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Journal Abstract Search

318 related items for PubMed ID: 34373735

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  • 2. Discovery, Structure-Activity Relationship and In Vitro Anticancer Activity of Small-Molecule Inhibitors of the Protein-Protein Interactions between AF9/ENL and AF4 or DOT1L.
    Li X, Wu X, Nie S, Zhao J, Yao Y, Wu F, Mishra CB, Ashraf-Uz-Zaman M, Moku BK, Song Y.
    Cancers (Basel); 2023 Nov 03; 15(21):. PubMed ID: 37958457
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  • 3. Targeting recruitment of disruptor of telomeric silencing 1-like (DOT1L): characterizing the interactions between DOT1L and mixed lineage leukemia (MLL) fusion proteins.
    Shen C, Jo SY, Liao C, Hess JL, Nikolovska-Coleska Z.
    J Biol Chem; 2013 Oct 18; 288(42):30585-30596. PubMed ID: 23996074
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  • 4. A proteolysis-targeting chimera molecule selectively degrades ENL and inhibits malignant gene expression and tumor growth.
    Li X, Yao Y, Wu F, Song Y.
    J Hematol Oncol; 2022 Apr 08; 15(1):41. PubMed ID: 35395864
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  • 5. Human Polymerase-Associated Factor complex (PAFc) connects the Super Elongation Complex (SEC) to RNA polymerase II on chromatin.
    He N, Chan CK, Sobhian B, Chou S, Xue Y, Liu M, Alber T, Benkirane M, Zhou Q.
    Proc Natl Acad Sci U S A; 2011 Sep 06; 108(36):E636-45. PubMed ID: 21873227
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  • 12. The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia.
    Wang WT, Chen TQ, Zeng ZC, Pan Q, Huang W, Han C, Fang K, Sun LY, Yang QQ, Wang D, Luo XQ, Sun YM, Chen YQ.
    J Hematol Oncol; 2020 Jun 17; 13(1):78. PubMed ID: 32552847
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  • 13. Peptidomimetics for Targeting Protein-Protein Interactions between DOT1L and MLL Oncofusion Proteins AF9 and ENL.
    Du L, Grigsby SM, Yao A, Chang Y, Johnson G, Sun H, Nikolovska-Coleska Z.
    ACS Med Chem Lett; 2018 Sep 13; 9(9):895-900. PubMed ID: 30258537
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  • 16. Selective DOT1L, LSD1, and HDAC Class I Inhibitors Reduce HOXA9 Expression in MLL-AF9 Rearranged Leukemia Cells, But Dysregulate the Expression of Many Histone-Modifying Enzymes.
    Lillico R, Lawrence CK, Lakowski TM.
    J Proteome Res; 2018 Aug 03; 17(8):2657-2667. PubMed ID: 29972300
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  • 17. DOT1L, the H3K79 methyltransferase, is required for MLL-AF9-mediated leukemogenesis.
    Nguyen AT, Taranova O, He J, Zhang Y.
    Blood; 2011 Jun 23; 117(25):6912-22. PubMed ID: 21521783
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  • 18. H3K79 methylation profiles define murine and human MLL-AF4 leukemias.
    Krivtsov AV, Feng Z, Lemieux ME, Faber J, Vempati S, Sinha AU, Xia X, Jesneck J, Bracken AP, Silverman LB, Kutok JL, Kung AL, Armstrong SA.
    Cancer Cell; 2008 Nov 04; 14(5):355-68. PubMed ID: 18977325
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  • 19. MLL-rearranged leukemia is dependent on aberrant H3K79 methylation by DOT1L.
    Bernt KM, Zhu N, Sinha AU, Vempati S, Faber J, Krivtsov AV, Feng Z, Punt N, Daigle A, Bullinger L, Pollock RM, Richon VM, Kung AL, Armstrong SA.
    Cancer Cell; 2011 Jul 12; 20(1):66-78. PubMed ID: 21741597
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  • 20. Degree of recruitment of DOT1L to MLL-AF9 defines level of H3K79 Di- and tri-methylation on target genes and transformation potential.
    Kuntimaddi A, Achille NJ, Thorpe J, Lokken AA, Singh R, Hemenway CS, Adli M, Zeleznik-Le NJ, Bushweller JH.
    Cell Rep; 2015 May 05; 11(5):808-20. PubMed ID: 25921540
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