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Journal Abstract Search

211 related items for PubMed ID: 3376114

  • 1. Effect of alteration of nerve terminal Ca2+ regulation on increased spontaneous quantal release of acetylcholine by methyl mercury.
    Levesque PC, Atchison WD.
    Toxicol Appl Pharmacol; 1988 Jun 15; 94(1):55-65. PubMed ID: 3376114
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  • 2. Interactions of mitochondrial inhibitors with methylmercury on spontaneous quantal release of acetylcholine.
    Levesque PC, Atchison WD.
    Toxicol Appl Pharmacol; 1987 Feb 15; 87(2):315-24. PubMed ID: 2435022
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  • 3. Methylmercury-induced depression of neuromuscular transmission in the rat.
    Atchison WD, Narahashi T.
    Neurotoxicology; 1982 Nov 15; 3(3):37-50. PubMed ID: 6298679
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  • 4. Inhibition of mitochondrial Ca2+ release diminishes the effectiveness of methyl mercury to release acetylcholine from synaptosomes.
    Levesque PC, Hare MF, Atchison WD.
    Toxicol Appl Pharmacol; 1992 Jul 15; 115(1):11-20. PubMed ID: 1378659
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  • 5. Extracellular calcium-dependent and -independent effects of methylmercury on spontaneous and potassium-evoked release of acetylcholine at the neuromuscular junction.
    Atchison WD.
    J Pharmacol Exp Ther; 1986 May 15; 237(2):672-80. PubMed ID: 3084765
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  • 11. Measurement of quantal secretion induced by ouabain and its correlation with depletion of synaptic vesicles.
    Haimann C, Torri-Tarelli F, Fesce R, Ceccarelli B.
    J Cell Biol; 1985 Nov 15; 101(5 Pt 1):1953-65. PubMed ID: 3932368
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  • 12. Reversal of methylmercury-induced block of nerve-evoked release of acetylcholine at the neuromuscular junction.
    Traxinger DL, Atchison WD.
    Toxicol Appl Pharmacol; 1987 Aug 15; 90(1):23-33. PubMed ID: 3629589
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  • 14. Comparative effects of divalent cations on the methylmercury-induced alterations of acetylcholine release.
    Traxinger DL, Atchison WD.
    J Pharmacol Exp Ther; 1987 Feb 15; 240(2):451-9. PubMed ID: 3027312
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  • 15. Changes in acetylcholine concentration, miniature end-plate potentials and synaptic vesicles in frog neuromuscular preparations during lanthanum treatment.
    Florey E, Kriebel ME.
    Comp Biochem Physiol C Comp Pharmacol Toxicol; 1983 Feb 15; 75(2):285-94. PubMed ID: 6138197
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  • 19. The thiol-oxidizing agent diamide increases transmitter release by decreasing calcium requirements for neuromuscular transmission in the frog.
    Carlen PL, Kosower EM, Werman R.
    Brain Res; 1976 Nov 26; 117(2):257-76. PubMed ID: 186154
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  • 20. Calcium-dependent actions of the convulsant barbiturate, CHEB, on transmitter release at the rat neuromuscular junction.
    Nicholson GM, Spence I, Johnston GA.
    Gen Pharmacol; 1990 Nov 26; 21(5):741-6. PubMed ID: 1980474
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