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131 related items for PubMed ID: 34089843
1. Arsenite impinges on endoplasmic reticulum-mitochondria crosstalk to elicit mitochondrial ROS formation and downstream toxicity. Cantoni O, Zito E, Fiorani M, Guidarelli A. Semin Cancer Biol; 2021 Nov; 76():132-138. PubMed ID: 34089843 [Abstract] [Full Text] [Related]
2. Arsenite-Induced Mitochondrial Superoxide Formation: Time and Concentration Requirements for the Effects of the Metalloid on the Endoplasmic Reticulum and Mitochondria. Guidarelli A, Cerioni L, Fiorani M, Catalani A, Cantoni O. J Pharmacol Exp Ther; 2020 Apr; 373(1):62-71. PubMed ID: 31941719 [Abstract] [Full Text] [Related]
4. Crosstalk between ERO1α and ryanodine receptor in arsenite-dependent mitochondrial ROS formation. Spina A, Guidarelli A, Fiorani M, Varone E, Catalani A, Zito E, Cantoni O. Biochem Pharmacol; 2022 Apr; 198():114973. PubMed ID: 35189109 [Abstract] [Full Text] [Related]
5. ERO1α primes the ryanodine receptor to respond to arsenite with concentration dependent Ca2+ release sequentially triggering two different mechanisms of ROS formation. Guidarelli A, Spina A, Buffi G, Blandino G, Fiorani M, Cantoni O. Chem Biol Interact; 2023 Sep 25; 383():110694. PubMed ID: 37659621 [Abstract] [Full Text] [Related]
6. Calcium signals between the ryanodine receptor- and mitochondria critically regulate the effects of arsenite on mitochondrial superoxide formation and on the ensuing survival vs apoptotic signaling. Guidarelli A, Fiorani M, Cerioni L, Cantoni O. Redox Biol; 2019 Jan 25; 20():285-295. PubMed ID: 30388683 [Abstract] [Full Text] [Related]
7. Low Concentrations of Arsenite Target the Intraluminal Inositol 1, 4, 5-Trisphosphate Receptor/Ryanodine Receptor Crosstalk to Significantly Elevate Intracellular Ca2. Guidarelli A, Fiorani M, Cantoni O. J Pharmacol Exp Ther; 2018 Oct 25; 367(1):184-193. PubMed ID: 30068729 [Abstract] [Full Text] [Related]
8. Arsenite enhances ERO1α expression via ryanodine receptor dependent and independent mechanisms. Guidarelli A, Spina A, Fiorani M, Zito E, Cantoni O. Environ Toxicol Pharmacol; 2023 Mar 25; 98():104080. PubMed ID: 36781116 [Abstract] [Full Text] [Related]
9. Inhibition of activity/expression, or genetic deletion, of ERO1α blunts arsenite geno- and cyto-toxicity. Guidarelli A, Spina A, Fiorani M, Zito E, Cantoni O. Food Chem Toxicol; 2022 Oct 25; 168():113360. PubMed ID: 35964836 [Abstract] [Full Text] [Related]
10. The dual role of mitochondrial superoxide in arsenite toxicity: Signaling at the boundary between apoptotic commitment and cytoprotection. Fiorani M, Guidarelli A, Capellacci V, Cerioni L, Crinelli R, Cantoni O. Toxicol Appl Pharmacol; 2018 Apr 15; 345():26-35. PubMed ID: 29526526 [Abstract] [Full Text] [Related]
11. The study of the mechanism of arsenite toxicity in respiration-deficient cells reveals that NADPH oxidase-derived superoxide promotes the same downstream events mediated by mitochondrial superoxide in respiration-proficient cells. Guidarelli A, Fiorani M, Carloni S, Cerioni L, Balduini W, Cantoni O. Toxicol Appl Pharmacol; 2016 Sep 15; 307():35-44. PubMed ID: 27450018 [Abstract] [Full Text] [Related]
12. The compartmentalised nature of the mechanisms governing superoxide formation and scavenging in cells exposed to arsenite. Guidarelli A, Fiorani M, Cerioni L, Cantoni O. Toxicol Appl Pharmacol; 2019 Dec 01; 384():114766. PubMed ID: 31647942 [Abstract] [Full Text] [Related]
13. Arsenite-induced endoplasmic reticulum-dependent apoptosis through disturbance of calcium homeostasis in HBE cell line. Chen C, Gu S, Jiang X, Zhang Z. Environ Toxicol; 2017 Jan 01; 32(1):197-216. PubMed ID: 26677073 [Abstract] [Full Text] [Related]
14. Arsenite induces DNA damage via mitochondrial ROS and induction of mitochondrial permeability transition. Guidarelli A, Fiorani M, Cerioni L, Scotti M, Cantoni O. Biofactors; 2017 Sep 10; 43(5):673-684. PubMed ID: 28703385 [Abstract] [Full Text] [Related]
15. Mitochondrial redox-driven mitofusin 2 S-glutathionylation promotes neuronal necroptosis via disrupting ER-mitochondria crosstalk in cadmium-induced neurotoxicity. Che L, Yang CL, Chen Y, Wu ZL, Du ZB, Wu JS, Gan CL, Yan SP, Huang J, Guo NJ, Lin YC, Lin ZN. Chemosphere; 2021 Jan 10; 262():127878. PubMed ID: 33182097 [Abstract] [Full Text] [Related]
16. Old players in a new role: mitochondria-associated membranes, VDAC, and ryanodine receptors as contributors to calcium signal propagation from endoplasmic reticulum to the mitochondria. Hajnóczky G, Csordás G, Yi M. Cell Calcium; 2002 Jan 10; 32(5-6):363-77. PubMed ID: 12543096 [Abstract] [Full Text] [Related]
17. Alleviation of palmitic acid-induced endoplasmic reticulum stress by augmenter of liver regeneration through IP3R-controlled Ca2+ release. Xiao WC, Zhang J, Chen SL, Shi YJ, Xiao F, An W. J Cell Physiol; 2018 Aug 10; 233(8):6148-6157. PubMed ID: 29323715 [Abstract] [Full Text] [Related]
18. Inhibition of IP3R/Ca2+ Dysregulation Protects Mice From Ventilator-Induced Lung Injury via Endoplasmic Reticulum and Mitochondrial Pathways. Ye L, Zeng Q, Ling M, Ma R, Chen H, Lin F, Li Z, Pan L. Front Immunol; 2021 Aug 10; 12():729094. PubMed ID: 34603302 [Abstract] [Full Text] [Related]
19. Calreticulin regulated intrinsic apoptosis through mitochondria-dependent and independent pathways mediated by ER stress in arsenite exposed HT-22 cells. Wang X, Liu X, Chen Y, Wang H, Zhang R, Zhang Q, Wei Y, Shi S, Li X. Chemosphere; 2020 Jul 10; 251():126466. PubMed ID: 32443253 [Abstract] [Full Text] [Related]
20. Roles of IP3R and RyR Ca2+ channels in endoplasmic reticulum stress and beta-cell death. Luciani DS, Gwiazda KS, Yang TL, Kalynyak TB, Bychkivska Y, Frey MH, Jeffrey KD, Sampaio AV, Underhill TM, Johnson JD. Diabetes; 2009 Feb 10; 58(2):422-32. PubMed ID: 19033399 [Abstract] [Full Text] [Related] Page: [Next] [New Search]