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PUBMED FOR HANDHELDS

Journal Abstract Search


131 related items for PubMed ID: 34089843

  • 1. Arsenite impinges on endoplasmic reticulum-mitochondria crosstalk to elicit mitochondrial ROS formation and downstream toxicity.
    Cantoni O, Zito E, Fiorani M, Guidarelli A.
    Semin Cancer Biol; 2021 Nov; 76():132-138. PubMed ID: 34089843
    [Abstract] [Full Text] [Related]

  • 2. Arsenite-Induced Mitochondrial Superoxide Formation: Time and Concentration Requirements for the Effects of the Metalloid on the Endoplasmic Reticulum and Mitochondria.
    Guidarelli A, Cerioni L, Fiorani M, Catalani A, Cantoni O.
    J Pharmacol Exp Ther; 2020 Apr; 373(1):62-71. PubMed ID: 31941719
    [Abstract] [Full Text] [Related]

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  • 4. Crosstalk between ERO1α and ryanodine receptor in arsenite-dependent mitochondrial ROS formation.
    Spina A, Guidarelli A, Fiorani M, Varone E, Catalani A, Zito E, Cantoni O.
    Biochem Pharmacol; 2022 Apr; 198():114973. PubMed ID: 35189109
    [Abstract] [Full Text] [Related]

  • 5. ERO1α primes the ryanodine receptor to respond to arsenite with concentration dependent Ca2+ release sequentially triggering two different mechanisms of ROS formation.
    Guidarelli A, Spina A, Buffi G, Blandino G, Fiorani M, Cantoni O.
    Chem Biol Interact; 2023 Sep 25; 383():110694. PubMed ID: 37659621
    [Abstract] [Full Text] [Related]

  • 6. Calcium signals between the ryanodine receptor- and mitochondria critically regulate the effects of arsenite on mitochondrial superoxide formation and on the ensuing survival vs apoptotic signaling.
    Guidarelli A, Fiorani M, Cerioni L, Cantoni O.
    Redox Biol; 2019 Jan 25; 20():285-295. PubMed ID: 30388683
    [Abstract] [Full Text] [Related]

  • 7. Low Concentrations of Arsenite Target the Intraluminal Inositol 1, 4, 5-Trisphosphate Receptor/Ryanodine Receptor Crosstalk to Significantly Elevate Intracellular Ca2.
    Guidarelli A, Fiorani M, Cantoni O.
    J Pharmacol Exp Ther; 2018 Oct 25; 367(1):184-193. PubMed ID: 30068729
    [Abstract] [Full Text] [Related]

  • 8. Arsenite enhances ERO1α expression via ryanodine receptor dependent and independent mechanisms.
    Guidarelli A, Spina A, Fiorani M, Zito E, Cantoni O.
    Environ Toxicol Pharmacol; 2023 Mar 25; 98():104080. PubMed ID: 36781116
    [Abstract] [Full Text] [Related]

  • 9. Inhibition of activity/expression, or genetic deletion, of ERO1α blunts arsenite geno- and cyto-toxicity.
    Guidarelli A, Spina A, Fiorani M, Zito E, Cantoni O.
    Food Chem Toxicol; 2022 Oct 25; 168():113360. PubMed ID: 35964836
    [Abstract] [Full Text] [Related]

  • 10. The dual role of mitochondrial superoxide in arsenite toxicity: Signaling at the boundary between apoptotic commitment and cytoprotection.
    Fiorani M, Guidarelli A, Capellacci V, Cerioni L, Crinelli R, Cantoni O.
    Toxicol Appl Pharmacol; 2018 Apr 15; 345():26-35. PubMed ID: 29526526
    [Abstract] [Full Text] [Related]

  • 11. The study of the mechanism of arsenite toxicity in respiration-deficient cells reveals that NADPH oxidase-derived superoxide promotes the same downstream events mediated by mitochondrial superoxide in respiration-proficient cells.
    Guidarelli A, Fiorani M, Carloni S, Cerioni L, Balduini W, Cantoni O.
    Toxicol Appl Pharmacol; 2016 Sep 15; 307():35-44. PubMed ID: 27450018
    [Abstract] [Full Text] [Related]

