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Journal Abstract Search

199 related items for PubMed ID: 34454011

  • 1. Intergenerational genetic programming mechanism and sex differences of the adrenal corticosterone synthesis dysfunction in offspring induced by prenatal ethanol exposure.
    Cao J, Chen Y, Xia X, Qu H, Ao Y, Wang H.
    Toxicol Lett; 2021 Oct 15; 351():78-88. PubMed ID: 34454011
    [Abstract] [Full Text] [Related]

  • 2. Prenatal ethanol exposure-induced adrenal developmental abnormality of male offspring rats and its possible intrauterine programming mechanisms.
    Huang H, He Z, Zhu C, Liu L, Kou H, Shen L, Wang H.
    Toxicol Appl Pharmacol; 2015 Oct 01; 288(1):84-94. PubMed ID: 26188107
    [Abstract] [Full Text] [Related]

  • 3. Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats.
    Liu M, Zhang Q, Pei L, Zou Y, Chen G, Wang H.
    Epigenetics; 2019 Mar 01; 14(3):245-259. PubMed ID: 30821590
    [Abstract] [Full Text] [Related]

  • 4. Glucocorticoid programming mechanism for hypercholesterolemia in prenatal ethanol-exposed adult offspring rats.
    Hu S, Qin J, Zhou J, Magdalou J, Chen L, Xu D, Wang H.
    Toxicol Appl Pharmacol; 2019 Jul 15; 375():46-56. PubMed ID: 31075344
    [Abstract] [Full Text] [Related]

  • 5. Glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis programming mediated hepatic lipid-metabolic in offspring caused by prenatal ethanol exposure.
    Hu W, Yuan C, Luo H, Hu S, Shen L, Chen L, Xu D, Wang H.
    Toxicol Lett; 2020 Oct 01; 331():167-177. PubMed ID: 32535229
    [Abstract] [Full Text] [Related]

  • 6. Intrauterine programming of the glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis mediates glomerulosclerosis in female adult offspring rats induced by prenatal ethanol exposure.
    He H, Xiong Y, Li B, Zhu Y, Chen H, Ao Y, Wang H.
    Toxicol Lett; 2019 Sep 01; 311():17-26. PubMed ID: 31039417
    [Abstract] [Full Text] [Related]

  • 7. Ethanol-induced inhibition of fetal hypothalamic-pituitary-adrenal axis due to prenatal overexposure to maternal glucocorticoid in mice.
    Liang G, Chen M, Pan XL, Zheng J, Wang H.
    Exp Toxicol Pathol; 2011 Nov 01; 63(7-8):607-11. PubMed ID: 20627497
    [Abstract] [Full Text] [Related]

  • 8. Nicotine-induced prenatal overexposure to maternal glucocorticoid and intrauterine growth retardation in rat.
    Chen M, Wang T, Liao ZX, Pan XL, Feng YH, Wang H.
    Exp Toxicol Pathol; 2007 Nov 01; 59(3-4):245-51. PubMed ID: 17881205
    [Abstract] [Full Text] [Related]

  • 9. Glucocorticoid-activation system mediated glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis programming alteration of adrenal dysfunction induced by prenatal caffeine exposure.
    He Z, Zhang J, Huang H, Yuan C, Zhu C, Magdalou J, Wang H.
    Toxicol Lett; 2019 Mar 01; 302():7-17. PubMed ID: 30528684
    [Abstract] [Full Text] [Related]

  • 10. Decreased H3K9ac level of KLF4 mediates podocyte developmental toxicity induced by prenatal caffeine exposure in male offspring rats.
    Zhu Y, Chen H, Zhao X, Li B, He H, Cheng H, Wang H, Ao Y.
    Toxicol Lett; 2019 Oct 10; 314():63-74. PubMed ID: 31306741
    [Abstract] [Full Text] [Related]

  • 11. Autophagy as a compensation mechanism participates in ethanol-induced fetal adrenal dysfunction in female rats.
    Huang H, Liu L, Li J, Zhu C, Xie X, Ao Y, Wang H.
    Toxicol Appl Pharmacol; 2018 Apr 15; 345():36-47. PubMed ID: 29524503
    [Abstract] [Full Text] [Related]

