Tsurumi H, Kanda S, Sugawara N, Miura K, Sekine T, Hattori S, Hattori M, Igarashi T.
    FEBS J; 2012 Sep 28; 279(17):3010-21. PubMed ID: 22747997
    [Abstract] [Full Text] [Related]

  • 10. Nck proteins maintain the adult glomerular filtration barrier.
    Jones N, New LA, Fortino MA, Eremina V, Ruston J, Blasutig IM, Aoudjit L, Zou Y, Liu X, Yu GL, Takano T, Quaggin SE, Pawson T.
    J Am Soc Nephrol; 2009 Jul 28; 20(7):1533-43. PubMed ID: 19443634
    [Abstract] [Full Text] [Related]

  • 11. [Structure and function of the glomerular filtration barrier].
    Musiał K, Zwolińska D.
    Pol Merkur Lekarski; 2005 Mar 28; 18(105):317-20. PubMed ID: 15997642
    [Abstract] [Full Text] [Related]

  • 12. Podocytes are firmly attached to glomerular basement membrane in kidneys with heavy proteinuria.
    Lahdenkari AT, Lounatmaa K, Patrakka J, Holmberg C, Wartiovaara J, Kestilä M, Koskimies O, Jalanko H.
    J Am Soc Nephrol; 2004 Oct 28; 15(10):2611-8. PubMed ID: 15466265
    [Abstract] [Full Text] [Related]

  • 13. The human nephrin Y1139RSL motif is essential for podocyte foot process organization and slit diaphragm formation during glomerular development.
    Espiritu EB, Jiang H, Moreau-Marquis S, Sullivan M, Yan K, Beer Stolz D, Sampson MG, Hukriede NA, Swiatecka-Urban A.
    J Biol Chem; 2019 Jul 12; 294(28):10773-10788. PubMed ID: 31152064
    [Abstract] [Full Text] [Related]

  • 14. Partitioning-Defective-6-Ephrin-B1 Interaction Is Regulated by Nephrin-Mediated Signal and Is Crucial in Maintaining Slit Diaphragm of Podocyte.
    Takamura S, Fukusumi Y, Zhang Y, Narita I, Kawachi H.
    Am J Pathol; 2020 Feb 12; 190(2):333-346. PubMed ID: 31837290
    [Abstract] [Full Text] [Related]

  • 15. Genetic kidney diseases disclose the pathogenesis of proteinuria.
    Jalanko H, Patrakka J, Tryggvason K, Holmberg C.
    Ann Med; 2001 Nov 12; 33(8):526-33. PubMed ID: 11730159
    [Abstract] [Full Text] [Related]

  • 16. Loss of the podocyte glucocorticoid receptor exacerbates proteinuria after injury.
    Zhou H, Tian X, Tufro A, Moeckel G, Ishibe S, Goodwin J.
    Sci Rep; 2017 Aug 29; 7(1):9833. PubMed ID: 28852159
    [Abstract] [Full Text] [Related]

  • 17. Organization of the pronephric filtration apparatus in zebrafish requires Nephrin, Podocin and the FERM domain protein Mosaic eyes.
    Kramer-Zucker AG, Wiessner S, Jensen AM, Drummond IA.
    Dev Biol; 2005 Sep 15; 285(2):316-29. PubMed ID: 16102746
    [Abstract] [Full Text] [Related]

  • 18. CD2-associated protein and glomerular disease.
    Wolf G, Stahl RA.
    Lancet; 2003 Nov 22; 362(9397):1746-8. PubMed ID: 14643126
    [Abstract] [Full Text] [Related]

  • 19. Loss of podocyte aPKClambda/iota causes polarity defects and nephrotic syndrome.
    Huber TB, Hartleben B, Winkelmann K, Schneider L, Becker JU, Leitges M, Walz G, Haller H, Schiffer M.
    J Am Soc Nephrol; 2009 Apr 22; 20(4):798-806. PubMed ID: 19279126
    [Abstract] [Full Text] [Related]

  • 20. Inducible podocyte injury and proteinuria in transgenic zebrafish.
    Zhou W, Hildebrandt F.
    J Am Soc Nephrol; 2012 Jun 22; 23(6):1039-47. PubMed ID: 22440901
    [Abstract] [Full Text] [Related]

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