  • 12. The compartmentalised nature of the mechanisms governing superoxide formation and scavenging in cells exposed to arsenite.
    Guidarelli A, Fiorani M, Cerioni L, Cantoni O.
    Toxicol Appl Pharmacol; 2019 Dec 01; 384():114766. PubMed ID: 31647942
    [Abstract] [Full Text] [Related]

  • 13. Arsenite-induced endoplasmic reticulum-dependent apoptosis through disturbance of calcium homeostasis in HBE cell line.
    Chen C, Gu S, Jiang X, Zhang Z.
    Environ Toxicol; 2017 Jan 01; 32(1):197-216. PubMed ID: 26677073
    [Abstract] [Full Text] [Related]

  • 14. Arsenite induces DNA damage via mitochondrial ROS and induction of mitochondrial permeability transition.
    Guidarelli A, Fiorani M, Cerioni L, Scotti M, Cantoni O.
    Biofactors; 2017 Sep 10; 43(5):673-684. PubMed ID: 28703385
    [Abstract] [Full Text] [Related]

  • 15. Mitochondrial redox-driven mitofusin 2 S-glutathionylation promotes neuronal necroptosis via disrupting ER-mitochondria crosstalk in cadmium-induced neurotoxicity.
    Che L, Yang CL, Chen Y, Wu ZL, Du ZB, Wu JS, Gan CL, Yan SP, Huang J, Guo NJ, Lin YC, Lin ZN.
    Chemosphere; 2021 Jan 10; 262():127878. PubMed ID: 33182097
    [Abstract] [Full Text] [Related]

  • 16. Old players in a new role: mitochondria-associated membranes, VDAC, and ryanodine receptors as contributors to calcium signal propagation from endoplasmic reticulum to the mitochondria.
    Hajnóczky G, Csordás G, Yi M.
    Cell Calcium; 2002 Jan 10; 32(5-6):363-77. PubMed ID: 12543096
    [Abstract] [Full Text] [Related]

  • 17. Alleviation of palmitic acid-induced endoplasmic reticulum stress by augmenter of liver regeneration through IP3R-controlled Ca2+ release.
    Xiao WC, Zhang J, Chen SL, Shi YJ, Xiao F, An W.
    J Cell Physiol; 2018 Aug 10; 233(8):6148-6157. PubMed ID: 29323715
    [Abstract] [Full Text] [Related]

  • 18. Inhibition of IP3R/Ca2+ Dysregulation Protects Mice From Ventilator-Induced Lung Injury via Endoplasmic Reticulum and Mitochondrial Pathways.
    Ye L, Zeng Q, Ling M, Ma R, Chen H, Lin F, Li Z, Pan L.
    Front Immunol; 2021 Aug 10; 12():729094. PubMed ID: 34603302
    [Abstract] [Full Text] [Related]

  • 19. Calreticulin regulated intrinsic apoptosis through mitochondria-dependent and independent pathways mediated by ER stress in arsenite exposed HT-22 cells.
    Wang X, Liu X, Chen Y, Wang H, Zhang R, Zhang Q, Wei Y, Shi S, Li X.
    Chemosphere; 2020 Jul 10; 251():126466. PubMed ID: 32443253
    [Abstract] [Full Text] [Related]

  • 20. Roles of IP3R and RyR Ca2+ channels in endoplasmic reticulum stress and beta-cell death.
    Luciani DS, Gwiazda KS, Yang TL, Kalynyak TB, Bychkivska Y, Frey MH, Jeffrey KD, Sampaio AV, Underhill TM, Johnson JD.
    Diabetes; 2009 Feb 10; 58(2):422-32. PubMed ID: 19033399
    [Abstract] [Full Text] [Related]


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