  • 12. H19/let-7 axis mediates caffeine exposure during pregnancy induced adrenal dysfunction and its multi-generation inheritance.
    He Z, Zhang J, Chen G, Cao J, Chen Y, Ai C, Wang H.
    Sci Total Environ; 2021 Oct 20; 792():148440. PubMed ID: 34465058
    [Abstract] [Full Text] [Related]

  • 13. Prenatal ethanol exposure programs an increased susceptibility of non-alcoholic fatty liver disease in female adult offspring rats.
    Shen L, Liu Z, Gong J, Zhang L, Wang L, Magdalou J, Chen L, Wang H.
    Toxicol Appl Pharmacol; 2014 Jan 15; 274(2):263-73. PubMed ID: 24275070
    [Abstract] [Full Text] [Related]

  • 14. Prenatal ethanol exposure-induced hypothalamic an imbalance of glutamatergic/GABAergic projections and low functional expression in male offspring rats.
    Lu J, Li Q, Ma G, Hong C, Zhang W, Wang Y, Wang H.
    Food Chem Toxicol; 2020 Jul 15; 141():111419. PubMed ID: 32437893
    [Abstract] [Full Text] [Related]

  • 15. The expressional disorder of the renal RAS mediates nephrotic syndrome of male rat offspring induced by prenatal ethanol exposure.
    Zhu Y, Zuo N, Li B, Xiong Y, Chen H, He H, Sun Z, Hu S, Cheng H, Ao Y, Wang H.
    Toxicology; 2018 May 01; 400-401():9-19. PubMed ID: 29548890
    [Abstract] [Full Text] [Related]

  • 16. Low H3K27 acetylation of SF1 in PBMC: a biomarker for prenatal dexamethasone exposure-caused adrenal insufficiency of steroid synthesis in male offspring.
    Chen G, Ai C, Duan F, Chen Y, Cao J, Zhang J, Ao Y, Wang H.
    Cell Biol Toxicol; 2023 Oct 01; 39(5):2051-2067. PubMed ID: 35246761
    [Abstract] [Full Text] [Related]

  • 17. Effects and Interactions of Prenatal Ethanol Exposure, a Post-Weaning High-Fat Diet and Gender on Adult Hypercholesterolemia Occurrence in Offspring Rats.
    Qi Y, Luo H, Hu S, Wu Y, Magdalou J, Chen L, Wang H.
    Cell Physiol Biochem; 2017 Oct 01; 44(2):657-670. PubMed ID: 29169169
    [Abstract] [Full Text] [Related]

  • 18. Programming for increased expression of hippocampal GAD67 mediated the hypersensitivity of the hypothalamic-pituitary-adrenal axis in male offspring rats with prenatal ethanol exposure.
    Lu J, Jiao Z, Yu Y, Zhang C, He X, Li Q, Xu D, Wang H.
    Cell Death Dis; 2018 May 31; 9(6):659. PubMed ID: 29855476
    [Abstract] [Full Text] [Related]

  • 19. Sex-specific increase in susceptibility to metabolic syndrome in adult offspring after prenatal ethanol exposure with post-weaning high-fat diet.
    He Z, Li J, Luo H, Zhang L, Ma L, Chen L, Wang H.
    Sci Rep; 2015 Dec 03; 5():17679. PubMed ID: 26631430
    [Abstract] [Full Text] [Related]

  • 20. Prenatal ethanol exposure induced disorder of hypothalamic-pituitary-adrenal axis-associated neuroendocrine metabolic programming alteration and dysfunction of glucose and lipid metabolism in 40-week-old female offspring rats.
    Xia L, Jiao Z, Pei L, Yuan C, Zhao Y, Guo Y, Wang H.
    Reprod Toxicol; 2020 Jun 03; 94():48-54. PubMed ID: 32333956
    [Abstract] [Full Text] [Related]

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