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PUBMED FOR HANDHELDS

Journal Abstract Search


260 related items for PubMed ID: 3422420

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  • 4. Erythrocyte and plasma oxidative stress appears to be compensated in patients with sickle cell disease during a period of relative health, despite the presence of known oxidative agents.
    Detterich JA, Liu H, Suriany S, Kato RM, Chalacheva P, Tedla B, Shah PM, Khoo MC, Wood JC, Coates TD, Milne GL, Oh JY, Patel RP, Forman HJ.
    Free Radic Biol Med; 2019 Sep; 141():408-415. PubMed ID: 31279092
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  • 7. Denaturing interaction between sickle hemoglobin and phosphatidylserine liposomes.
    Marva E, Hebbel RP.
    Blood; 1994 Jan 01; 83(1):242-9. PubMed ID: 8274739
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  • 8. Responses of normal and sickle cell hemoglobin to S-nitroscysteine: implications for therapeutic applications of NO in treatment of sickle cell disease.
    Bonaventura C, Godette G, Ferruzzi G, Tesh S, Stevens RD, Henkens R.
    Biophys Chem; 2002 Jul 10; 98(1-2):165-81. PubMed ID: 12128197
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  • 9. Denatured hemoglobin in sickle erythrocytes.
    Asakura T, Minakata K, Adachi K, Russell MO, Schwartz E.
    J Clin Invest; 1977 Apr 10; 59(4):633-40. PubMed ID: 845254
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  • 10. Substitutions in the β subunits of sickle-cell hemoglobin improve oxidative stability and increase the delay time of sickle-cell fiber formation.
    Meng F, Kassa T, Strader MB, Soman J, Olson JS, Alayash AI.
    J Biol Chem; 2019 Mar 15; 294(11):4145-4159. PubMed ID: 30630954
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  • 12. Oxidative pathways in the sickle cell and beyond.
    Alayash AI.
    Blood Cells Mol Dis; 2018 May 15; 70():78-86. PubMed ID: 28554826
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  • 17. Influence of sickle hemoglobin polymerization and membrane properties on deformability of sickle erythrocytes in the microcirculation.
    Dong C, Chadwick RS, Schechter AN.
    Biophys J; 1992 Sep 15; 63(3):774-83. PubMed ID: 1420913
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  • 20. Autoxidation, dehydration, and adhesivity may be related abnormalities of sickle erythrocytes.
    Hebbel RP, Ney PA, Foker W.
    Am J Physiol; 1989 Mar 15; 256(3 Pt 1):C579-83. PubMed ID: 2923193
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