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Journal Abstract Search

175 related items for PubMed ID: 33790807

  • 1. Spleen Tyrosine Kinase Inhibition Ameliorates Tubular Inflammation in IgA Nephropathy.
    Yiu WH, Chan KW, Chan LYY, Leung JCK, Lai KN, Tang SCW.
    Front Physiol; 2021; 12():650888. PubMed ID: 33790807
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  • 3. Spleen tyrosine kinase is important in the production of proinflammatory cytokines and cell proliferation in human mesangial cells following stimulation with IgA1 isolated from IgA nephropathy patients.
    Kim MJ, McDaid JP, McAdoo SP, Barratt J, Molyneux K, Masuda ES, Pusey CD, Tam FW.
    J Immunol; 2012 Oct 01; 189(7):3751-8. PubMed ID: 22956578
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  • 4. Protective effect of peroxisome proliferator-activated receptor-gamma agonists on activated renal proximal tubular epithelial cells in IgA nephropathy.
    Xiao J, Leung JC, Chan LY, Guo H, Lai KN.
    Nephrol Dial Transplant; 2009 Jul 01; 24(7):2067-77. PubMed ID: 19155534
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  • 5. Synthesis of TNF-alpha by mesangial cells cultured with polymeric anionic IgA--role of MAPK and NF-kappaB.
    Leung JC, Tang SC, Chan LY, Chan WL, Lai KN.
    Nephrol Dial Transplant; 2008 Jan 01; 23(1):72-81. PubMed ID: 17898021
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  • 6. Crosstalk between peroxisome proliferator-activated receptor-gamma and angiotensin II in renal tubular epithelial cells in IgA nephropathy.
    Xiao J, Leung JC, Chan LY, Tang SC, Lai KN.
    Clin Immunol; 2009 Aug 01; 132(2):266-76. PubMed ID: 19443277
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  • 7. Role of the Spleen Tyrosine Kinase Pathway in Driving Inflammation in IgA Nephropathy.
    McAdoo S, Tam FWK.
    Semin Nephrol; 2018 Sep 01; 38(5):496-503. PubMed ID: 30177021
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  • 8. Role of Human Mesangial-Tubular Crosstalk in Secretory IgA-Induced IgA Nephropathy.
    Zhang J, Zhou R, Mi Y, Liu Z, Huang B, Guo R, Wang P, Quan S, Zhou Y.
    Kidney Blood Press Res; 2021 Sep 01; 46(3):286-297. PubMed ID: 33866316
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  • 11. Tubular expression of angiotensin II receptors and their regulation in IgA nephropathy.
    Chan LY, Leung JC, Tang SC, Choy CB, Lai KN.
    J Am Soc Nephrol; 2005 Aug 01; 16(8):2306-17. PubMed ID: 15930094
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  • 14. Role of Mesangial-Podocytic-Tubular Cross-Talk in IgA Nephropathy.
    Leung JCK, Lai KN, Tang SCW.
    Semin Nephrol; 2018 Sep 01; 38(5):485-495. PubMed ID: 30177020
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  • 15. Macrophage interactions with collecting duct epithelial cells are capable of driving tubulointerstitial inflammation and fibrosis in immunoglobulin A nephropathy.
    Pawluczyk IZA, Soares MSF, Barratt WA, Brown JR, Bhachu JS, Selvaskandan H, Zeng Y, Sarania R, Molyneux K, Roberts ISD, Barratt J.
    Nephrol Dial Transplant; 2020 Nov 01; 35(11):1865-1877. PubMed ID: 32830258
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  • 16. Inhibition of activated human mesangial cell proliferation by the natural product of Cordyceps sinensis (H1-A): an implication for treatment of IgA mesangial nephropathy.
    Lin CY, Ku FM, Kuo YC, Chen CF, Chen WP, Chen A, Shiao MS.
    J Lab Clin Med; 1999 Jan 01; 133(1):55-63. PubMed ID: 10385482
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  • 17. Combined blockade of angiotensin II and prorenin receptors ameliorates podocytic apoptosis induced by IgA-activated mesangial cells.
    Leung JC, Chan LY, Saleem MA, Mathieson PW, Tang SC, Lai KN.
    Apoptosis; 2015 Jul 01; 20(7):907-20. PubMed ID: 25808596
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  • 18. Complement-C1q TNF-Related Protein 3 Alleviates Mesangial Cell Activation and Inflammatory Response Stimulated by Secretory IgA.
    Zhang R, Zhong L, Zhou J, Peng Y.
    Am J Nephrol; 2016 Jul 01; 43(6):460-8. PubMed ID: 27309491
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