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Journal Abstract Search

426 related items for PubMed ID: 34023353

  • 1. Dimethyl fumarate preserves left ventricular infarct integrity following myocardial infarction via modulation of cardiac macrophage and fibroblast oxidative metabolism.
    Mouton AJ, Flynn ER, Moak SP, Aitken NM, Omoto ACM, Li X, da Silva AA, Wang Z, do Carmo JM, Hall JE.
    J Mol Cell Cardiol; 2021 Sep; 158():38-48. PubMed ID: 34023353
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  • 2. Topiramate modulates post-infarction inflammation primarily by targeting monocytes or macrophages.
    Wang Z, Huang S, Sheng Y, Peng X, Liu H, Jin N, Cai J, Shu Y, Li T, Li P, Fan C, Hu X, Zhang W, Long R, You Y, Huang C, Song Y, Xiang C, Wang J, Yang Y, Liu K.
    Cardiovasc Res; 2017 Apr 01; 113(5):475-487. PubMed ID: 28339742
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  • 4. Lgr4 Governs a Pro-Inflammatory Program in Macrophages to Antagonize Post-Infarction Cardiac Repair.
    Huang CK, Dai D, Xie H, Zhu Z, Hu J, Su M, Liu M, Lu L, Shen W, Ning G, Wang J, Zhang R, Yan X.
    Circ Res; 2020 Sep 25; 127(8):953-973. PubMed ID: 32600176
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  • 6. Aminooxyacetic acid attenuates post-infarct cardiac dysfunction by balancing macrophage polarization through modulating macrophage metabolism in mice.
    Zhao P, Zhou W, Zhang Y, Li J, Zhao Y, Pan L, Shen Z, Chen W, Hui J.
    J Cell Mol Med; 2020 Feb 25; 24(4):2593-2609. PubMed ID: 31930778
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  • 7. Post-infarct morphine treatment mitigates left ventricular remodeling and dysfunction in a rat model of ischemia-reperfusion.
    Rajani SF, Imani A, Faghihi M, Izad M, Kardar GA, Salehi Z.
    Eur J Pharmacol; 2019 Mar 15; 847():61-71. PubMed ID: 30684466
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  • 8. C-X-C Motif Chemokine Receptor 4 Blockade Promotes Tissue Repair After Myocardial Infarction by Enhancing Regulatory T Cell Mobilization and Immune-Regulatory Function.
    Wang Y, Dembowsky K, Chevalier E, Stüve P, Korf-Klingebiel M, Lochner M, Napp LC, Frank H, Brinkmann E, Kanwischer A, Bauersachs J, Gyöngyösi M, Sparwasser T, Wollert KC.
    Circulation; 2019 Apr 09; 139(15):1798-1812. PubMed ID: 30696265
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  • 14. Fibroblast polarization over the myocardial infarction time continuum shifts roles from inflammation to angiogenesis.
    Mouton AJ, Ma Y, Rivera Gonzalez OJ, Daseke MJ, Flynn ER, Freeman TC, Garrett MR, DeLeon-Pennell KY, Lindsey ML.
    Basic Res Cardiol; 2019 Jan 11; 114(2):6. PubMed ID: 30635789
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  • 17. Bone marrow-derived CXCR4+ cells mobilized by macrophage colony-stimulating factor participate in the reduction of infarct area and improvement of cardiac remodeling after myocardial infarction in mice.
    Morimoto H, Takahashi M, Shiba Y, Izawa A, Ise H, Hongo M, Hatake K, Motoyoshi K, Ikeda U.
    Am J Pathol; 2007 Sep 11; 171(3):755-66. PubMed ID: 17640962
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  • 18. Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.
    Meschiari CA, Jung M, Iyer RP, Yabluchanskiy A, Toba H, Garrett MR, Lindsey ML.
    Am J Physiol Heart Circ Physiol; 2018 Feb 01; 314(2):H224-H235. PubMed ID: 29030341
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  • 20. IL-33 induces type-2-cytokine phenotype but exacerbates cardiac remodeling post-myocardial infarction with eosinophil recruitment, worsened systolic dysfunction, and ventricular wall rupture.
    Ghali R, Habeichi NJ, Kaplan A, Tannous C, Abidi E, Bekdash A, Farhat R, Itani H, Jurjus A, Booz GW, Mallat Z, Zouein FA.
    Clin Sci (Lond); 2020 Jun 12; 134(11):1191-1218. PubMed ID: 32432676
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